Autocrine interaction between IL-5 and IL-1beta mediates altered responsiveness of atopic asthmatic sensitized airway smooth muscle (Q28343578)

2

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C Carter

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S Chuang

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M M Grunstein

Journal of Clinical Investigation

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language of work or name

English

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publication date

September 1999

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published in

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volume

104

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issue

5

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page(s)

657-67

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12 December 2020

Predominant TH2-like bronchoalveolar T-lymphocyte population in atopic asthma

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12 December 2020

Allergic and nonallergic asthmatics have distinct patterns of T-cell activation and cytokine production in peripheral blood and bronchoalveolar lavage

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Cytokines contribute to airway dysfunction in antigen-challenged guinea pigs: inhibition of airway hyperreactivity, pulmonary eosinophil accumulation, and tumor necrosis factor generation by pretreatment with an interleukin-1 receptor antagonist

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Prednisolone treatment in asthma is associated with modulation of bronchoalveolar lavage cell interleukin-4, interleukin-5, and interferon-gamma cytokine gene expression

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Detection of cytokines and their cell sources in bronchial biopsy specimens from asthmatic patients. Relationship to atopic status, symptoms, and level of airway hyperresponsiveness

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sVCAM-1 levels after segmental antigen challenge correlate with eosinophil influx, IL-4 and IL-5 production, and the late phase response

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Mechanism of impaired beta-adrenoceptor responsiveness in atopic sensitized airway smooth muscle

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T cells subsets and cytokines in allergic and non-allergic children. II. Analysis and IL-5 and IL-10 mRNA expression and protein production

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TRFK-5 reverses established airway eosinophilia but not established hyperresponsiveness in a murine model of chronic asthma

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12 December 2020