PML-nuclear bodies decrease with age and their stress response is impaired in aged individuals. (Q35138015)

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12 March 2020

title

PML-nuclear bodies decrease with age and their stress response is impaired in aged individuals (English)

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12 March 2020

4

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Luitpold Distel

7

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12 March 2020

5

Manfred Schmidt

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12 March 2020

author name string

Barbara Wenger

1

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12 March 2020

Manuela Schwegler

2

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Maria Brunner

3

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Rainer Fietkau

6

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publication date

2 April 2014

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12 March 2020

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volume

14

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page(s)

42

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Identification of new therapeutic targets by genome-wide analysis of gene expression in the ipsilateral cortex of aged rats after stroke

12 March 2020

cites work

1 reference

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Age-dependent down-regulation of DNA polymerase δ1 in human lymphocytes

13 July 2018

1 reference

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Prolonged gaseous hypothermia prevents the upregulation of phagocytosis-specific protein annexin 1 and causes low-amplitude EEG activity in the aged rat brain after cerebral ischemia

13 July 2018

1 reference

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A senescent cell bystander effect: senescence-induced senescence

13 July 2018

1 reference

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Promyelocytic leukemia nuclear bodies support a late step in DNA double-strand break repair by homologous recombination

13 July 2018

1 reference

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Structural basis of γH2AX recognition by human PTIP BRCT5-BRCT6 domains in the DNA damage response pathway

13 July 2018

1 reference

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Functional connection between Rad51 and PML in homology-directed repair

13 July 2018

1 reference

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Detailed Analysis of DNA Repair and Senescence Marker Kinetics Over the Life Span of a Human Fibroblast Cell Line

13 July 2018

1 reference

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γ-H2AX as a biomarker of DNA damage induced by ionizing radiation in human peripheral blood lymphocytes and artificial skin

13 July 2018

1 reference

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Structure, dynamics and functions of promyelocytic leukaemia nuclear bodies

13 July 2018

1 reference

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Cellular senescence: when bad things happen to good cells

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR.

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

What has senescence got to do with cancer?

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

PML nuclear bodies: dynamic sensors of DNA damage and cellular stress

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

Pondering the promyelocytic leukemia protein (PML) puzzle: possible functions for PML nuclear bodies

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

PML is induced by oncogenic ras and promotes premature senescence

13 July 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3992156

Delayed kinetics of DNA double-strand break processing in normal and pathological aging

13 July 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/24694011

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1186/1471-2318-14-42

1 reference

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Dimensions Publication ID

12 March 2020

0 references

1 reference

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12 March 2020

PubMed publication ID

24694011

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24694011%20AND%20SRC:MED&resulttype=core&format=json