Ibotenic acid lesions of prefrontal cortex do not prevent expression of behavioral sensitization to amphetamine (Q48787253)

15 February 2018

title

Ibotenic acid lesions of prefrontal cortex do not prevent expression of behavioral sensitization to amphetamine (English)

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15 February 2018

author name string

Li Y

series ordinal

1

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15 February 2018

Wolf ME

series ordinal

2

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15 February 2018

language of work or name

English

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publication date

1 March 1997

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15 February 2018

Behavioural Brain Research

published in

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15 February 2018

volume

84

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15 February 2018

issue

1-2

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15 February 2018

page(s)

285-289

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15 February 2018

Evidence of a complete independence of the neurobiological substrates for the induction and expression of behavioral sensitization to amphetamine

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Antagonist of N-methyl-D-aspartate receptors partially prevent the development of cocaine sensitization

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Inhibition of cocaine sensitization by MK-801, a noncompetitive N-methyl-D-aspartate (NMDA) receptor antagonist: evaluation by ambulatory activity in mice.

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MK-801 prevents the development of behavioral sensitization during repeated morphine administration

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D1 receptors modulate glutamate transmission in the ventral tegmental area

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DNQX blockade of amphetamine behavioral sensitization

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Cocaine behavioral sensitization and the excitatory amino acids

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Time course of the development of the enhanced behavioral and biochemical responses to amphetamine after pretreatment with amphetamine

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Two dissociable components of behavioral sensitization following repeated amphetamine administration

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Effects of amphetamine on the release of excitatory amino acid neurotransmitters in the basal ganglia of the conscious rat.

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Prefrontal cortex regulates burst firing and transmitter release in rat mesolimbic dopamine neurons studied in vivo

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Competitive and noncompetitive NMDA antagonists block sensitization to methamphetamine

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Sensitization to systemic amphetamine produces an enhanced locomotor response to a subsequent intra-accumbens amphetamine challenge in rats

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Repeated cocaine augments excitatory amino acid transmission in the nucleus accumbens only in rats having developed behavioral sensitization.

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The neural basis of drug craving: an incentive-sensitization theory of addiction

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Sensitization to cocaine's motor activating properties produced by electrical kindling of the medial prefrontal cortex but not of the hippocampus

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Topographical organization of the efferent projections of the medial prefrontal cortex in the rat: an anterograde tract-tracing study with Phaseolus vulgaris leucoagglutinin

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Prefrontal cortical efferents in the rat synapse on unlabeled neuronal targets of catecholamine terminals in the nucleus accumbens septi and on dopamine neurons in the ventral tegmental area

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7 January 2021

Effect of repeated methamphetamine administrations on dopamine and glutamate efflux in rat prefrontal cortex

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7 January 2021

Development of both conditioning and sensitization of the behavioral activating effects of amphetamine is blocked by the non-competitive NMDA receptor antagonist, MK-801.

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Local cooling of pre-frontal cortex induces pacemaker-like firing of dopamine neurons in rat ventral tegmental area in vivo

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7 January 2021

Chronic administration of (+)-amphetamine alters the reactivity of midbrain dopaminergic neurons to prefrontal cortex stimulation in the rat

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7 January 2021

Stimulation of the prefrontal cortex in the rat induces patterns of activity in midbrain dopaminergic neurons which resemble natural burst events

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7 January 2021

Amphetamine administered to the ventral tegmental area but not to the nucleus accumbens sensitizes rats to systemic morphine: lack of conditioned effects

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Amphetamine injected into the ventral tegmental area sensitizes the nucleus accumbens dopaminergic response to systemic amphetamine: an in vivo microdialysis study in the rat.

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L-glutamate excitation of A10 dopamine neurons is preferentially mediated by activation of NMDA receptors: extra- and intracellular electrophysiological studies in brain slices

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7 January 2021

Repeated administration of MK-801 produces sensitization to its own locomotor stimulant effects but blocks sensitization to amphetamine

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7 January 2021

Coadministration of MK-801 with amphetamine, cocaine or morphine prevents rather than transiently masks the development of behavioral sensitization

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7 January 2021

MK-801 prevents alterations in the mesoaccumbens dopamine system associated with behavioral sensitization to amphetamine.

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7 January 2021

Effects of lesions of prefrontal cortex, amygdala, or fornix on behavioral sensitization to amphetamine: comparison with N-methyl-D-aspartate antagonists

1 reference

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7 January 2021

10.1016/S0166-4328(96)00158-1

Identifiers

DOI

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15 February 2018

1 reference

15 February 2018