Apoptosis: a potential target for discovering novel therapies for cardiovascular diseases (Q33695709)

28 July 2017

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title

Apoptosis: a potential target for discovering novel therapies for cardiovascular diseases (English)

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28 July 2017

main subject

cardiovascular disease

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Yue TL

1

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28 July 2017

Ohlstein EH

series ordinal

2

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28 July 2017

Ruffolo RR Jr

series ordinal

3

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28 July 2017

publication date

1 August 1999

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Current Opinion in Chemical Biology

28 July 2017

published in

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28 July 2017

volume

3

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28 July 2017

issue

4

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28 July 2017

page(s)

474-480

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

28 July 2017

cites work

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Caspases: enemies within

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Caspases: the proteases of the apoptotic pathway

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Staurosporine-induced Apoptosis in Cardiomyocytes: A Potential Role of Caspase-3

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Attenuation of Ischemia/Reperfusion Injury in Rats by a Caspase Inhibitor

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Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

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The Bcl-2 protein family: arbiters of cell survival

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An inhibitor of p38 mitogen-activated protein kinase protects neonatal cardiac myocytes from ischemia

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7 January 2021

Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy

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Insulin-like Growth Factor 1 Inhibits Apoptosis Using the Phosphatidylinositol 3′-Kinase and Mitogen-activated Protein Kinase Pathways

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7 January 2021

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7 January 2021

Possible Involvement of Stress-Activated Protein Kinase Signaling Pathway and Fas Receptor Expression in Prevention of Ischemia/Reperfusion-Induced Cardiomyocyte Apoptosis by Carvedilol

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7 January 2021

Reduction of restenosis after angioplasty in an atheromatous rabbit model by suicide gene therapy

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7 January 2021

Inhibition of neointimal cell bcl-x expression induces apoptosis and regression of vascular disease

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7 January 2021

Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats

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7 January 2021

Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion

1 reference

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7 January 2021

10.1016/S1367-5931(99)80069-6

Identifiers

DOI

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28 July 2017

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