STAT-3 activation is required for normal G-CSF-dependent proliferation and granulocytic differentiation (Q38303450)

1 September 2017

title

STAT-3 activation is required for normal G-CSF-dependent proliferation and granulocytic differentiation (English)

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1 September 2017

McLemore ML

1

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1 September 2017

Grewal S

2

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1 September 2017

Liu F

3

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1 September 2017

Archambault A

4

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1 September 2017

Poursine-Laurent J

5

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1 September 2017

Haug J

6

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1 September 2017

Link DC

7

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1 September 2017

language of work or name

English

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publication date

1 February 2001

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1 September 2017

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1 September 2017

volume

14

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1 September 2017

issue

2

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1 September 2017

page(s)

193-204

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The effect of GM-CSF and G-CSF on human neutrophil function

1 September 2017

cites work

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Constitutive activation of Stat3 signaling confers resistance to apoptosis in human U266 myeloma cells

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Stat3 and G-CSF-induced myeloid differentiation

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STAT3-mediated differentiation and survival and of myeloid cells in response to granulocyte colony-stimulating factor: role for the cyclin-dependent kinase inhibitor p27(Kip1)

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Distinct cytoplasmic regions of the human granulocyte colony-stimulating factor receptor involved in induction of proliferation and maturation

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Growth and differentiation signals mediated by different regions in the cytoplasmic domain of granulocyte colony-stimulating factor receptor

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7 January 2021

Activated Stat Related Transcription Factors in Acute Leukemia

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Sustained receptor activation and hyperproliferation in response to granulocyte colony-stimulating factor (G-CSF) in mice with a severe congenital neutropenia/acute myeloid leukemia-derived mutation in the G-CSF receptor gene.

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Effects of in vivo administration of G-CSF on neutrophil functions in healthy volunteers

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Analysis of mice containing a targeted deletion of beta-globin locus control region 5' hypersensitive site 3.

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Impaired production and increased apoptosis of neutrophils in granulocyte colony-stimulating factor receptor-deficient mice

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The kinetics of human granulopoiesis following treatment with granulocyte colony-stimulating factor in vivo.

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Myeloblastin is a granulocyte colony-stimulating factor-responsive gene conferring factor-independent growth to hematopoietic cells

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7 January 2021

Increased granulocyte colony-stimulating factor responsiveness but normal resting granulopoiesis in mice carrying a targeted granulocyte colony-stimulating factor receptor mutation derived from a patient with severe congenital neutropenia

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A central role for Stat3 in IL-6-induced regulation of growth and differentiation in M1 leukemia cells

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Binding of 125I-labeled granulocyte colony-stimulating factor to normal murine hemopoietic cells

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Use of the green fluorescent protein as a marker to identify and track genetically modified hematopoietic cells

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A role for G-CSF receptor signaling in the regulation of hematopoietic cell function but not lineage commitment or differentiation

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Involvement of STAT3 in the Granulocyte Colony-stimulating Factor-induced Differentiation of Myeloid Cells

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Control of myeloid differentiation and survival by Stats

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STAT signaling in head and neck cancer

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Targeted disruption of the mouse Stat3 gene leads to early embryonic lethality

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Enhanced Th1 activity and development of chronic enterocolitis in mice devoid of Stat3 in macrophages and neutrophils.

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7 January 2021

Stat5a and Stat5b proteins have essential and nonessential, or redundant, roles in cytokine responses

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7 January 2021

Mutant protein of recombinant human granulocyte colony-stimulating factor for receptor binding assay

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DNA-mediated transfer of the adenine phosphoribosyltransferase locus into mammalian cells

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Distinct Regions of the Human Granulocyte-Colony-Stimulating Factor Receptor Cytoplasmic Domain Are Required for Proliferation and Gene Induction

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7 January 2021

10.1016/S1074-7613(01)00101-7

Identifiers

DOI

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1 September 2017

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