Proteomic analysis of Nrf2 deficient transgenic mice reveals cellular defence and lipid metabolism as primary Nrf2-dependent pathways in the liver. (Q33939951)

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English	Proteomic analysis of Nrf2 deficient transgenic mice reveals cellular defence and lipid metabolism as primary Nrf2-dependent pathways in the liver.	scientific article

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31 January 2020

Proteomic analysis of Nrf2 deficient transgenic mice reveals cellular defence and lipid metabolism as primary Nrf2-dependent pathways in the liver (English)

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31 January 2020

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31 January 2020

Laura E. Randle

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31 January 2020

Rosalind Jenkins

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31 January 2020

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31 January 2020

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Neil R Kitteringham

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31 January 2020

Azman Abdullah

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31 January 2020

Rowena Sison

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31 January 2020

Christopher E P Goldring

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31 January 2020

Helen Powell

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31 January 2020

Christopher Sanderson

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31 January 2020

Samantha Williams

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31 January 2020

Larry Higgins

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31 January 2020

Masayuki Yamamoto

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31 January 2020

B Kevin Park

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31 January 2020

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24 April 2010

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31 January 2020

Journal of Proteomics

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31 January 2020

73

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31 January 2020

8

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31 January 2020

1612-1631

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31 January 2020

Plenary Lecture 2: Transcription factors, regulatory elements and nutrient-gene communication

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Genetic versus chemoprotective activation of Nrf2 signaling: overlapping yet distinct gene expression profiles between Keap1 knockout and triterpenoid-treated mice.

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Pyrazole induced oxidative liver injury independent of CYP2E1/2A5 induction due to Nrf2 deficiency

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The antioxidant/electrophile response element motif

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28 July 2018

Regulatory sequence analysis tools

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Identification of a novel Nrf2-regulated antioxidant response element (ARE) in the mouse NAD(P)H:quinone oxidoreductase 1 gene: reassessment of the ARE consensus sequence

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28 July 2018

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An important function of Nrf2 in combating oxidative stress: detoxification of acetaminophen

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28 July 2018

Nrf2-dependent activation of the antioxidant responsive element by tert-butylhydroquinone is independent of oxidative stress in IMR-32 human neuroblastoma cells

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28 July 2018

High sensitivity of Nrf2 knockout mice to acetaminophen hepatotoxicity associated with decreased expression of ARE-regulated drug metabolizing enzymes and antioxidant genes

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Two-dimensional database of mouse liver proteins: changes in hepatic protein levels following treatment with acetaminophen or its nontoxic regioisomer 3-acetamidophenol

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Mechanism of heme oxygenase-1 gene activation by cadmium in MCF-7 mammary epithelial cells. Role of p38 kinase and Nrf2 transcription factor

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The Nrf2 transcription factor contributes both to the basal expression of glutathione S-transferases in mouse liver and to their induction by the chemopreventive synthetic antioxidants, butylated hydroxyanisole and ethoxyquin

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Transcriptional regulation of the antioxidant response element. Activation by Nrf2 and repression by MafK.

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An Nrf2/Small Maf Heterodimer Mediates the Induction of Phase II Detoxifying Enzyme Genes through Antioxidant Response Elements

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Constitutive and beta-naphthoflavone-induced expression of the human gamma-glutamylcysteine synthetase heavy subunit gene is regulated by a distal antioxidant response element/TRE sequence

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The rat quinone reductase antioxidant response element. Identification of the nucleotide sequence required for basal and inducible activity and detection of antioxidant response element-binding proteins in hepatoma and non-hepatoma cell lines

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Carcinogenesis by chemicals: an overview--G. H. A. Clowes memorial lecture

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Transcriptional regulation of the rat glutathione S-transferase Ya subunit gene. Characterization of a xenobiotic-responsive element controlling inducible expression by phenolic antioxidants

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ATP-citrate lyase links cellular metabolism to histone acetylation

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12 December 2020

based on heuristic

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Metabolic activation of benzo(a)pyrene proceeds by a diol-epoxide

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12 December 2020

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Induction of Phase I and Phase II Drug-Metabolizing Enzyme mRNA, Protein, and Activity by BHA, Ethoxyquin, and Oltipraz

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12 December 2020

based on heuristic

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10.1016/J.JPROT.2010.03.018

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Europe PubMed Central

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31 January 2020

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31 January 2020

PubMed publication ID

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31 January 2020

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