NAD(P)H oxidase-derived hydrogen peroxide mediates endothelial nitric oxide production in response to angiotensin II. (Q44177966)

30 November 2017

title

NAD(P)H oxidase-derived hydrogen peroxide mediates endothelial nitric oxide production in response to angiotensin II. (English)

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30 November 2017

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6

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7

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30 November 2017

Hua Cai

1

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30 November 2017

Zongming Li

2

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30 November 2017

Sergey Dikalov

3

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30 November 2017

Steven M Holland

4

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30 November 2017

Jinah Hwang

5

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30 November 2017

David G Harrison

8

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language of work or name

English

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publication date

10 October 2002

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Journal of Biological Chemistry

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number of pages

7

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based on heuristic

inferred from page(s)

published in

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volume

277

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issue

50

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30 November 2017

page(s)

48311-48317

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Expression of functional neutrophil-type NADPH oxidase in cultured rat coronary microvascular endothelial cells.

30 November 2017

cites work

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Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone

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Transcriptional and posttranscriptional regulation of endothelial nitric oxide synthase expression by hydrogen peroxide

21 January 2018

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Hydrogen peroxide activates endothelial nitric-oxide synthase through coordinated phosphorylation and dephosphorylation via a phosphoinositide 3-kinase-dependent signaling pathway.

21 January 2018

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21 January 2018

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Diffusion of nitric oxide in the aorta wall monitored in situ by porphyrinic microsensors.

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Angiotensin II type 2 receptor overexpression activates the vascular kinin system and causes vasodilation

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Selective inhibition of the renal angiotensin type 2 receptor increases blood pressure in conscious rats

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Role of insulin receptor phosphorylation in the insulinomimetic effects of hydrogen peroxide

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Effects of H2O2 on protein tyrosine phosphatase activity in HER14 cells

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Requirement for generation of H2O2 for platelet-derived growth factor signal transduction

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p38 Mitogen-activated protein kinase is a critical component of the redox-sensitive signaling pathways activated by angiotensin II. Role in vascular smooth muscle cell hypertrophy

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c-Src is required for oxidative stress-mediated activation of big mitogen-activated protein kinase 1.

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Src and Cas mediate JNK activation but not ERK1/2 and p38 kinases by reactive oxygen species

21 January 2018

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Reactive oxygen species mediate the activation of Akt/protein kinase B by angiotensin II in vascular smooth muscle cells

21 January 2018

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Activation of protein kinase C by tyrosine phosphorylation in response to H 2 O 2

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p21 as a Common Signaling Target of Reactive Free Radicals and Cellular Redox Stress

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21 January 2018

Identifiers

DOI

10.1074/JBC.M208884200

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30 November 2017

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