HD CAG repeat implicates a dominant property of huntingtin in mitochondrial energy metabolism (Q45298206)

11 December 2017

title

HD CAG repeat implicates a dominant property of huntingtin in mitochondrial energy metabolism (English)

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James F Gusella

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Marcy E MacDonald

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Ihn Sik Seong

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Elisa Fossale

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Jason M Laramie

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Elena Ivanova

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Jong-Min Lee

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Yeun Su Choo

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MaryAnne Anderson

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Mathieu Lesort

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publication date

22 August 2005

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11 December 2017

published in

Human Molecular Genetics

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volume

14

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issue

19

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11 December 2017

page(s)

2871-2880

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The predominantly HEAT-like motif structure of huntingtin and its association and coincident nuclear entry with dorsal, an NF-kB/Rel/dorsal family transcription factor

11 December 2017

cites work

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Widespread expression of Huntington's disease gene (IT15) protein product

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Polyglutamines placed into context

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Alternative excitotoxic hypotheses

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Disturbed Ca2+ signaling and apoptosis of medium spiny neurons in Huntington's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Striatal cells from mutant huntingtin knock-in mice are selectively vulnerable to mitochondrial complex II inhibitor-induced cell death through a non-apoptotic pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

The adenine nucleotide translocase: a central component of the mitochondrial permeability transition pore and key player in cell death.

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

State-dependent calcium signaling in dendritic spines of striatal medium spiny neurons

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Mitochondrial respiration and ATP production are significantly impaired in striatal cells expressing mutant huntingtin

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

In vitro effects of polyglutamine tracts on Ca2+-dependent depolarization of rat and human mitochondria: relevance to Huntington's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Mechanisms for neuronal cell death and dysfunction in Huntington's disease: pathological cross-talk between the nucleus and the mitochondria?

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Excitotoxic injury to mitochondria isolated from cultured neurons.

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Pathogenesis of polyglutamine disorders: aggregation revisited

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Autophagosome-like vacuole formation in Huntington's disease lymphoblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Raised intracellular glucose concentrations reduce aggregation and cell death caused by mutant huntingtin exon 1 by decreasing mTOR phosphorylation and inducing autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Microtubule destabilization and nuclear entry are sequential steps leading to toxicity in Huntington's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

Huntingtin controls neurotrophic support and survival of neurons by enhancing BDNF vesicular transport along microtubules

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

A neural signaling triumvirate that influences ageing and age-related disease: insulin/IGF-1, BDNF and serotonin

21 January 2018

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https://api.crossref.org/works/10.1093%2FHMG%2FDDI319

21 January 2018

Identifiers

DOI

10.1093/HMG/DDI319

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11 December 2017

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