Deletion of Tricellulin Causes Progressive Hearing Loss Associated with Degeneration of Cochlear Hair Cells (Q27303592)

26 June 2017

title

Deletion of Tricellulin Causes Progressive Hearing Loss Associated with Degeneration of Cochlear Hair Cells (English)

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26 June 2017

1 reference

Toru Kamitani

1

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26 June 2017

Hirofumi Sakaguchi

2

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26 June 2017

Atsushi Tamura

3

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26 June 2017

Takenori Miyashita

4

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26 June 2017

Yuji Yamazaki

5

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26 June 2017

Reitaro Tokumasu

6

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26 June 2017

Ryuhei Inamoto

7

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26 June 2017

Ai Matsubara

8

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26 June 2017

Nozomu Mori

9

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26 June 2017

Yasuo Hisa

10

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26 June 2017

Sachiko Tsukita

11

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26 June 2017

language of work or name

English

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publication date

18 December 2015

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26 June 2017

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26 June 2017

volume

5

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26 June 2017

page(s)

18402

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Creative Commons Attribution 4.0 International

26 June 2017

copyright license

start time

1 December 2015

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4683410/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

CZ Software Mentions: A large dataset of software mentions in the biomedical literature

cites work

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Tricellulin is a tight-junction protein necessary for hearing

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Tricellulin constitutes a novel barrier at tricellular contacts of epithelial cells

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Tricellulin deficiency affects tight junction architecture and cochlear hair cells.

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ILDR1 null mice, a model of human deafness DFNB42, show structural aberrations of tricellular tight junctions and degeneration of auditory hair cells

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Perivascular-resident macrophage-like melanocytes in the inner ear are essential for the integrity of the intrastrial fluid-blood barrier.

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28 September 2017

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28 September 2017

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How is the highly positive endocochlear potential formed? The specific architecture of the stria vascularis and the roles of the ion-transport apparatus

28 September 2017

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A claudin-9-based ion permeability barrier is essential for hearing.

28 September 2017

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Tricellulin forms a barrier to macromolecules in tricellular tight junctions without affecting ion permeability

28 September 2017

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Splice-site mutations in the TRIC gene underlie autosomal recessive nonsyndromic hearing impairment in Pakistani families.

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Deafness in Claudin 11-null mice reveals the critical contribution of basal cell tight junctions to stria vascularis function.

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Loss of K-Cl co-transporter KCC3 causes deafness, neurodegeneration and reduced seizure threshold

28 September 2017

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Claudin 14 knockout mice, a model for autosomal recessive deafness DFNB29, are deaf due to cochlear hair cell degeneration

28 September 2017

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28 September 2017

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28 September 2017

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28 September 2017

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28 September 2017

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28 September 2017

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Compartmentalization established by claudin-11-based tight junctions in stria vascularis is required for hearing through generation of endocochlear potential.

31 May 2018

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31 May 2018

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31 May 2018

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31 May 2018

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DFNB49 is an important cause of non‐syndromic deafness in Czech Roma patients but not in the general Czech population

31 May 2018

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Localization of angulin-1/LSR and tricellulin at tricellular contacts of brain and retinal endothelial cells in vivo.

21 January 2018

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Development of endocochlear potential and its negative component in mouse cochlea.

21 January 2018

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Altered cochlear fibrocytes in a mouse model of DFN3 nonsyndromic deafness.

21 January 2018

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Pou3f4 deficiency causes defects in otic fibrocytes and stria vascularis by different mechanisms.

21 January 2018

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Evidence of tricellulin expression by immune cells, particularly microglia

21 January 2018

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Claudin 2 deficiency reduces bile flow and increases susceptibility to cholesterol gallstone disease in mice

21 January 2018

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21 January 2018

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28 November 2018

Identifiers

DOI

10.1038/SREP18402

1 reference

Dimensions Publication ID

26 June 2017

1003145443

0 references

OpenCitations bibliographic resource ID

2243385

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2243385

PMC publication ID

4683410

0 references

1 reference