Scribd Logo
Upload

Welcome to Scribd!

  • 494 views

    Pathophysiology of Inflammatory Response

    Uploaded by

    Deo Factuar
    The inflammatory response involves the release of chemical mediators that cause vasodilation, increased capillary permeability, and leukocyte migration. This leads to hyperemia, exudate formation, and leukocytosis as neutrophils, monocytes, and lymphocytes are attracted to the injured area. The leaked fluid and cells cause pain, swelling, and heat as the body walls off the injured tissue and clears debris via phagocytosis to promote healing.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd

    Pathophysiology of Inflammatory Response

    Uploaded by

    Deo Factuar
    494 views1 page
    The inflammatory response involves the release of chemical mediators that cause vasodilation, increased capillary permeability, and leukocyte migration. This leads to hyperemia, exudate formation, and leukocytosis as neutrophils, monocytes, and lymphocytes are attracted to the injured area. The leaked fluid and cells cause pain, swelling, and heat as the body walls off the injured tissue and clears debris via phagocytosis to promote healing.

    Copyright

    © © All Rights Reserved

    Available Formats

    PPTX, PDF, TXT or read online from Scribd

    Did you find this document useful?

    Is this content inappropriate?

    The inflammatory response involves the release of chemical mediators that cause vasodilation, increased capillary permeability, and leukocyte migration. This leads to hyperemia, exudate formation, and leukocytosis as neutrophils, monocytes, and lymphocytes are attracted to the injured area. The leaked fluid and cells cause pain, swelling, and heat as the body walls off the injured tissue and clears debris via phagocytosis to promote healing.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Download as pptx, pdf, or txt
    494 views1 page

    Pathophysiology of Inflammatory Response

    Uploaded by

    Deo Factuar
    The inflammatory response involves the release of chemical mediators that cause vasodilation, increased capillary permeability, and leukocyte migration. This leads to hyperemia, exudate formation, and leukocytosis as neutrophils, monocytes, and lymphocytes are attracted to the injured area. The leaked fluid and cells cause pain, swelling, and heat as the body walls off the injured tissue and clears debris via phagocytosis to promote healing.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Download as pptx, pdf, or txt
    of 1

    Pathophysiology of Inflammatory Response

    Etiology: Infection/ Mechanical Damage/


    Ischemia/ Nutritional Deprivation/ Extremes Tissue Injury
    Temperature/ Radiation/ etc.

    Release of chemical
    mediators (histamine, Release of
    complement, kinins, leukocytosis-inducing
    prostaglandins) factors

    Vasodilation Increased Attract neutrophils,


    capillary Leukocytosis
    of arterioles monocytes & lymphocyte to
    permeability area (chemotaxis) (increased of WBC
    in blood stream)
    Local hyperemia Capillaries leak
    fluid (exudate Migration to
    formation) injury area
    Heat Redness
    Increased Blood flow
    oxygen & Leaked protein-rich slows
    Leaked
    Increased nutrients fluid in tissue Margination
    clothing
    temperature spaces (leukocytes cling to
    proteins
    increases metabolic capillary walls)
    rate of cells
    Pain Swelling
    Walling-off process(blood clots Diapedesis (leukocytes pas
    wall off area to prevent injury to through capillary walls)
    surrounding area
    Possible temporary limitation
    of joint movement
    Phagocytosis of pathogen & dead
    Temporary fibrin tissue cells (by neutrophils, short
    patch forms term; by macrophages, long term)
    scaffolding for
    repair Pus may
    Area cleared of form
    debris

    Prepared by:
    Czarina Sales, RN Healing
    Student Name

    You might also like