Pages that link to "Q24632892"
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The following pages link to Reversing EphB2 depletion rescues cognitive functions in Alzheimer model (Q24632892):
Displaying 50 items.
- Amyloid beta precursor protein (Q423510) (← links)
- Eph receptor B2 (Q21496072) (← links)
- Alzheimer mechanisms and therapeutic strategies (Q22252311) (← links)
- Metabotropic glutamate receptor 5 is a coreceptor for Alzheimer aβ oligomer bound to cellular prion protein (Q24322990) (← links)
- Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model (Q24603560) (← links)
- Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model (Q24624879) (← links)
- Synapses and Alzheimer's disease (Q26269868) (← links)
- Canonical and Non-canonical Reelin Signaling (Q26738423) (← links)
- Amyloid-β Receptors: The Good, the Bad, and the Prion Protein (Q26771532) (← links)
- The Role of Ephs and Ephrins in Memory Formation (Q26786905) (← links)
- Targeting the proper amyloid-beta neuronal toxins: a path forward for Alzheimer's disease immunotherapeutics (Q26822002) (← links)
- Wnts in action: from synapse formation to synaptic maintenance (Q26823548) (← links)
- Abnormal kalirin signaling in neuropsychiatric disorders. (Q26824648) (← links)
- TNF-α/NF-κB signaling in the CNS: possible connection to EPHB2 (Q26828831) (← links)
- Pathogenesis of synaptic degeneration in Alzheimer's disease and Lewy body disease (Q26853610) (← links)
- EphBs: an integral link between synaptic function and synaptopathies (Q26863347) (← links)
- Hijacking PrP(c)-dependent signal transduction: when prions impair Aβ clearance (Q27009790) (← links)
- Structural evolution and membrane interactions of Alzheimer's amyloid-beta peptide oligomers: new knowledge from single-molecule fluorescence studies (Q27010584) (← links)
- Amyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's disease (Q27012950) (← links)
- HMGB1, a pathogenic molecule that induces neurite degeneration via TLR4-MARCKS, is a potential therapeutic target for Alzheimer's disease. (Q27315985) (← links)
- The dendritic hypothesis for Alzheimer's disease pathophysiology (Q27687404) (← links)
- Elevation of brain magnesium prevents synaptic loss and reverses cognitive deficits in Alzheimer's disease mouse model (Q28247764) (← links)
- Ephrin regulation of synapse formation, function and plasticity (Q28262811) (← links)
- Aβ induces astrocytic glutamate release, extrasynaptic NMDA receptor activation, and synaptic loss (Q28293023) (← links)
- Astrocytic Ephrin-B1 Regulates Synapse Remodeling Following Traumatic Brain Injury (Q28388525) (← links)
- Direct Observation of Single Amyloid-β(1-40) Oligomers on Live Cells: Binding and Growth at Physiological Concentrations (Q28476715) (← links)
- Alzheimer's therapeutics targeting amyloid beta 1-42 oligomers I: Abeta 42 oligomer binding to specific neuronal receptors is displaced by drug candidates that improve cognitive deficits (Q28544876) (← links)
- Nanoscale Synaptic Membrane Mimetic Allows Unbiased High Throughput Screen That Targets Binding Sites for Alzheimer's-Associated Aβ Oligomers (Q28546877) (← links)
- Geniposide Protects Primary Cortical Neurons against Oligomeric Aβ1-42-Induced Neurotoxicity through a Mitochondrial Pathway (Q28551172) (← links)
- Na, K-ATPase α3 is a death target of Alzheimer patient amyloid-β assembly. (Q28572007) (← links)
- EphA4 activation of c-Abl mediates synaptic loss and LTP blockade caused by amyloid-β oligomers (Q29347252) (← links)
- Alzheimer's therapeutics targeting amyloid beta 1-42 oligomers II: Sigma-2/PGRMC1 receptors mediate Abeta 42 oligomer binding and synaptotoxicity (Q29465470) (← links)
- Epidermal growth factor receptor is a preferred target for treating amyloid-β-induced memory loss (Q30420041) (← links)
- Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β (Q30539255) (← links)
- Reducing hippocampal extracellular matrix reverses early memory deficits in a mouse model of Alzheimer's disease (Q30586616) (← links)
- Network fingerprint: a knowledge-based characterization of biomedical networks (Q30662482) (← links)
- FcγRIIb mediates amyloid-β neurotoxicity and memory impairment in Alzheimer's disease (Q30664429) (← links)
- Intercellular protein-protein interactions at synapses (Q33632852) (← links)
- Amyloid β Protein and Alzheimer's Disease: When Computer Simulations Complement Experimental Studies (Q33893017) (← links)
- Blockade of EphA4 signaling ameliorates hippocampal synaptic dysfunctions in mouse models of Alzheimer's disease (Q33919522) (← links)
- The intersection of amyloid beta and tau at synapses in Alzheimer's disease (Q34054680) (← links)
- Altered distribution of the EphA4 kinase in hippocampal brain tissue of patients with Alzheimer's disease correlates with pathology (Q34109035) (← links)
- Natural amyloid-β oligomers acutely impair the formation of a contextual fear memory in mice (Q34125360) (← links)
- Ephrin reverse signaling in axon guidance and synaptogenesis. (Q34229381) (← links)
- Life extension factor klotho enhances cognition (Q34252649) (← links)
- Toward a unified therapeutics approach targeting putative amyloid-β oligomer receptors (Q34281219) (← links)
- Human P301L-mutant tau expression in mouse entorhinal-hippocampal network causes tau aggregation and presynaptic pathology but no cognitive deficits (Q34430288) (← links)
- Life extension factor klotho prevents mortality and enhances cognition in hAPP transgenic mice (Q34462326) (← links)
- Smart Soup, a traditional Chinese medicine formula, ameliorates amyloid pathology and related cognitive deficits (Q34489284) (← links)
- Oligomeric Aβ-induced synaptic dysfunction in Alzheimer's disease (Q34544243) (← links)