Reactive oxygen species regulate quiescent T-cell apoptosis via the BH3-only proapoptotic protein BIM (Q75233308)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14713957%20AND%20SRC:MED&resulttype=core&format=json

17 November 2019

title

Reactive oxygen species regulate quiescent T-cell apoptosis via the BH3-only proapoptotic protein BIM (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14713957%20AND%20SRC:MED&resulttype=core&format=json

17 November 2019

main subject

reactive oxygen species

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1 reference

H Sade

1

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17 November 2019

A Sarin

series ordinal

2

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17 November 2019

publication date

1 April 2004

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Cell Death & Differentiation

17 November 2019

published in

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17 November 2019

volume

11

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17 November 2019

issue

4

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17 November 2019

page(s)

416-423

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The domains of death: evolution of the apoptosis machinery

17 November 2019

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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

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The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

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Microarray analysis uncovers the induction of the proapoptotic BH3-only protein Bim in multiple models of glucocorticoid-induced apoptosis

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BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

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