Role of p38 mitogen-activated protein kinase pathway in estrogen-mediated cardioprotection following trauma-hemorrhage (Q28568566)

31 January 2017

title

Role of p38 mitogen-activated protein kinase pathway in estrogen-mediated cardioprotection following trauma-hemorrhage (English)

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31 January 2017

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8

Irshad H. Chaudry

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31 January 2017

Jun-Te Hsu

1

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31 January 2017

Ya-Ching Hsieh

2

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31 January 2017

Wen Hong Kan

3

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31 January 2017

Jian Guo Chen

4

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31 January 2017

Mashkoor A. Choudhry

5

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31 January 2017

Martin G. Schwacha

6

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Kirby I. Bland

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publication date

1 June 2007

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American Journal of Physiology Heart and Circulatory Physiology

31 January 2017

published in

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31 January 2017

volume

292

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31 January 2017

issue

6

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31 January 2017

page(s)

H2982–2987

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Alpha B-crystallin in cardiac tissue. Association with actin and desmin filaments

31 January 2017

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PGC-1 upregulation via estrogen receptors: a common mechanism of salutary effects of estrogen and flutamide on heart function after trauma-hemorrhage

21 January 2018

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Hypertonicity regulates the function of human neutrophils by modulating chemoattractant receptor signaling and activating mitogen-activated protein kinase p38.

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The p38 MAPK inhibitor, SB203580, abrogates ischaemic preconditioning in rat heart but timing of administration is critical

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Sympathetic denervation does not prevent a reduction in fat pad size of rats or mice treated with peripherally administered leptin

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Mechanism of cardiac depression after trauma-hemorrhage: increased cardiomyocyte IL-6 and effect of sex steroids on IL-6 regulation and cardiac function

21 January 2018

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Mechanism of cardioprotection following trauma-hemorrhagic shock by a selective estrogen receptor-beta agonist: up-regulation of cardiac heat shock factor-1 and heat shock proteins

21 January 2018

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Binding of Cytosolic Proteins to Myofibrils in Ischemic Rat Hearts

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10.1152/AJPHEART.01303.2006

21 January 2018

Identifiers

DOI

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31 January 2017

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