16S mitochondrial ribosomal RNA degradation is associated with apoptosis. (Q41140269)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9098105%20AND%20SRC:MED&resulttype=core&format=json

2 November 2019

title

16S mitochondrial ribosomal RNA degradation is associated with apoptosis (English)

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2 November 2019

6

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2 November 2019

author name string

D R Crawford

1

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2 November 2019

R J Lauzon

2

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2 November 2019

Y Wang

3

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2 November 2019

J E Mazurkiewicz

4

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2 November 2019

G P Schools

5

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2 November 2019

language of work or name

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1 January 1997

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Free Radical Biology and Medicine

2 November 2019

published in

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2 November 2019

volume

22

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2 November 2019

issue

7

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2 November 2019

page(s)

1295-1300

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Mechanisms and Functions of Cell Death

2 November 2019

cites work

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Hydrogen peroxide induces the expression of adapt15, a novel RNA associated with polysomes in hamster HA-1 cells.

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A rapid and simple method for the isolation of apoptotic DNA fragments.

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Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.

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Commitment to apoptosis is associated with changes in mitochondrial biogenesis and activity in cell lines conditionally immortalized with simian virus 40.

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Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo.

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Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

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Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis

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Induction of a Common Pathway of Apoptosis by Staurosporine

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Gene transfer investigations of p56-LCK function in IL-2-dependent T-cell lines: implications for mechanisms of IL-2-signal transduction

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7 January 2021

Fine mapping of 28S rRNA sites specifically cleaved in cells undergoing apoptosis

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7 January 2021

Bcl-2 functions in an antioxidant pathway to prevent apoptosis

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7 January 2021

Identification of Genes Induced during Apoptosis in T Lymphocytes

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10.1016/S0891-5849(96)00544-8

Identifiers

DOI

1 reference

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2 November 2019

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