PET in LRRK2 mutations: comparison to sporadic Parkinson's disease and evidence for presymptomatic compensation (Q46635617)

23 December 2017

title

PET in LRRK2 mutations: comparison to sporadic Parkinson's disease and evidence for presymptomatic compensation (English)

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10

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Thomas Gasser

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Thomas Gasser

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Ryan J Uitti

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Zbigniew K Wszolek

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Donald B Calne

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Vesna Sossi

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A Jon Stoessl

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M Schulzer

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Michael Schulzer

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E Mak

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Edwin Mak

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Audrey J Strongosky

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Audrey Strongosky

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author name string

John R Adams

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Hinke van Netten

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Jessamyn Mckenzie

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Thomas J Ruth

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Chong S Lee

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language of work or name

English

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publication date

4 August 2005

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23 December 2017

published in

Brain

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volume

128

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issue

Pt 12

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23 December 2017

page(s)

2777-2785

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Clinical features of LRRK2-associated Parkinson's disease in central Norway

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A common LRRK2 mutation in idiopathic Parkinson's disease

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Clinical and positron emission tomography of Parkinson's disease caused by LRRK2.

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Clinical and subclinical dopaminergic dysfunction in PARK6-linked parkinsonism: an 18F-dopa PET study

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Binding of α-dihydrotetrabenazine to the vesicular monoamine transporter is stereospecific

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Human positron emission tomographic [18F]fluorodopa studies correlate with dopamine cell counts and levels

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Genetic clues to the pathogenesis of Parkinson's disease

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Differential changes in neurochemical markers of striatal dopamine nerve terminals in idiopathic Parkinson's disease

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Autosomal dominant parkinsonism associated with variable synuclein and tau pathology

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Mutations in LRRK2 cause autosomal-dominant parkinsonism with pleomorphic pathology

21 January 2018

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The PARK8 locus in autosomal dominant parkinsonism: confirmation of linkage and further delineation of the disease-containing interval

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Genetic screening for a single common LRRK2 mutation in familial Parkinson's disease

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A frequent LRRK2 gene mutation associated with autosomal dominant Parkinson's disease

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21 January 2018

Identifiers

DOI

10.1093/BRAIN/AWH607

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23 December 2017

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23 December 2017