Molecular mechanisms of cisplatin-induced nephrotoxicity: a balance on the knife edge between renoprotection and tumor toxicity (Q64098643)

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29 April 2020

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Molecular mechanisms of cisplatin-induced nephrotoxicity: a balance on the knife edge between renoprotection and tumor toxicity (English)

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Valentin Djonov

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author name string

Vladislav Volarevic

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Bojana Djokovic

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Marina Gazdic Jankovic

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C Randall Harrell

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Nebojsa Arsenijevic

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13 March 2019

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Journal of Biomedical Science

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Creative Commons Attribution 4.0 International

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2 references

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Cisplatin rescue therapy: experience with sodium thiosulfate, WR2721, and diethyldithiocarbamate

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Characterization of mitochondria in cisplatin-resistant human ovarian carcinoma cells

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Cisplatin increases TNF-alpha mRNA stability in kidney proximal tubule cells.

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Cisplatin-induced nephrotoxicity is mediated by tumor necrosis factor-alpha produced by renal parenchymal cells

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Cisplatin-induced acute renal failure is associated with an increase in the cytokines interleukin (IL)-1beta, IL-18, IL-6, and neutrophil infiltration in the kidney.

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The pathological role of Bax in cisplatin nephrotoxicity

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10.1186/S12929-019-0518-9

Identifiers

DOI

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29 April 2020

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9 September 2019

https://scigraph.springernature.com/pub.10.1186/s12929-019-0518-9

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29 April 2020

PubMed publication ID

30866950

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