NOX2 protects against prolonged inflammation, lung injury, and mortality following systemic insults. (Q42264626)

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29 February 2020

title

NOX2 protects against prolonged inflammation, lung injury, and mortality following systemic insults (English)

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5

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29 February 2020

Steven M Varga

7

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29 February 2020

author name string

Laura C Whitmore

1

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29 February 2020

Brieanna M Hilkin

2

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29 February 2020

Kelli L Goss

3

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29 February 2020

Erin M Wahle

4

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29 February 2020

Paola M Boggiatto

6

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29 February 2020

Francis J Miller

8

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Jessica G Moreland

9

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publication date

27 April 2013

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Journal of Innate Immunity

29 February 2020

published in

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volume

5

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issue

6

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page(s)

565-580

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Interleukin-17-producing gammadelta T cells selectively expand in response to pathogen products and environmental signals

29 February 2020

cites work

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The systemic inflammatory response syndrome

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Expansion of T helper type 17 lymphocytes in patients with chronic granulomatous disease

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NADPH oxidase limits innate immune responses in the lungs in mice

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Antimicrobial actions of the NADPH phagocyte oxidase and inducible nitric oxide synthase in experimental salmonellosis. II. Effects on microbial proliferation and host survival in vivo

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Protective effect of N-acetylcysteine on multiple organ failure induced by zymosan in the rat

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Defective tryptophan catabolism underlies inflammation in mouse chronic granulomatous disease.

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Impaired apoptotic cell clearance in CGD due to altered macrophage programming is reversed by phosphatidylserine-dependent production of IL-4

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Deficiency of NADPH Oxidase Components p47phox and gp91phox Caused Granulomatous Synovitis and Increased Connective Tissue Destruction in Experimental Arthritis Models

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The Effects of Intravenous Antioxidants in Patients With Septic Shock

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Macrophage inflammatory protein-1

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Early activation of gammadelta T lymphocytes in patients with severe systemic inflammatory response syndrome

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Monocyte Chemoattractant Protein 1 Regulates Pulmonary Host Defense via Neutrophil Recruitment during Escherichia coli Infection

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Zymosan-induced generalized inflammation: experimental studies into mechanisms leading to multiple organ dysfunction syndrome

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Hyperinflammation in chronic granulomatous disease and anti-inflammatory role of the phagocyte NADPH oxidase

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Endotoxin priming of neutrophils requires NADPH oxidase-generated oxidants and is regulated by the anion transporter ClC-3.

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N-acetyl-L-cysteine depresses cardiac performance in patients with septic shock

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Spontaneous and aging-dependent development of arthritis in NADPH oxidase 2 deficiency through altered differentiation of CD11b+ and Th/Treg cells

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Influence of N-acetylcysteine on indirect indicators of tissue oxygenation in septic shock patients: results from a prospective, randomized, double-blind study.

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12 December 2020

Critical role of myeloperoxidase and nicotinamide adenine dinucleotide phosphate-oxidase in high-burden systemic infection of mice with Candida albicans

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Toll-like receptor 2 signaling in CD4(+) T lymphocytes promotes T helper 17 responses and regulates the pathogenesis of autoimmune disease.

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Chronic granulomatous disease

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12 December 2020

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

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Targeting granulocyte apoptosis: mechanisms, models, and therapies

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Functional specialization of interleukin-17 family members

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Current Concepts of Hyperinflammation in Chronic Granulomatous Disease

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Genetic deficiency of NADPH oxidase does not diminish, but rather enhances, LPS-induced acute inflammatory responses in vivo

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Resolution-phase macrophages possess a unique inflammatory phenotype that is controlled by cAMP.

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12 December 2020

Genetic ablation of phagocytic NADPH oxidase in mice limits TNFα-induced inflammation in the lungs but not other tissues

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12 December 2020

Identifiers

DOI

10.1159/000347212

1 reference

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29 February 2020

1 reference

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29 February 2020

PubMed publication ID

23635512

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