Intestinal neuroendocrine cells and goblet cells are mediators of IL-17A-amplified epithelial IL-17C production in human inflammatory bowel disease. (Q55069382)

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scientific article published on 10 December 2014

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English	Intestinal neuroendocrine cells and goblet cells are mediators of IL-17A-amplified epithelial IL-17C production in human inflammatory bowel disease.	scientific article published on 10 December 2014

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

Intestinal neuroendocrine cells and goblet cells are mediators of IL-17A-amplified epithelial IL-17C production in human inflammatory bowel disease. (English)

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

inflammatory bowel diseases

1 reference

based on heuristic

inferred from title

author name string

M Friedrich

1

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

J Diegelmann

2

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

J Schauber

3

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

C J Auernhammer

4

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

S Brand

5

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

publication date

10 December 2014

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

Mucosal Immunology

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Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

8

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

4

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

943-958

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

Crohn's disease: Th1, Th17 or both? The change of a paradigm: new immunological and genetic insights implicate Th17 cells in the pathogenesis of Crohn's disease

1 reference

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Th17-related cytokines in inflammatory bowel diseases: friends or foes?

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IL-23 and autoimmunity: new insights into the pathogenesis of inflammatory bowel disease

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Interleukin-23/Th17 pathways and inflammatory bowel disease

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Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

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Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci

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Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47

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Novel genetic risk markers for ulcerative colitis in the IL2/IL21 region are in epistasis with IL23R and suggest a common genetic background for ulcerative colitis and celiac disease

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Identification of an IL-17-producing NK1.1(neg) iNKT cell population involved in airway neutrophilia.

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Targeting IL-17 and TH17 cells in chronic inflammation

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Structure and signalling in the IL-17 receptor family

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IL-17RE is the functional receptor for IL-17C and mediates mucosal immunity to infection with intestinal pathogens

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IL-17C regulates the innate immune function of epithelial cells in an autocrine manner

1 reference

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Cutting edge: regulation of intestinal inflammation and barrier function by IL-17C.

1 reference

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Interleukin-17C promotes Th17 cell responses and autoimmune disease via interleukin-17 receptor E.

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Toll-like receptor 5 engagement induces interleukin-17C expression in intestinal epithelial cells

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Differential alteration in intestinal epithelial cell expression of toll-like receptor 3 (TLR3) and TLR4 in inflammatory bowel disease

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Loss of TLR2 worsens spontaneous colitis in MDR1A deficiency through commensally induced pyroptosis

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Secukinumab, a human anti-IL-17A monoclonal antibody, for moderate to severe Crohn's disease: unexpected results of a randomised, double-blind placebo-controlled trial

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Effects of AIN457, a Fully Human Antibody to Interleukin-17A, on Psoriasis, Rheumatoid Arthritis, and Uveitis

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Dextran sodium sulfate (DSS) induces colitis in mice by forming nano-lipocomplexes with medium-chain-length fatty acids in the colon

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Transfer of CD4+ T cells to C.B-17 SCID mice: a model to study Th1 and Th2 cell differentiation and regulation in vivo

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Genomic responses in mouse models poorly mimic human inflammatory diseases

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Of mice and not men: differences between mouse and human immunology

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Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation

1 reference

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A genome-wide association study identifies IL23R as an inflammatory bowel disease gene

1 reference

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rs1004819 is the main disease-associated IL23R variant in German Crohn's disease patients: combined analysis of IL23R, CARD15, and OCTN1/2 variants

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21 January 2018

Intestinal DMBT1 expression is modulated by Crohn's disease-associated IL23R variants and by a DMBT1 variant which influences binding of the transcription factors CREB1 and ATF-2

1 reference

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21 January 2018

The role of the novel Th17 cytokine IL-26 in intestinal inflammation

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21 January 2018

Cell differentiation dependent expressed CCR6 mediates ERK-1/2, SAPK/JNK, and Akt signaling resulting in proliferation and migration of colorectal cancer cells

1 reference

https://api.crossref.org/works/10.1038%2FMI.2014.124

21 January 2018

Increased expression of interleukin 17 in inflammatory bowel disease

1 reference

https://api.crossref.org/works/10.1038%2FMI.2014.124

21 January 2018

IL-17A alone weakly affects the transcriptome of intestinal epithelial cells but strongly modulates the TNF-α-induced expression of inflammatory mediators and inflammatory bowel disease susceptibility genes.

1 reference

https://api.crossref.org/works/10.1038%2FMI.2014.124

21 January 2018

Alterations in the microbiota drive interleukin-17C production from intestinal epithelial cells to promote tumorigenesis

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Preferential recruitment of CCR6-expressing Th17 cells to inflamed joints via CCL20 in rheumatoid arthritis and its animal model.

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A transplantable human carcinoid as model for somatostatin receptor-mediated and amine transporter-mediated radionuclide uptake

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The adaptor Act1 is required for interleukin 17-dependent signaling associated with autoimmune and inflammatory disease.

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An overview of IL-17 function and signaling

1 reference

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Zc3h12a is an RNase essential for controlling immune responses by regulating mRNA decay

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Treatment with IL-17 prolongs the half-life of chemokine CXCL1 mRNA via the adaptor TRAF5 and the splicing-regulatory factor SF2 (ASF)

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A member of the C/EBP family, NF-IL6 beta, forms a heterodimer and transcriptionally synergizes with NF-IL6

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Functional Cooperation between Interleukin-17 and Tumor Necrosis Factor-α Is Mediated by CCAAT/Enhancer-binding Protein Family Members

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Brain-gut interactions in inflammatory bowel disease

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The IL-17 cytokine family.

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Disease activity, ANCA, and IL23R genotype status determine early response to infliximab in patients with ulcerative colitis

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Epithelial NEMO links innate immunity to chronic intestinal inflammation

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Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-γ-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment

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Identifiers

10.1038/MI.2014.124

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

16 June 2018

http://europepmc.org/abstract/MED/25492478

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