MELASMA (Ardy, Kintan, Fransisca)
MELASMA (Ardy, Kintan, Fransisca)
MELASMA (Ardy, Kintan, Fransisca)
Supervisor
Preface
Definition
Melasma is a common hypermelanosis that typically occur on sun exposed area on face.
Characterized
Epidemiology
Age
Sex
Etiology
Idiophatic
Ultraviolet Radiation
Drugs
EtioPathoge nesis
Pregnancy
Hormonal Cosmetics
Race
Examination
Diagnoses
from
clinical
symmetrical hyperpigmented macules, may form confluent or striped. Lesions: macular light brown or dark brown demarcated with irregular edges.
Count.
Predilection :
Cheeks, upper
lip,
chin,
Forehead
Histophatology
Diagnosis
Anamnesis
Physycal examination
Histopathology
Wood lamp
Differential Diagnoses
1. 2. 3.
4.
5. 6.
7.
Post-inflammatory hyperpigmentation Lentigines Addison disease Drug-induced photosensitivity Lupus erythematosus, discoid Mastocytosis Poikiloderma
Melasma
Etiology Sunshine (UV), hormone, drugs, race (black skin peoples), genetics, cosmetics, idiopathic
PIH
Infection, allergic reactions, mechanical injuries, reactions to medications, phototoxic eruptions, trauma (eg, burns), and inflammatory diseases (eg, lichen planus) any age Depending on the location of infection light brown -black brown macules or patches often with a poorly defined border, irregular in outline and usually with little surface change Asymmetric
Lentigines
History of exposures, Genetic, race (light-skinned ),
Addison Deases
Infection of the adrenal gland
Age
14-54 > Forehead, nose, cheeks, the upper lip, chin Macula (nut brown -deep brown) Macular hyperpigmentation
children and adults; buccal mucosa, gums, hard palate, lips, face, hands and feet Macula (brown -black) Macular hyperpigmentation
Predilection
Shape of the Lesion Pain in the Lesions Itching of the Lesions Burning Sensation of the Lesion Hyperasthesia
symmetric (irregular) -
Asymmetric
Melasma
Etiology Sunshine (UV), hormonal, drugs, race (heavily pigmented skin), genetics, cosmetics, idiopathic 14-54 > Forehead, nose, cheeks, the upper lip, chin Macula (nutbrown deep brown) Macular hyperpigmentation
DIP
Chemicals photosensitizing, race (heavily pigmented skin)
LED
Trauma : sunshine, chill, wind, friction; race (fair-skinned); descent
Mastocytosis
race (fair-skinned),
Poikiloderma
Sunshine (UV), chemicals photosensitizing, hormonal(low estrogen levels), race (fairskinned), genetics Middle-aged or elderly women Lateral cheek and neck
Age
The highest prevalence : 40-60 > Face, ears, chest, arm, head, back eyes Macula (Reddish blackish brown) Macular erythematous, hyperpigmented macules numular placards (butterfly app.), teleangiektasis , follicular occlusion. symmetric
The highest prevalence :30-49 = Scalp, face, extremity, palms and soles Macula (yellow-tan to red-brown) papules, nodules, and plaques , blisters and bullae
Predilection
Reddish
reddish brown
Eff.
Macular erythematous
Shape of the Lesion Pain in the Lesions Itching of the Lesions Burning Sensation of the Lesion Hyperasthesia
symmetric (irregular) -
Asymmetric
symmetric
Thank You
Chemical Peeling
Laser Therapy
Dermabration
Skin Needling