3-Nitropropionic acid increases frataxin expression in human lymphoblasts and in transgenic rat PC12 cells (Q40627408)

22 September 2017

title

3-Nitropropionic acid increases frataxin expression in human lymphoblasts and in transgenic rat PC12 cells (English)

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22 September 2017

Mimmo Turano

1

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ORCID Public Data File 2021

author name string

Angela Tammaro

2

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22 September 2017

Irene De Biase

3

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22 September 2017

Maria Simona Lo Casale

4

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22 September 2017

Giuseppina Ruggiero

5

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22 September 2017

Antonella Monticelli

6

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22 September 2017

Sergio Cocozza

7

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22 September 2017

Luigi Pianese

8

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22 September 2017

language of work or name

English

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publication date

1 October 2003

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22 September 2017

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22 September 2017

volume

350

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22 September 2017

issue

3

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22 September 2017

page(s)

184-186

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Modulation of apoptotic regulatory proteins and early activation of cytochrome C following systemic 3-nitropropionic acid administration

22 September 2017

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Regulation of mitochondrial iron accumulation by Yfh1p, a putative homolog of frataxin

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Erythroid differentiation and protoporphyrin IX down-regulate frataxin expression in Friend cells: characterization of frataxin expression compared to molecules involved in iron metabolism and hemoglobinization

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The GAA triplet-repeat expansion in Friedreich ataxia interferes with transcription and may be associated with an unusual DNA structure

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Frataxin is reduced in Friedreich ataxia patients and is associated with mitochondrial membranes

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Friedreich's ataxia: autosomal recessive disease caused by an intronic GAA triplet repeat expansion

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7 January 2021

Two-step processing of human frataxin by mitochondrial processing peptidase. Precursor and intermediate forms are cleaved at different rates

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Deletion of the yeast homologue of the human gene associated with Friedreich's ataxia elicits iron accumulation in mitochondria

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Butyric acid induces apoptosis by up-regulating Bax expression via stimulation of the c-Jun N-terminal kinase/activation protein-1 pathway in human colon cancer cells

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Up-regulation of c-Jun N-terminal kinase pathway in Friedreich's ataxia cells

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Aconitase and mitochondrial iron–sulphur protein deficiency in Friedreich ataxia

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Frataxin deficiency enhances apoptosis in cells differentiating into neuroectoderm

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Upregulation of expression from the FRDA genomic locus for the therapy of Friedreich ataxia

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Decreased expression of bcl-2 and bcl-x mRNA coincides with apoptosis following intracerebral administration of 3-nitropropionic acid

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Respiratory deficiency due to loss of mitochondrial DNA in yeast lacking the frataxin homologue

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10.1016/S0304-3940(03)00906-6

Identifiers

DOI

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22 September 2017

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