CD8+ T cells and IFN-γ mediate the time-dependent accumulation of infected red blood cells in deep organs during experimental cerebral malaria. (Q33873437)

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English	CD8+ T cells and IFN-γ mediate the time-dependent accumulation of infected red blood cells in deep organs during experimental cerebral malaria.	scientific article

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28 January 2020

CD8+ T cells and IFN-γ mediate the time-dependent accumulation of infected red blood cells in deep organs during experimental cerebral malaria (English)

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28 January 2020

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cerebral malaria

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1

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28 January 2020

Benoit Malleret

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Shanshan W. Howland

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28 January 2020

Florent Ginhoux

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Florent Ginhoux

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Laurent Rénia

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Alicia Yoke Wei Wong

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28 January 2020

Zi Wei Chang

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11 April 2011

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28 January 2020

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28 January 2020

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28 January 2020

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Creative Commons Attribution 4.0 International

11 April 2011

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Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration

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Comparative study of brain CD8+ T cells induced by sporozoites and those induced by blood-stage Plasmodium berghei ANKA involved in the development of cerebral malaria

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Cytokines: accelerators and brakes in the pathogenesis of cerebral malaria

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Chemokine receptor CCR2 is not essential for the development of experimental cerebral malaria

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Locally up-regulated lymphotoxin alpha, not systemic tumor necrosis factor alpha, is the principle mediator of murine cerebral malaria

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CD8(+)-T-cell depletion ameliorates circulatory shock in Plasmodium berghei-infected mice.

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Immunopathology of cerebral malaria: morphological evidence of parasite sequestration in murine brain microvasculature

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Cloned lines of Plasmodium berghei ANKA differ in their abilities to induce experimental cerebral malaria

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10.1371/JOURNAL.PONE.0018720

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28 January 2020

2011PLoSO...618720C

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