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    Pathophysiology of Bronchial Asthma

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    Carlos Niño
    Bronchial asthma is caused by a combination of predisposing genetic and environmental factors and precipitating triggers like allergens, pollution, and exercise. During an asthma attack, these triggers stimulate the immune system and cause the release of chemicals that lead to bronchospasm, inflammation, increased mucus production, and airway constriction. Without treatment, this can cause respiratory distress, hypoxemia, respiratory failure, and potentially death. Medical interventions like inhaled steroids, bronchodilators, and oxygen therapy can reverse the symptoms and return breathing to normal.

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    Pathophysiology of Bronchial Asthma

    Uploaded by

    Carlos Niño
    78 views2 pages
    Bronchial asthma is caused by a combination of predisposing genetic and environmental factors and precipitating triggers like allergens, pollution, and exercise. During an asthma attack, these triggers stimulate the immune system and cause the release of chemicals that lead to bronchospasm, inflammation, increased mucus production, and airway constriction. Without treatment, this can cause respiratory distress, hypoxemia, respiratory failure, and potentially death. Medical interventions like inhaled steroids, bronchodilators, and oxygen therapy can reverse the symptoms and return breathing to normal.

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    3806310-PATHOPHYSIOLOGY-OF-BRONCHIAL-ASTHMA

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    Bronchial asthma is caused by a combination of predisposing genetic and environmental factors and precipitating triggers like allergens, pollution, and exercise. During an asthma attack, these triggers stimulate the immune system and cause the release of chemicals that lead to bronchospasm, inflammation, increased mucus production, and airway constriction. Without treatment, this can cause respiratory distress, hypoxemia, respiratory failure, and potentially death. Medical interventions like inhaled steroids, bronchodilators, and oxygen therapy can reverse the symptoms and return breathing to normal.

    Copyright:

    © All Rights Reserved
    Download as DOC, PDF, TXT or read online from Scribd
    Download as doc, pdf, or txt
    78 views2 pages

    Pathophysiology of Bronchial Asthma

    Uploaded by

    Carlos Niño
    Bronchial asthma is caused by a combination of predisposing genetic and environmental factors and precipitating triggers like allergens, pollution, and exercise. During an asthma attack, these triggers stimulate the immune system and cause the release of chemicals that lead to bronchospasm, inflammation, increased mucus production, and airway constriction. Without treatment, this can cause respiratory distress, hypoxemia, respiratory failure, and potentially death. Medical interventions like inhaled steroids, bronchodilators, and oxygen therapy can reverse the symptoms and return breathing to normal.

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    PATHOPHYSILOGY OF BRONCHIAL ASTHMA

    *  Present in the patient

    Predisposing Factors Precipitating Factors


    * Genetics * Environmental factors (change in temperature)
    *Race * Atmospheric pollutants (perfume, smoke, etc.)
    * Age (3-8 years old) * allergens (pets)
    * Gender exercise, stress on emotional upset,
    chemicals,
    medications

    1 gE stimulations

    mast cell degranultation

    asthma attack

    altered immunologic response increased airway resistance increased lung compliance impaired mucocilary function altered O2-CO2
    exchange
    -lungs become
    chemical mediators are released mucus inflammation bronchospasm hyperinflated increase mucus production increase airway
    resistance
    secretion
    * audible expiratory and inspiratory slowed clearance of mucus respiratory muscles works
    histamine SRS-A lenkotrimes - accessory muscle breathing harder
    Prostaglandins Bradykinins - nasal flaring increase water loss from mucus
    * tachypnea muscle fatigue and
    SPASM - tachycardia mucus becomes increasingly viscous exhaustion

    *whezzing respiratory alkalosis


    * paroxysmal deppnea
    * --------/production cough

    without medical intervention with medical interventions

    individual compensates without O2 therapy


    increased RR
    medications: inhaled steroid, beta2
    respiratory alkalosis adrenergic agents *bronchodilators
    (salbutamol)
    mild ---------- hypoxemia
    opens the airway
    severe ----------- hypoventilation
    deep breathing and coughing exercise
    respiratory acidosis
    coughing up thick tenacious spultum
    severe hypoxemia
    adequate rest
    death
    H teachings
    BAD PROGNOSIS - increase fluid intake
    - elevate HOB
    - limit exposure to allergens
    - minimize stenous activities

    GOOD PROGNOSIS

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