Inactivation of the RAD51 recombination pathway stimulates UV-induced mutagenesis in mammalian cells (Q74202416)

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9 November 2019

title

Inactivation of the RAD51 recombination pathway stimulates UV-induced mutagenesis in mammalian cells (English)

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9 November 2019

1

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9 November 2019

Bernard Lopez

series ordinal

2

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9 November 2019

language of work or name

English

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publication date

1 June 2002

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9 November 2019

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9 November 2019

volume

21

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9 November 2019

issue

25

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9 November 2019

page(s)

4065-4069

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Intrachromosomal homologous recombination in human cells which differ in nucleotide excision-repair capacity

9 November 2019

cites work

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Domain structure, localization, and function of DNA polymerase eta, defective in xeroderma pigmentosum variant cells

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Characterization of mammalian RAD51 double strand break repair using non-lethal dominant-negative forms.

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Replication of UV-damaged DNA: new insights into links between DNA polymerases, mutagenesis and human disease

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Homology-directed repair is a major double-strand break repair pathway in mammalian cells

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Mutant p53 proteins stimulate spontaneous and radiation-induced intrachromosomal homologous recombination independently of the alteration of the transactivation activity and of the G1 checkpoint.

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7 January 2021

The double-strand-break repair model for recombination

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Frequency of intrachromosomal homologous recombination induced by UV radiation in normally repairing and excision repair-deficient human cells

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Targeted disruption of the Rad51 gene leads to lethality in embryonic mice

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7 January 2021

Carcinogens can induce homologous recombination between duplicated chromosomal sequences in mouse L cells

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A plethora of lesion-replicating DNA polymerases

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