Dysphonia

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DYSPHONIA

Meng Juan
Applied anatomy Laryngology
 The larynx is an air
passage,
 between the
laryngopharynx and
trachea.
 an organ of
phonation
Skeleton of Larynx
 Unpaired:Thyroid, cricoid, epiglottis.
Paired: Arytenoid, corniculate, cuneiform
Epiglottic
cartilage
Thyroid cartilage
The largest cartilage
Cricoid cartilage
The only complete
cartilaginous ring
Arytenoid cartilage
•Base :connect with cricoid cartilage
•Muscular process: give attachment to
intrinsic laryngeal muscles
•Vocal process :give attachment to vocal
cord
Ligaments and membranes
of larynx
Thyrohyoid membrane

Cricothyroid membrane
Muscles of larynx
extrinsic laryngeal muscles

intrinsic laryngeal muscles


Extrinsic laryngeal
muscles-move entire larynx, active
during swallowing
Suprahyoid Muscles
Infrahyoid Muscles  mylohyoid
 thyrohyoid  digastric
 omohyoid  geniohyoid
 sternohyoid  stylohyoid
 sternothyroid  genioglossus
 styloglossus
Intrinsic laryngeal muscles
 Abductor: Posterior
cricoarytenoid
 Adductors: Lateral
cricoarytenoid,
Transverse arytenoid
 Adjust tension of
the vocal cord:
Cricothyroid
Thyroarytenoid
Laryngeal cavity

supraglottic
portion
glottic portion
infraglottic
portion
Laryngeal nerves
Superior laryngeal nerve
Recurrent laryngeal nerve
Physiology
of the larynx

Respiration
Phonation
Protective function
Fixation of the chest
DYSPHONIA(hoarseness)
 Dysphonia is the result of noise
formed by turbulent airflow in the
larynx
 as well as by irregularities of the
normally periodic vibrations of the
vocal cords
Aetiology
 Congenital

 Traumatic

 Inflammatory

 Neoplastic

 Functional
Congenital
 Laryngomalacia (75%)

---Manifests at birth or soon after


birth.
---The soft, immature cartilage of the
upper larynx collapses inward during
inhalation, causing stridor or airway
obstruction
---Usually disappears by 2 years of age,
so the treatment is conservative.
---Tracheostomy may be required for
severe respiratory obstruction
Congenital

 vocal cord paralysis(10%)

- unilateral or bilateral recurrent


nerve palsies (idiopathic or birth
trauma)
Traumatic
 Insidethe Laryngeal cavity(internal )
- intubation
A:dislocation of the cricoarytenoid joint
Traumatic
 Insidethe Laryngeal cavity(internal )
- intubation
A:dislocation of the cricoarytenoid joint
--- shortly after removal of the tube
--- dysphonia, coughing and haemoptysis
B:granuloma of larynx
---2 to 8 weeks later after intubation
--- repeated /incorrect/over longtime
intubation, oversized tube
Intubation granulomata
Traumatic
 Outside of the laryngeal cavity(external)

---blunt or penetrating injuries


--- haematoma, oedema and
arytenoid dislocation.
--- in contact sport, assault
Recurrent laryngeal nerve damage
- unilateral - breathy voice due to air
loss. Improves later as other cord
compensates

- bilateral – may result in aphonia or


laryngeal obstruction
Recurrent laryngeal nerve damage
 causes
- Blunt or sharp trauma
- Surgery(thyroidectomy)
- Ca bronchus / oesophagus
- Pulmonary TB
-bacteria /virus infection--neuritis
- Idiopathic
Inflammatory
 Acute laryngitis

 Chronic laryngitis
Acute laryngitis
 Aninflammation of the vocal fold
mucosa and larynx lasting fewer
than 3 weeks.
Etilogy
Infection (usually viral upper
respiratory infection)
Gastroesophageal reflux
Environmental insults
(pollution)
vocal abuse
Clinical manifestation
Symptom of an upper
respiratory tract infection (ie.,
fever, cough, rhinitis, etc)
Dysphonia or hoarseness
Discomfort or pain in throat
Dry, irritating cough
Sign
 Diffuse congestion and
edema of the laryngeal
mucous, particularly the
vocal folds.
 get thicken of the vocal
folds.
 Note the presence of normal
vocal fold morbility and
the absence of airway
obstruction.
Treatment
Complete voice rest:
Ultrasonic nebulization:
glucocorticoid
Antibiotics if there are infection
of bacteria.
Chinese herbs
Chronic laryngitis
 Is a diffuse inflammatory condition
symmetrically involving the whole
larynx
Cause
 Voice abuse can be pertinent to professional
singers and to occasional shouters.
 Environmental factor: such as dust, fumes,
smoking, drinking, chemical and toxins, etc.
 Chronic inflammation of nasal cavity, sinus
and throat:
 Acute laryngitis attacks repeatedly or retains
for a long time.
 Chronic infection in the lower respiratory tract,
cough for a long-term and purulent secretion
stimulating the laryngeal mucosa.
Clinical manifestation
 dysphonia: Voice quality and
quantity may fluctuate, although
complete recovery never occurs.
 Discomfort and dryness of the
larynx
 Chronic cough: usually is dry cough.
 Laryngeal secretion is increasingly
Sign

 Redness and thickening of the entire


laryngeal mucosa in a diffuse form.
Treatment
Treatment depends upon
cause and often involves the
patient completely altering
their lifestyle.
Nebulization
Chinese herbs
Vocal cord polyp
and nodulus

 They are special forms of chronic


laryngitis.
Sign
Nodule: bilateral opposing “knot”
occur at junction of anterior and
middle third of vocal cord
(maximum work load in the cords)
Polyp: larger than nodules, but similiar location
Treatment
Strict voice rest

Surgical excision

Speech therapy for


proper voice production
Tumor of larynx

Benign tumor Malignant tumor


papiloma carcinoma
hemangioma
fibroma
neurofibroma
Functional dysphonia
 an abnormal voice is produced in the
absence of vocal pathologies
 women: men =8:1
 created by anxiety, stress,
depression, or interpersonal conflict
 Enables the person to avoid facing
the interpersonal conflict directly or
uncomfortable situation
Thank you

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