Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury (Q38244750)

31 August 2017

title

Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury (English)

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31 August 2017

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8

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31 August 2017

Carlotta Giorgi

2

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Rita Pavasini

Rita Pavasini

5

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Gianluca Campo

Gianluca Campo

7

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Mariusz R Wieckowski

Mariusz R Wieckowski

6

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Giampaolo Morciano

1

Giampaolo Morciano

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31 August 2017

3

Massimo Bonora

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31 August 2017

4

Silvia Punzetti

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31 August 2017

publication date

27 August 2014

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Journal of Molecular and Cellular Cardiology

31 August 2017

published in

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31 August 2017

volume

78

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31 August 2017

page(s)

142-153

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The worldwide environment of cardiovascular disease: prevalence, diagnosis, therapy, and policy issues: a report from the American College of Cardiology

31 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Prognostic impact of hospital readmissions after primary percutaneous coronary intervention

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Postconditioning against ischaemia-reperfusion injury: ready for wide application in patients?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Myocardial reperfusion injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Medical and cellular implications of stunning, hibernation, and preconditioning: an NHLBI workshop.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The Ca2+-induced membrane transition in mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

A heart mitochondrial Ca2(+)-dependent pore of possible relevance to re-perfusion-induced injury. Evidence that ADP facilitates pore interconversion between the closed and open states

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial permeability transition pore may comprise VDAC molecules

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial permeability transition pore may comprise VDAC molecules

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Modulation of Ca2+ efflux from heart mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Involvement of the ADP/ATP carrier in calcium-induced perturbations of the mitochondrial inner membrane permeability: importance of the orientation of the nucleotide binding site

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclosporin A-sensitive and insensitive mechanisms produce the permeability transition in mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial ADP/ATP carrier can be reversibly converted into a large channel by Ca2+

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Reconstituted adenine nucleotide translocase forms a channel for small molecules comparable to the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Complexes between kinases, mitochondrial porin and adenylate translocator in rat brain resemble the permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Physiologic functions of cyclophilin D and the mitochondrial permeability transition pore.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Involvement of Cyclophilin D in the Activation of A mitochondrial Pore by Ca2+ and Oxidant Stress

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cysteine 203 of Cyclophilin D Is Critical for Cyclophilin D Activation of the Mitochondrial Permeability Transition Pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

ATP synthesis and storage

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial phosphate carrier interacts with cyclophilin D and may play a key role in the permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

A high-throughput screening for mammalian cell death effectors identifies the mitochondrial phosphate carrier as a regulator of cytochrome c release.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The reversible antiport-uniport conversion of the phosphate carrier from yeast mitochondria depends on the presence of a single cysteine

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The roles of phosphate and the phosphate carrier in the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Genetic manipulation of the cardiac mitochondrial phosphate carrier does not affect permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Isolation of the mitochondrial benzodiazepine receptor: association with the voltage-dependent anion channel and the adenine nucleotide carrier

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Regulation of the inner membrane mitochondrial permeability transition by the outer membrane translocator protein (peripheral benzodiazepine receptor)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Peripheral benzodiazepine receptor ligand, PK11195 induces mitochondria cytochrome c release and dissipation of mitochondria potential via induction of mitochondria permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Effect of peripheral benzodiazepine receptor (PBR/TSPO) ligands on opening of Ca2+-induced pore and phosphorylation of 3.5-kDa polypeptide in rat brain mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Specific ligands of the peripheral benzodiazepine receptor induce apoptosis and cell cycle arrest in human colorectal cancer cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Targeting mitochondria for cancer therapy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Resveratrol-induced apoptosis is enhanced in acute lymphoblastic leukemia cells by modulation of the mitochondrial permeability transition pore.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bax directly induces release of cytochrome c from isolated mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Movement of Bax from the Cytosol to Mitochondria during Apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bad targets the permeability transition pore independent of Bax or Bak to switch between Ca2+-dependent cell survival and death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mediation of the antiapoptotic activity of Bcl-xL protein upon interaction with VDAC1 protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Inhibition of GSK-3beta as a target for cardioprotection: the importance of timing, location, duration and degree of inhibition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

N6-(3-iodobenzyl)-adenosine-5'-N-methylcarboxamide confers cardioprotection at reperfusion by inhibiting mitochondrial permeability transition pore opening via glycogen synthase kinase 3 beta

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cardioprotection of the aged rat heart by GSK-3β inhibitor is attenuated: age-related changes in mitochondrial permeability transition pore modulation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Direct evidence for inhibition of mitochondrial permeability transition pore opening by sevoflurane preconditioning in cardiomyocytes: comparison with cyclosporine A.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Glycogen synthase kinase 3 inhibition slows mitochondrial adenine nucleotide transport and regulates voltage-dependent anion channel phosphorylation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Protein kinase C and preconditioning: role of the sarcoplasmic reticulum

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Anti-apoptotic effect of cGMP in cultured astrocytes: inhibition by cGMP-dependent protein kinase of mitochondrial permeable transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

p53 opens the mitochondrial permeability transition pore to trigger necrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Complement 1q-binding protein inhibits the mitochondrial permeability transition pore and protects against oxidative stress-induced death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Regulatory mechanisms of proton-translocating F(O)F (1)-ATP synthase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Redox regulation of mitochondrial ATP synthase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial ATP synthasome. Cristae-enriched membranes and a multiwell detergent screening assay yield dispersed single complexes containing the ATP synthase and carriers for Pi and ADP/ATP.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Structural organization of mitochondrial ATP synthase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclophilin D modulates mitochondrial F0F1-ATP synthase by interacting with the lateral stalk of the complex

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Oligomycin, inhibitor of the F0 part of H+-ATP-synthase, suppresses the TNF-induced apoptosis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Role of the c subunit of the FO ATP synthase in mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial permeability transition pore is a dispensable element for mitochondrial calcium efflux

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Secondary structure composition of reconstituted subunit b of the Escherichia coli ATP synthase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The transmembrane domain of subunit b of the Escherichia coli F1F(O) ATP synthase is sufficient for H(+)-translocating activity together with subunits a and c.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

A 0.1–700 Hz current through a voltage-clamped pore: candidate protein for initiator of neural oscillations

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Phosphorylation of a peptide related to subunit c of the F0F1-ATPase/ATP synthase and relationship to permeability transition pore opening in mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Potential role of subunit c of F0F1-ATPase and subunit c of storage body in the mitochondrial permeability transition. Effect of the phosphorylation status of subunit c on pore opening

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Dimers of mitochondrial ATP synthase form the permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Assembly and oligomerization of human ATP synthase lacking mitochondrial subunits a and A6L.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Regulation of mitochondrial structure and function by the F1Fo-ATPase inhibitor protein, IF1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Age-dependent dissociation of ATP synthase dimers and loss of inner-membrane cristae in mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

New horizons in cardioprotection: recommendations from the 2010 National Heart, Lung, and Blood Institute Workshop

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondria: more than just a powerhouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The role of cell death in heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Apoptosis: basic mechanisms and implications for cardiovascular disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Pathophysiology of myocardial reperfusion injury: preconditioning, postconditioning, and translational aspects of protective measures

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Evidence for the presence of a reversible Ca2+-dependent pore activated by oxidative stress in heart mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Apoptosis and myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Cyclosporine protects the heart during aortic valve surgery.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Apoptosis in human acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial membrane permeabilization in cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Involvement of CD95/Apo1/Fas in Cell Death After Myocardial Ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Tumor necrosis factor-alpha and tumor necrosis factor receptors in the failing human heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Tumor necrosis factor alpha-induced apoptosis in cardiac myocytes. Involvement of the sphingolipid signaling cascade in cardiac cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Nafazatrom-induced salvage of ischemic myocardium in anesthetized dogs is mediated through inhibition of neutrophil function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

A randomized, double-blinded, placebo-controlled multicenter trial of adenosine as an adjunct to reperfusion in the treatment of acute myocardial infarction (AMISTAD-II).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial control of cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial Ca(2+) and apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial apoptotic pathway is activated by serum and glucose deprivation in cardiac myocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Reperfusion injury induces apoptosis in rabbit cardiomyocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Differential contribution of necrosis and apoptosis in myocardial ischemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Quantitation of acute necrosis after experimental myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The variable morphological coexistence of apoptosis and necrosis in human myocardial infarction: significance for understanding its pathogenesis, clinical course, diagnosis and prognosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondria-ros crosstalk in the control of cell death and aging

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The role of oxidants and free radicals in reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Ca2+ signaling, mitochondria and cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

beta-Adrenergic pathway induces apoptosis through calcineurin activation in cardiac myocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial permeability transition pore and calcium handling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Role of the mitochondrial permeability transition in myocardial disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The mitochondrial permeability transition pore and ischemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

An update on cardioprotection: a review of the latest adjunctive therapies to limit myocardial infarction size in clinical trials

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Effect of cyclosporine on reperfusion injury in acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The effect of cyclosporin-A on peri-operative myocardial injury in adult patients undergoing coronary artery bypass graft surgery: a randomised controlled clinical trial

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transition.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Rationale and Design of the MITOCARE Study: A Phase II, Multicenter, Randomized, Double-Blind, Placebo-Controlled Study to Assess the Safety and Efficacy of TRO40303 for the Reduction of Reperfusion Injury in Patients Undergoing Percutaneous Coro

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Exenatide reduces final infarct size in patients with ST-segment-elevation myocardial infarction and short-duration of ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Atrial natriuretic peptide prevents the mitochondrial permeability transition pore opening by inactivating glycogen synthase kinase 3β via PKG and PI3K in cardiac H9c2 cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Reperfusion injury salvage kinase and survivor activating factor enhancement prosurvival signaling pathways in ischemic postconditioning: two sides of the same coin

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Optimising cardioprotection during myocardial ischaemia: targeting potential intracellular pathways with glucagon-like peptide-1

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Out-of-hospital administration of intravenous glucose-insulin-potassium in patients with suspected acute coronary syndromes: the IMMEDIATE randomized controlled trial

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Rationale and design of the EMBRACE STEMI study: a phase 2a, randomized, double-blind, placebo-controlled trial to evaluate the safety, tolerability and efficacy of intravenous Bendavia on reperfusion injury in patients treated with standard therapy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The salvaged area at risk in reperfused acute myocardial infarction as visualized by cardiovascular magnetic resonance

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

The use of contrast-enhanced magnetic resonance imaging to identify reversible myocardial dysfunction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Human atrial natriuretic peptide and nicorandil as adjuncts to reperfusion treatment for acute myocardial infarction (J-WIND): two randomised trials

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

Efficacy of high-dose atorvastatin loading before primary percutaneous coronary intervention in ST-segment elevation myocardial infarction: the STATIN STEMI trial.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

10.1016/J.YJMCC.2014.08.015

7 January 2021

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DOI

1 reference

31 August 2017

1 reference