Recombinant adeno-associated virus-mediated high-efficiency, transient expression of the murine cationic amino acid transporter (ecotropic retroviral receptor) permits stable transduction of human HeLa cells by ecotropic retroviral vectors. (Q35869915)

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scientific article published in October 1996

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English	Recombinant adeno-associated virus-mediated high-efficiency, transient expression of the murine cationic amino acid transporter (ecotropic retroviral receptor) permits stable transduction of human HeLa cells by ecotropic retroviral vectors.	scientific article published in October 1996

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scholarly article

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19 October 2019

Recombinant adeno-associated virus-mediated high-efficiency, transient expression of the murine cationic amino acid transporter (ecotropic retroviral receptor) permits stable transduction of human HeLa cells by ecotropic retroviral vectors (English)

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19 October 2019

vector-borne disease

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J Bertran

1

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19 October 2019

J L Miller

2

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19 October 2019

Y Yang

3

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19 October 2019

A Fenimore-Justman

4

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19 October 2019

F Rueda

5

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19 October 2019

E F Vanin

6

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19 October 2019

A W Nienhuis

7

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19 October 2019

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1 October 1996

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19 October 2019

Journal of Virology

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19 October 2019

70

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19 October 2019

10

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19 October 2019

6759-6766

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19 October 2019

High-efficiency transfer of the T cell co-stimulatory molecule B7-2 to lymphoid cells using high-titer recombinant adeno-associated virus vectors

1 reference

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Second-strand synthesis is a rate-limiting step for efficient transduction by recombinant adeno-associated virus vectors

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Cell lines for the production of recombinant adeno-associated virus

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Transduction with recombinant adeno-associated virus for gene therapy is limited by leading-strand synthesis

1 reference

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Envelope-binding domain in the cationic amino acid transporter determines the host range of ecotropic murine retroviruses.

1 reference

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Use of retroviral vectors for gene transfer and expression

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Retroviral infection and expression of cationic amino acid transporters in rodent hepatocytes.

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Adeno-associated virus vectors preferentially transduce cells in S phase.

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Efficient gene transfer into nondividing cells by adeno-associated virus-based vectors.

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DNA-damaging agents greatly increase the transduction of nondividing cells by adeno-associated virus vectors.

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Adeno-associated virus vector gene expression occurs in nondividing cells in the absence of vector DNA integration

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Phenotypic correction of Fanconi anemia in human hematopoietic cells with a recombinant adeno-associated virus vector

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Asymmetric replication in vitro from a human sequence element is dependent on adeno-associated virus Rep protein

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Long-term gene expression and phenotypic correction using adeno-associated virus vectors in the mammalian brain

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DNA synthesis and topoisomerase inhibitors increase transduction by adeno-associated virus vectors

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Gene marking after bone marrow transplantation.

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