Pages that link to "Q28379416"
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The following pages link to Sensitizing soluble guanylyl cyclase to become a highly CO-sensitive enzyme (Q28379416):
Displaying 50 items.
- Human soluble guanylate cyclase: functional expression and revised isoenzyme family (Q24314629) (← links)
- Carbon monoxide and hydrogen sulfide: gaseous messengers in cerebrovascular circulation. (Q24537955) (← links)
- Neural roles for heme oxygenase: contrasts to nitric oxide synthase (Q24555196) (← links)
- CO-sensing mechanisms (Q24562799) (← links)
- Dynamics of carbon monoxide binding to cystathionine beta-synthase (Q24652874) (← links)
- NO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potential (Q24656131) (← links)
- NO-independent regulatory site of direct sGC stimulators like YC-1 and BAY 41-2272 (Q24798007) (← links)
- Carbon monoxide, hydrogen sulfide, and nitric oxide as signaling molecules in the gastrointestinal tract (Q27026586) (← links)
- Guanylate cyclase and the .NO/cGMP signaling pathway (Q28143751) (← links)
- Heme oxygenase-1/carbon monoxide: from basic science to therapeutic applications (Q28306266) (← links)
- Functional properties of a naturally occurring isoform of soluble guanylyl cyclase (Q28368954) (← links)
- Spare guanylyl cyclase NO receptors ensure high NO sensitivity in the vascular system (Q28513504) (← links)
- The Concise Guide to PHARMACOLOGY 2013/14: enzymes. (Q30486745) (← links)
- NO-independent regulatory site on soluble guanylate cyclase (Q32165040) (← links)
- Release of nitric oxide from endothelial cells stimulated by YC-1, an activator of soluble guanylyl cyclase (Q33179221) (← links)
- Attenuated response of L-type calcium current to nitric oxide in atrial fibrillation (Q33585378) (← links)
- Heme-based sensors in biological systems (Q33606960) (← links)
- Pathophysiology of nitric oxide and related species: free radical reactions and modification of biomolecules (Q33610044) (← links)
- Protective Effects of YC-1 Against Glutamate Induced PC12 Cell Apoptosis (Q33743118) (← links)
- The heme oxygenase-carbon monoxide system: a regulator of hepatobiliary function (Q33805174) (← links)
- Carbon monoxide--physiology, detection and controlled release. (Q33811171) (← links)
- Tonic and acute nitric oxide signaling through soluble guanylate cyclase is mediated by nonheme nitric oxide, ATP, and GTP (Q33948069) (← links)
- YC-1 activation of human soluble guanylyl cyclase has both heme-dependent and heme-independent components (Q33948966) (← links)
- Hydrogen sulfide as a gasotransmitter (Q34248391) (← links)
- Targeting heme oxygenase-1 in vascular disease (Q34299531) (← links)
- Tonic and phasic nitric oxide signals in hippocampal long-term potentiation. (Q34580308) (← links)
- Isoforms of NO-sensitive guanylyl cyclase (Q34599889) (← links)
- Oxygen mediates vascular smooth muscle relaxation in hypoxia (Q34603164) (← links)
- Importance of NO and its related compounds in enteric nervous system regulation of gut homeostasis and disease susceptibility (Q34623553) (← links)
- Discovery of riociguat (BAY 63-2521): a potent, oral stimulator of soluble guanylate cyclase for the treatment of pulmonary hypertension (Q34957241) (← links)
- Effects of nitrogen monoxide and carbon monoxide on molecular and cellular iron metabolism: mirror-image effector molecules that target iron (Q34992429) (← links)
- More than a retrograde messenger: nitric oxide needs two cGMP pathways to induce hippocampal long-term potentiation. (Q34993540) (← links)
- Altered antioxidant-oxidant status in the aqueous humor and peripheral blood of patients with retinitis pigmentosa. (Q34999044) (← links)
- Soluble guanylate cyclase as an emerging therapeutic target in cardiopulmonary disease (Q35010064) (← links)
- Activation of soluble guanylyl cyclase by YC‐1 in aortic smooth muscle but not in ventricular myocardium from rat (Q35026974) (← links)
- YC-1 potentiates nitric oxide-induced relaxation in guinea-pig trachea. (Q35041586) (← links)
- Cardiovascular actions of a novel NO-independent guanylyl cyclase stimulator, BAY 41-8543: in vivo studies (Q35044037) (← links)
- Pharmacological actions of a novel NO-independent guanylyl cyclase stimulator, BAY 41-8543: in vitro studies (Q35044042) (← links)
- NO- and haem-independent activation of soluble guanylyl cyclase: molecular basis and cardiovascular implications of a new pharmacological principle (Q35044464) (← links)
- Inhibition of rat platelet aggregation by the diazeniumdiolate nitric oxide donor MAHMA NONOate (Q35044777) (← links)
- Heme oxygenase‐1 in growth control and its clinical application to vascular disease (Q35110952) (← links)
- The soluble guanylyl cyclase activator YC-1 increases intracellular cGMP and cAMP via independent mechanisms in INS-1E cells (Q35188169) (← links)
- Gas what: NO is not the only answer to sexual function (Q35204833) (← links)
- The Soluble Guanylate Cyclase Stimulator BAY 41-2272 Inhibits Vascular Smooth Muscle Growth through the cAMP-Dependent Protein Kinase and cGMP-Dependent Protein Kinase Pathways (Q35429867) (← links)
- Novel neural modulators (Q35550535) (← links)
- Protein kinase G-regulated production of H2S governs oxygen sensing (Q35565640) (← links)
- The role of carbon monoxide in the gastrointestinal tract. (Q35651212) (← links)
- Cobinamides are novel coactivators of nitric oxide receptor that target soluble guanylyl cyclase catalytic domain (Q35776824) (← links)
- Carbon monoxide stimulates Ca2+ -dependent big-conductance K channels in the cortical collecting duct (Q36699843) (← links)
- Molecular model of a soluble guanylyl cyclase fragment determined by small-angle X-ray scattering and chemical cross-linking (Q36713981) (← links)