Hepatitis C virus-induced oxidative stress suppresses hepcidin expression through increased histone deacetylase activity (Q39955308)

15 September 2017

title

Hepatitis C virus-induced oxidative stress suppresses hepcidin expression through increased histone deacetylase activity (English)

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15 September 2017

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David A Brenner

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Bernd Schnabl

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4

object named as

Bernd Schnabl

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15 September 2017

Kouichi Miura

1

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15 September 2017

Kojiro Taura

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2

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15 September 2017

Yuzo Kodama

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3

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15 September 2017

language of work or name

English

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publication date

1 November 2008

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15 September 2017

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15 September 2017

volume

48

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15 September 2017

issue

5

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15 September 2017

page(s)

1420-1429

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The prevalence of hepatitis C virus infection in the United States, 1999 through 2002

15 September 2017

cites work

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Successful interferon therapy reverses enhanced hepatic iron accumulation and lipid peroxidation in chronic hepatitis C

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Hepatic iron overload induces hepatocellular carcinoma in transgenic mice expressing the hepatitis C virus polyprotein

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Hepcidin antimicrobial peptide transgenic mice exhibit features of the anemia of inflammation

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Targeted disruption of the hepcidin 1 gene results in severe hemochromatosis

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Regulation of iron metabolism by hepcidin

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7 January 2021

Hepcidin expression in the liver: relatively low level in patients with chronic hepatitis C

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7 January 2021

Regulatory failure of serum prohepcidin levels in patients with hepatitis C.

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7 January 2021

Hepatitis C virus-induced reactive oxygen species raise hepatic iron level in mice by reducing hepcidin transcription

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7 January 2021

ROS mediate the hypoxic repression of the hepcidin gene by inhibiting C/EBPalpha and STAT-3.

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7 January 2021

Alcohol metabolism-mediated oxidative stress down-regulates hepcidin transcription and leads to increased duodenal iron transporter expression

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7 January 2021

Regulation of iron homeostasis by the hypoxia-inducible transcription factors (HIFs).

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7 January 2021

Hepatitis C virus core protein increases mitochondrial ROS production by stimulation of Ca2+ uniporter activity.

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7 January 2021

Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein

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7 January 2021

Highly permissive cell lines for subgenomic and genomic hepatitis C virus RNA replication

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7 January 2021

Hepatitis C virus core and nonstructural proteins induce fibrogenic effects in hepatic stellate cells

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Histone deacetylase inhibitors: molecular mechanisms of action

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Hypoxia-Inducible Factor-1 (HIF-1)

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Hypoxia down-regulates CCAAT/enhancer binding protein-alpha expression in breast cancer cells

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Mitochondrial reactive oxygen species control the transcription factor CHOP-10/GADD153 and adipocyte differentiation: a mechanism for hypoxia-dependent effect

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STAT3 is required for IL-6-gp130-dependent activation of hepcidin in vivo.

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7 January 2021

Insulin resistance and liver injury in hepatitis C is not associated with virus-specific changes in adipocytokines

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7 January 2021

Mechanisms of liver injury. III. Oxidative stress in the pathogenesis of hepatitis C virus

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7 January 2021

Oxidative stress, chromatin remodeling and gene transcription in inflammation and chronic lung diseases

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High levels of oxidative stress globally inhibit gene transcription and histone acetylation

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7 January 2021

Class II histone deacetylases are associated with VHL-independent regulation of hypoxia-inducible factor 1 alpha

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7 January 2021

Histone deacetylase 7 associates with hypoxia-inducible factor 1alpha and increases transcriptional activity

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7 January 2021

Hepatitis C Virus Stabilizes Hypoxia-Inducible Factor 1 and Stimulates the Synthesis of Vascular Endothelial Growth Factor

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Risk and management of blood-borne infections in health care workers

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Antioxidant therapy for chronic hepatitis C after failure of interferon: results of phase II randomized, double-blind placebo controlled clinical trial

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A histone deacetylase inhibitor, trichostatin A, suppresses myofibroblastic differentiation of rat hepatic stellate cells in primary culture

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7 January 2021

Identifiers

DOI

10.1002/HEP.22486

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15 September 2017

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