Pathological Gaits: Quadriceps Weakness (Hand 2 Knee Gait)

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Pathological Gaits

Quadriceps Weakness (Hand 2 knee gait)


In quadriceps weakness, forces tend to flex the knee. The
compensatory mechanisms that take place are:
Patients lean forward to bring the body weight anterior
to the knee resulting in a straight knee with excessive
plantar flexion, hip flexion and a forward trunk.

Pretibial Muscle Weakness (weak dorsiflexors)


Weakness of the pretibial muscles causes slapping of the
foot during the stance phase at the time of loading response.
The compensatory mechanisms that prevent this foot
slapping are:
• Ipsilateral circumduction of the limb
• Ipsilateral high stepping gait (steppage or equine gait)
(Fig. 4)
• Contralateral hip hiking
• Instead of normal heel strike, there is slapping of whole
Foot

Antalgic Gait(antalgic = anti- + alge, "against pain").


When the hip joint is painful, there is lurch of trunk
towards the painful side. This moves the center of
gravity of body towards the painful hip decreasing the
moment arm of body weight to the hip joint. Unlike in
the Trendelenburg gait, the lurch is to the same side.

Hip Hiking
In the swing phase, there is elevation of pelvis by quadratus
lumborum and abductors to clear the foot.It is seen in cases
of ipsilateral lengthening as in case of hip
flexor weakness, ankylosis in extension.
Circumduction
It is another mechanism to gain ground clearance. It is seen
in cases of spasticity, weak hip flexors, weak hamstrings,
knee ankylosis and weak dorsiflexors of foot.

Anterior Trunk Bending


It is seen during mid-stance late swing in cases of quadriceps
Weakness.

Hemiplegic Gait
Arm on affected side is flexed, adducted and internally
rotated with loss of normal arm swing while walking.
Affected leg is in extension with plantar flexion of the
foot and toes. The affected leg is dragged in a semicircle
(circumduction) due to weakness of distal muscles (foot
drop) and extensor hypertonia in lower limb. This is most
commonly seen in stroke.

Diplegic Gait
The patient walks with an abnormally narrow base, dragging
both legs. This gait is seen in bilateral periventricular lesions,
such as those seen in cerebral palsy. There is tightness of hip
adductors which can cause legs to cross the midline known
as a scissors gait
Parkinsonian Gait
The patient has rigidity and bradykinesia. He will stoop
forward with flexion at the knees. The patient walks with
slow little steps known at marche à petits pas (walk of little
steps) or shuffling gait. Patient also has difficulty in initiating
steps. The patient has an involuntary inclination to take
accelerating steps and finds it difficult to stop once gait is
initiated known as festination.

Ataxic (Cerebellar) Gait (Drunkers Gait)


Ataxic gait is a clumsy, staggering movement with a
widebased gait. Even while standing still, the patient’s body
may swagger back and forth and from side to side, known
as titubation. Patient is not able to walk from heel to toe or
in a straight line. It is also known as reeling gait.

Reeling / Drunkers Gait .


Gluteus Maximus Gait or Rocking Horse Gait
This is seen in gluteus maximus weakness. Here the trunk
shifts posteriorly at heel strike to shift the center of gravity
posterior to gluteus maximus, thus reducing the effort
required by it to keep hip in extension during stance phase.
This forward-backward lurching is also known as rocking
horse gait .

Gluteus Medius or Trendelenburg Gait


In normal gait during single stance phase when weight
of body is on one limb the sag of pelvis to the opposite
side is prevented by the ipsilateral gluteus medius. When
this muscle is weak, the trunk is shifted the same side to
reduce the lever arm of the muscle, thus reducing the effort
required by it to hold up the pelvis.When this is
bilateral, it is known as waddling gait or duck gait.

Hamstring Gait
During stance phase, the knee hyperextends (genu
recurvatum gait). During terminal swing, the weak
hamstring cannot slow the leg and it snaps into extension.

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