Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury (Q28571240)

31 January 2017

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29 December 2019

title

Alterations in mammalian target of rapamycin signaling pathways after traumatic brain injury (English)

2 references

31 January 2017

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29 December 2019

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29 December 2019

author name string

Shaoyi Chen

1

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31 January 2017

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29 December 2019

Chunli L Liu

3

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29 December 2019

Ofelia F Alonso

4

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29 December 2019

W Dalton Dietrich

5

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29 December 2019

Bingren R Hu

6

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29 December 2019

publication date

30 August 2006

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Journal of Cerebral Blood Flow & Metabolism

29 December 2019

published in

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volume

27

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issue

5

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29 December 2019

page(s)

939-949

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Adaptation of the Fluid Percussion Injury Model to the Mouse

29 December 2019

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TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling

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Prolonged memory impairment in the absence of hippocampal cell death following traumatic brain injury in the rat.

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Prospective and declarative memory problems following moderate and severe traumatic brain injury

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There is differential loss of pyramidal cells from the human hippocampus with survival after blunt head injury

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Synthesis of the translational apparatus is regulated at the translational level

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S6K1(-/-)/S6K2(-/-) mice exhibit perinatal lethality and rapamycin-sensitive 5'-terminal oligopyrimidine mRNA translation and reveal a mitogen-activated protein kinase-dependent S6 kinase pathway

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Long-term hyperexcitability in the hippocampus after experimental head trauma.

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Synaptogenesis in the hippocampal CA1 field following traumatic brain injury

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BDNF regulates the translation of a select group of mRNAs by a mammalian target of rapamycin-phosphatidylinositol 3-kinase-dependent pathway during neuronal development.

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Amino acid-induced translation of TOP mRNAs is fully dependent on phosphatidylinositol 3-kinase-mediated signaling, is partially inhibited by rapamycin, and is independent of S6K1 and rpS6 phosphorylation

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7 January 2021

Physiological and structural evidence for hippocampal involvement in persistent seizure susceptibility after traumatic brain injury.

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Identifiers

DOI

10.1038/SJ.JCBFM.9600393

2 references

Consolidated OpenCitations Corpus – April 2017

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29 December 2019

1 reference

Consolidated OpenCitations Corpus – April 2017

2 references

Consolidated OpenCitations Corpus – April 2017