Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2. (Q44372580)

2 December 2017

title

Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2. (English)

1 reference

2 December 2017

Anje Cauwels

1

1 reference

2 December 2017

Ben Janssen

2

1 reference

2 December 2017

Anouk Waeytens

3

1 reference

2 December 2017

Claude Cuvelier

4

1 reference

2 December 2017

Peter Brouckaert

5

1 reference

2 December 2017

language of work or name

English

0 references

publication date

24 March 2003

1 reference

2 December 2017

1 reference

2 December 2017

volume

4

1 reference

2 December 2017

issue

4

1 reference

2 December 2017

page(s)

387-393

1 reference

Pathological aspects of apoptosis in severe sepsis and shock?

2 December 2017

cites work

1 reference

7 January 2021

CD95's deadly mission in the immune system

1 reference

7 January 2021

The biochemistry of apoptosis

1 reference

7 January 2021

Four deaths and a funeral: from caspases to alternative mechanisms.

1 reference

7 January 2021

Signal transduction by tumor necrosis factor and its relatives

1 reference

7 January 2021

Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

1 reference

7 January 2021

More than one way to die: apoptosis, necrosis and reactive oxygen damage

1 reference

7 January 2021

Involvement of oxidants and oxidant-generating enzyme(s) in tumour-necrosis-factor-α-mediated apoptosis: role for lipoxygenase pathway but not mitochondrial respiratory chain

1 reference

7 January 2021

ICE-protease inhibitors block murine liver injury and apoptosis caused by CD95 or by TNF-alpha

1 reference

7 January 2021

TNF-induced enterocyte apoptosis in mice is mediated by the TNF receptor 1 and does not require p53

1 reference

7 January 2021

Systemic injection of a tripeptide inhibits the intracellular activation of CPP32-like proteases in vivo and fully protects mice against Fas-mediated fulminant liver destruction and death

1 reference

7 January 2021

Bioavailability of recombinant tumor necrosis factor determines its lethality in mice

1 reference

7 January 2021

Non-specific effects of methyl ketone peptide inhibitors of caspases

1 reference

7 January 2021

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

1 reference

7 January 2021

Diphenyleneiodonium, an NAD(P)H oxidase inhibitor, also potently inhibits mitochondrial reactive oxygen species production

1 reference

7 January 2021

Effects of aristolochic acid on phospholipase A2 activity and arachidonate metabolism of human neutrophils

1 reference

7 January 2021

Cytosolic phospholipase A2 is required for optimal ATP activation of BK channels in GH(3) cells.

1 reference

7 January 2021

Contrasting effects of cPLA2 on epithelial Na+ transport

1 reference

7 January 2021

Cleavage of human cytosolic phospholipase A2 by caspase-1 (ICE) and caspase-8 (FLICE).

1 reference

7 January 2021

Protection against TNF-induced lethal shock by soluble guanylate cyclase inhibition requires functional inducible nitric oxide synthase

1 reference

7 January 2021

Fas-induced arachidonic acid release is mediated by Ca2+-independent phospholipase A2 but not cytosolic phospholipase A2, which undergoes proteolytic inactivation

1 reference

7 January 2021

Role of caspases in TNF-mediated regulation of cPLA(2).

1 reference

7 January 2021

The Death Domain Kinase RIP Mediates the TNF-Induced NF-κB Signal

1 reference

7 January 2021

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

1 reference

7 January 2021

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

1 reference

7 January 2021

Signal transduction by tumor necrosis factor and gene regulation of the inflammatory cytokine interleukin-6.

1 reference

7 January 2021

Signaling to gene activation and cell death by tumor necrosis factor receptors and Fas.

1 reference

7 January 2021

Oxidative stress interference with the nuclear factor-kappa B activation pathways

1 reference

7 January 2021

Caspase-independent cell killing by Fas-associated protein with death domain

1 reference

7 January 2021

Cathepsin B acts as a dominant execution protease in tumor cell apoptosis induced by tumor necrosis factor

1 reference

7 January 2021

Sensitization to death receptor cytotoxicity by inhibition of fas-associated death domain protein (FADD)/caspase signaling. Requirement of cell cycle progression

1 reference

7 January 2021

Deficiency in caspase-9 or caspase-3 induces compensatory caspase activation

1 reference

7 January 2021

Caspase-2 activation is redundant during seizure-induced neuronal death

1 reference

7 January 2021

Cathepsin B mediates tumor necrosis factor-induced arachidonic acid release in tumor cells

1 reference

7 January 2021

Mice lacking the tumour necrosis factor receptor 1 are resistant to TNF-mediated toxicity but highly susceptible to infection by Listeria monocytogenes

1 reference

7 January 2021

Decreased sensitivity to tumour-necrosis factor but normal T-cell development in TNF receptor-2-deficient mice

1 reference

7 January 2021

Purification and NH2-terminal amino acid sequence of a T-cell-derived lymphokine with growth factor activity for B-cell hybridomas

1 reference

7 January 2021

Analysis of nitrate, nitrite, and [15N]nitrate in biological fluids

1 reference

7 January 2021

Identifiers

DOI

10.1038/NI914

1 reference

2 December 2017

1 reference