Pages that link to "Q28248323"
Jump to navigation
Jump to search
The following pages link to Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane (Q28248323):
Displaying 50 items.
- Cloning of the genes encoding mouse cardiac and skeletal calsequestrins: expression pattern during embryogenesis (Q22004245) (← links)
- cDNA cloning and characterization of human cardiac junctin (Q24290196) (← links)
- Molecular cloning, expression, functional characterization, chromosomal localization, and gene structure of junctate, a novel integral calcium binding protein of sarco(endo)plasmic reticulum membrane (Q24290330) (← links)
- Interaction of HRC (histidine-rich Ca(2+)-binding protein) and triadin in the lumen of sarcoplasmic reticulum (Q24291561) (← links)
- Characterization of human cardiac calsequestrin and its deleterious mutants (Q24294413) (← links)
- Identification of novel ryanodine receptor 1 (RyR1) protein interaction with calcium homeostasis endoplasmic reticulum protein (CHERP) (Q24298436) (← links)
- Role of Junctin protein interactions in cellular dynamics of calsequestrin polymer upon calcium perturbation (Q24298576) (← links)
- Calsequestrin 2 and arrhythmias (Q24300626) (← links)
- On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure (Q24301449) (← links)
- Histidine-rich Ca-binding protein interacts with sarcoplasmic reticulum Ca-ATPase (Q24307062) (← links)
- Absence of triadin, a protein of the calcium release complex, is responsible for cardiac arrhythmia with sudden death in human (Q24307681) (← links)
- Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes (Q24310819) (← links)
- Abnormal interactions of calsequestrin with the ryanodine receptor calcium release channel complex linked to exercise-induced sudden cardiac death (Q24315099) (← links)
- The role of calsequestrin, triadin, and junctin in conferring cardiac ryanodine receptor responsiveness to luminal calcium (Q24319842) (← links)
- Junctin and the histidine-rich Ca2+ binding protein: potential roles in heart failure and arrhythmogenesis (Q24320070) (← links)
- Abnormal calcium cycling and cardiac arrhythmias associated with the human Ser96Ala genetic variant of histidine-rich calcium-binding protein (Q24338370) (← links)
- Junctate is a key element in calcium entry induced by activation of InsP3 receptors and/or calcium store depletion (Q24684783) (← links)
- Triadin regulation of the ryanodine receptor complex (Q26801317) (← links)
- The ryanodine receptor leak: how a tattered receptor plunges the failing heart into crisis (Q26861625) (← links)
- Calcium signalling of human pluripotent stem cell-derived cardiomyocytes (Q27002443) (← links)
- Crystal structure and functional analysis of Drosophila Wind, a protein-disulfide isomerase-related protein (Q27641912) (← links)
- Comparing skeletal and cardiac calsequestrin structures and their calcium binding: a proposed mechanism for coupled calcium binding and protein polymerization (Q27643112) (← links)
- Structural determination of the phosphorylation domain of the ryanodine receptor (Q27671662) (← links)
- Crystal structure of calsequestrin from rabbit skeletal muscle sarcoplasmic reticulum (Q27758879) (← links)
- ATP2C1 gene mutations in Hailey-Hailey disease and possible roles of SPCA1 isoforms in membrane trafficking (Q28066726) (← links)
- Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1 (Q28143329) (← links)
- Aspartyl beta -hydroxylase (Asph) and an evolutionarily conserved isoform of Asph missing the catalytic domain share exons with junctin (Q28144446) (← links)
- A missense mutation in a highly conserved region of CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from Israel (Q28204838) (← links)
- Cardiac excitation-contraction coupling (Q28216347) (← links)
- Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia (Q28235110) (← links)
- Calsequestrin mutations and catecholaminergic polymorphic ventricular tachycardia (Q28262015) (← links)
- Characteristics of irreversible ATP activation suggest that native skeletal ryanodine receptors can be phosphorylated via an endogenous CaMKII (Q28365128) (← links)
- Calsequestrin is an inhibitor of skeletal muscle ryanodine receptor calcium release channels (Q28366867) (← links)
- Simultaneous non-negative matrix factorization for multiple large scale gene expression datasets in toxicology (Q28484389) (← links)
- Absence of post-translational aspartyl beta-hydroxylation of epidermal growth factor domains in mice leads to developmental defects and an increased incidence of intestinal neoplasia (Q28509997) (← links)
- Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias (Q28511129) (← links)
- Anesthetic- and heat-induced sudden death in calsequestrin-1-knockout mice (Q28511309) (← links)
- Molecular cloning and characterization of mouse cardiac triadin isoforms (Q28511772) (← links)
- Junctin is a prominent regulator of contractility in cardiomyocytes (Q28565063) (← links)
- Cloning and characterization of a new isoform of skeletal muscle triadin (Q28575170) (← links)
- Triadin (Trisk 95) overexpression blocks excitation-contraction coupling in rat skeletal myotubes (Q28576225) (← links)
- Characterization of the binding and phosphorylation of cardiac calsequestrin by epsilon protein kinase C (Q28581520) (← links)
- Regulation of myocardial function by histidine-rich, calcium-binding protein (Q28585120) (← links)
- A retrograde signal from calsequestrin for the regulation of store-operated Ca2+ entry in skeletal muscle (Q28586041) (← links)
- The junctional SR protein JP-45 affects the functional expression of the voltage-dependent Ca2+ channel Cav1.1 (Q28587534) (← links)
- The calsequestrin mutation CASQ2D307H does not affect protein stability and targeting to the junctional sarcoplasmic reticulum but compromises its dynamic regulation of calcium buffering (Q28589131) (← links)
- Casq2 deletion causes sarcoplasmic reticulum volume increase, premature Ca2+ release, and catecholaminergic polymorphic ventricular tachycardia (Q28589230) (← links)
- Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin (Q28590847) (← links)
- The novel skeletal muscle sarcoplasmic reticulum JP-45 protein. Molecular cloning, tissue distribution, developmental expression, and interaction with alpha 1.1 subunit of the voltage-gated calcium channel (Q28592119) (← links)
- Calcium signalling: dynamics, homeostasis and remodelling (Q29547635) (← links)