Individual components of the SWI/SNF chromatin remodelling complex have distinct roles in memory neurons of the mushroom body (Q64054787)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:30923190%20AND%20SRC:MED&resulttype=core&format=json

1 May 2020

title

Individual components of the SWI/SNF chromatin remodelling complex have distinct roles in memory neurons of the Drosophila mushroom body (English)

1 reference

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1 May 2020

main subject

intellectual disability

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2

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1 May 2020

Jamie M Kramer

13

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1 May 2020

author name string

Melissa C Chubak

1

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1 May 2020

Max H Stone

3

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1 May 2020

Nicholas Raun

4

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1 May 2020

Shelby L Rice

5

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1 May 2020

Mohammed Sarikahya

6

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1 May 2020

Spencer G Jones

7

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1 May 2020

Taylor A Lyons

8

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1 May 2020

Taryn E Jakub

9

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1 May 2020

Roslyn L M Mainland

10

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1 May 2020

Maria J Knip

11

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1 May 2020

Tara N Edwards

12

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1 May 2020

language of work or name

English

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publication date

25 March 2019

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:30923190%20AND%20SRC:MED&resulttype=core&format=json

Disease Models & Mechanisms

1 May 2020

published in

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1 May 2020

volume

12

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1 May 2020

issue

3

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Creative Commons Attribution 4.0 International

1 May 2020

page(s)

dmm037325

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copyright license

start time

4 September 2020

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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describes a project that uses

ImageJ

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC6451433/fullTextXML

inferred from PubMed Central ID database lookup

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Differential targeting of two distinct SWI/SNF-related Drosophila chromatin-remodeling complexes

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ftz-f1 and Hr39 opposing roles on EcR expression during Drosophila mushroom body neuron remodeling

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Heterozygous missense mutations in SMARCA2 cause Nicolaides-Baraitser syndrome

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Mutations affecting components of the SWI/SNF complex cause Coffin-Siris syndrome

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Mushroom body neuronal remodelling is necessary for short-term but not for long-term courtship memory in Drosophila

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Disruption of an EHMT1-associated chromatin-modification module causes intellectual disability

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De novo mutations in human genetic disease

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Chromatin-remodeling factor Brg1 is required for Schwann cell differentiation and myelination

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Dopamine neurons modulate pheromone responses in Drosophila courtship learning

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Range of genetic mutations associated with severe non-syndromic sporadic intellectual disability: an exome sequencing study

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Diagnostic exome sequencing in persons with severe intellectual disability

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A GAL4-driver line resource for Drosophila neurobiology

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The neuron-specific chromatin regulatory subunit BAF53b is necessary for synaptic plasticity and memory

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Chromatin regulation by BAF170 controls cerebral cortical size and thickness

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Exome sequencing to identify de novo mutations in sporadic ALS trios

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Kinetic analysis of npBAF to nBAF switching reveals exchange of SS18 with CREST and integration with neural developmental pathways

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The Brm-HDAC3-Erm repressor complex suppresses dedifferentiation in Drosophila type II neuroblast lineages

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SWI/SNF complex prevents lineage reversion and induces temporal patterning in neural stem cells

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Genome sequencing identifies major causes of severe intellectual disability

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Coffin-Siris and Nicolaides-Baraitser syndromes are a common well recognizable cause of intellectual disability

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The neuronal architecture of the mushroom body provides a logic for associative learning

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Developmental Axon Pruning Requires Destabilization of Cell Adhesion by JNK Signaling.

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Loss of BAF (mSWI/SNF) Complexes Causes Global Transcriptional and Chromatin State Changes in Forebrain Development.

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The genetic basis of natural variation in mushroom body size in Drosophila melanogaster

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Systematic Phenomics Analysis Deconvolutes Genes Mutated in Intellectual Disability into Biologically Coherent Modules.

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Ablation of BAF170 in Developing and Postnatal Dentate Gyrus Affects Neural Stem Cell Proliferation, Differentiation, and Learning

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BCL11A Haploinsufficiency Causes an Intellectual Disability Syndrome and Dysregulates Transcription

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mSWI/SNF (BAF) Complexes Are Indispensable for the Neurogenesis and Development of Embryonic Olfactory Epithelium

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Drosophila microRNA-34 Impairs Axon Pruning of Mushroom Body γ Neurons by Downregulating the Expression of Ecdysone Receptor

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Heterozygous variants in ACTL6A, encoding a component of the BAF complex, are associated with intellectual disability

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Chromatin Remodeling BAF (SWI/SNF) Complexes in Neural Development and Disorders

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AP-1 Transcription Factors and the BAF Complex Mediate Signal-Dependent Enhancer Selection

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Combining Developmental and Perturbation-Seq Uncovers Transcriptional Modules Orchestrating Neuronal Remodeling

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A Syndromic Neurodevelopmental Disorder Caused by Mutations in SMARCD1, a Core SWI/SNF Subunit Needed for Context-Dependent Neuronal Gene Regulation in Flies

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Identifiers

DOI

10.1242/DMM.037325

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:30923190%20AND%20SRC:MED&resulttype=core&format=json

1 May 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:30923190%20AND%20SRC:MED&resulttype=core&format=json

1 May 2020

PubMed publication ID

30923190

1 reference