Constitutive expression of aryl hydrocarbon receptor in keratinocytes causes inflammatory skin lesions (Q34097422)

31 July 2017

title

Constitutive expression of aryl hydrocarbon receptor in keratinocytes causes inflammatory skin lesions (English)

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31 July 2017

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Masafumi Tauchi

1

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31 July 2017

Azumi Hida

2

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31 July 2017

Takaaki Negishi

3

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31 July 2017

Fumiki Katsuoka

4

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31 July 2017

Shuhei Noda

5

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31 July 2017

Junsei Mimura

6

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31 July 2017

Tomonori Hosoya

7

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31 July 2017

Akinori Yanaka

8

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31 July 2017

Hiroyuki Aburatani

9

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31 July 2017

Yoshiaki Fujii-Kuriyama

10

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31 July 2017

Hozumi Motohashi

11

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31 July 2017

Masayuki Yamamoto

12

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31 July 2017

language of work or name

English

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publication date

1 November 2005

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Molecular and Cellular Biology

31 July 2017

published in

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31 July 2017

volume

25

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31 July 2017

issue

21

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31 July 2017

page(s)

9360-9368

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Risk factors for asthma and atopy

31 July 2017

cites work

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5 July 2018

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5 July 2018

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5 July 2018

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IL-18 contributes to the spontaneous development of atopic dermatitis-like inflammatory skin lesion independently of IgE/stat6 under specific pathogen-free conditions

5 July 2018

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5 July 2018

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Differentiated keratinocytes are responsible for TNF-alpha regulated production of macrophage inflammatory protein 3alpha/CCL20, a potent chemokine for Langerhans cells

5 July 2018

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5 July 2018

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Inducible expression of a CC chemokine liver- and activation-regulated chemokine (LARC)/macrophage inflammatory protein (MIP)-3 alpha/CCL20 by epidermal keratinocytes and its role in atopic dermatitis

5 July 2018

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5 July 2018

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5 July 2018

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Skin innate immune system in psoriasis: friend or foe?

5 July 2018

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Pathogenesis of atopic dermatitis

5 July 2018

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The role of diesel exhaust particles and their associated polyaromatic hydrocarbons in the induction of allergic airway disease

5 July 2018

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5 July 2018

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Chloracne. Some recent issues

5 July 2018

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Effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on interleukin-4-mediated mechanisms of immunity

5 July 2018

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Role of alterations in Ca(2+)-associated signaling pathways in the immunotoxicity of polycyclic aromatic hydrocarbons

5 July 2018

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Targeting expression of keratinocyte growth factor to keratinocytes elicits striking changes in epithelial differentiation in transgenic mice

5 July 2018

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5 July 2018

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5 July 2018

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Constitutively active aryl hydrocarbon receptor expressed specifically in T-lineage cells causes thymus involution and suppresses the immunization-induced increase in splenocytes

5 July 2018

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A constitutively active dioxin/aryl hydrocarbon receptor promotes hepatocarcinogenesis in mice

28 October 2018

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Persistent suppression of contact hypersensitivity, and altered T-cell parameters in F344 rats exposed perinatally to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).

28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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Aromatic hydrocarbon receptor-driven Bax gene expression is required for premature ovarian failure caused by biohazardous environmental chemicals

28 October 2018

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28 October 2018

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12 December 2020

Enhanced human IgE production results from exposure to the aromatic hydrocarbons from diesel exhaust: direct effects on B-cell IgE production

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12 December 2020

Loss of teratogenic response to 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin (TCDD) in mice lacking the Ah (dioxin) receptor

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12 December 2020

Split tolerance to a viral antigen expressed in thymic epithelium and keratinocytes

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12 December 2020

Immunological Findings in a Group of Coke-Oven Workers Exposed to Polycyclic Aromatic Hydrocarbons

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12 December 2020

Profiles of cytokine mRNAs in the skin and lymph nodes of SENCAR mice treated epicutaneously with dibenzo[a,l]pyrene or dimethylbenz[a]anthracene reveal a direct correlation between carcinogen-induced contact hypersensitivity and epidermal hyperplas

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12 December 2020

Organic compounds from diesel exhaust particles elicit a proinflammatory response in human airway epithelial cells and induce cytochrome p450 1A1 expression

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12 December 2020

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12 December 2020

10.1128/MCB.25.21.9360-9368.2005

Identifiers

DOI

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31 July 2017

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31 July 2017

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