Long-term inactivation particle for voltage-gated sodium channels (Q39672545)

13 September 2017

title

Long-term inactivation particle for voltage-gated sodium channels (English)

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13 September 2017

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Egidio D'Angelo

3

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author name string

Katarzyna Dover

series ordinal

1

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13 September 2017

Sergio Solinas

series ordinal

2

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13 September 2017

Mitchell Goldfarb

series ordinal

4

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13 September 2017

language of work or name

English

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publication date

2 August 2010

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13 September 2017

published in

Journal of Physiology

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13 September 2017

volume

588

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13 September 2017

issue

Pt 19

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13 September 2017

page(s)

3695-3711

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From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

13 September 2017

cites work

1 reference

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Axonal Na+ channels ensure fast spike activation and back-propagation in cerebellar granule cells

21 January 2018

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Block of brain sodium channels by peptide mimetics of the isoleucine, phenylalanine, and methionine (IFM) motif from the inactivation gate

21 January 2018

1 reference

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Restoration of inactivation and block of open sodium channels by an inactivation gate peptide

21 January 2018

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Interaction between fast and slow inactivation in Skm1 sodium channels

21 January 2018

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21 January 2018

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Crystal Structure of a Fibroblast Growth Factor Homologous Factor (FHF) Defines a Conserved Surface on FHFs for Binding and Modulation of Voltage-gated Sodium Channels

21 January 2018

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21 January 2018

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Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels

21 January 2018

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Open-channel block by the cytoplasmic tail of sodium channel beta4 as a mechanism for resurgent sodium current

21 January 2018

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FGF14 N-terminal splice variants differentially modulate Nav1.2 and Nav1.6-encoded sodium channels

21 January 2018

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Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B

21 January 2018

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Voltage sensor of Kv1.2: structural basis of electromechanical coupling

21 January 2018

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Fibroblast growth factor 14 is an intracellular modulator of voltage-gated sodium channels

21 January 2018

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Closing in on the resting state of the Shaker K(+) channel.

21 January 2018

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Inactivation and recovery of sodium currents in cerebellar Purkinje neurons: evidence for two mechanisms

21 January 2018

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Activity-dependent control of neuronal output by local and global dendritic spike attenuation

21 January 2018

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Differential modulation of sodium channel Na(v)1.6 by two members of the fibroblast growth factor homologous factor 2 subfamily

21 January 2018

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Fibroblast growth factor homologous factors are intracellular signaling proteins

21 January 2018

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FGF14 regulates the intrinsic excitability of cerebellar Purkinje neurons

21 January 2018

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21 January 2018

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Interaction between the sodium channel inactivation linker and domain III S4-S5.

21 January 2018

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Neurotrophin modulation of voltage-gated potassium channels in rat through TrkB receptors is time and sensory experience dependent

21 January 2018

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Sodium channel inactivation: molecular determinants and modulation

21 January 2018

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Subcellular and developmental expression of alternatively spliced forms of fibroblast growth factor 14.

21 January 2018

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A cluster of hydrophobic amino acid residues required for fast Na(+)-channel inactivation

21 January 2018

1 reference

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Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons

21 January 2018

1 reference

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21 January 2018

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Prolonged Sodium Channel Inactivation Contributes to Dendritic Action Potential Attenuation in Hippocampal Pyramidal Neurons

1 October 2018

1 reference

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Resurgent sodium current and action potential formation in dissociated cerebellar Purkinje neurons.

1 October 2018

1 reference

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Neurotrophins protect cultured cerebellar granule neurons against the early phase of cell death by a two-component mechanism

1 October 2018

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Amino acid residues required for fast Na(+)-channel inactivation: charge neutralizations and deletions in the III-IV linker

1 October 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2998221

Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na+ channel disrupt fast and enhance slow inactivation

1 October 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/20679355

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1113/JPHYSIOL.2010.192559

1 reference

13 September 2017

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