TandA 9912
TandA 9912
TandA 9912
History
Celsus 50 A.D. Caque of Rheims Philip Syng Wilhelm Meyer 1867 Samuel Crowe
Embryology
8 weeks: Tonsillar fossa and palatine tonsils develop from the dorsal wing of the 1st pharyngeal pouch and the ventral wing of the 2nd pouch; tonsillar pillars originate from 2nd/3rd arches Crypts 3-6 months; capsule 5th month; germinal centers after birth 16 weeks: Adenoids develop as a subepithelial infiltration of lymphocytes
Anatomy
Tonsils
Plica triangularis Gerlachs tonsil
Adenoids
Fossa of Rosenmller Passavants ridge
Blood Supply
Tonsils
Ascending and descending palatine arteries Tonsillar artery 1% aberrant ICA just deep to superior constrictor
Adenoids
Ascending pharyngeal, sphenopalatine arteries
Histology
Tonsils
Specialized squamous Extrafollicular Mantle zone Germinal center
Adenoids
Ciliated pseudostratified columnar Stratified squamous Transitional
Acute Adenotonsillitis
Etiology 5-30% bacterial; of these 39% are beta-lactamaseproducing (BLPO) Anaerobic BLPO GABHS most important pathogen because of potential sequelae Throat culture Treatment
Microbiology of Adenotonsillitis
Most common organisms cultured from patients with chronic tonsillar disease (recurrent/chronic infection, hyperplasia): Streptococcus pyogenes (Group A beta-hemolytic streptococcus) H.influenza S. aureus Streptococcus pneumoniae Tonsil weight is directly proportional to bacterial load.
Acute Adenotonsillitis
Differential diagnosis
Infectious mononucleosis Malignancy: lymphoma, leukemia, carcinoma Diptheria Scarlet fever Agranulocytosis
Medical Management
PCN is first line, even if throat culture is negative for GABHS For acute UAO: NP airway, steroids, IV abx, and immediate tonsillectomy for poor response Recurrent tonsillitis: PCN injection if concerned about noncompliance or antibiotics aimed against BLPO and anaerobes For chronic tonsillitis or obstruction, antibiotics directed against BLPO and anaerobes for 3-6 weeks will eliminate need for surgery in 17%
Obstructive Hyperplasia
Adenotonsillar hypertrophy most common cause of SDB in children Diagnosis Indications for polysomnography Interpretation of polysomnography Perioperative considerations
Peritonsillar Abscess
ICA Aneurysm
Pleomorphic Adenoma
Candidiasis
Syphilis
Retention Cysts
Supratonsillar Cleft
Gates et al (1994)
Recommend adenoidectomy with M & T as the initial surgical treatment for children with MEE > 90 days and CHL > 20 dB
Infection:
Recurrent/chronic adenoiditis (3 or more episodes/year) Recurrent/chronic OME (+/- previous BMT)
Surgeons preference
Complications
#1 Postoperative bleeding Other:
Sore throat, otalgia, uvular swelling Respiratory compromise Dehydration Burns and iatrogenic trauma
Rare Complications
Velopharyngeal Insufficiency Nasopharyngeal stenosis Atlantoaxial subluxation/ Grisels syndrome Regrowth Eustachian tube injury Depression Laceration of ICA/ pseudoaneursym of ICA
Management of Hemorrhage
Ice water gargle, afrin Overnight observation and IV fluids Dangerous induction ECA ligation Arteriography
Case study
13 year old female referred by PCP for frequent throat infections Shes always sick. Shes been on four different antibiotics this year. You call her pediatrician he is out of town and his nurse cant find the chart
Case study
No known medical problems, no prior surgical procedures Takes motrin for menustrual cramps No personal history of bleeding other than occasional nose bleeds and extremely heavy periods. Family history unknown. Patient is adopted.
Case study
Physical exam is unremarkable. Mom breaks down in tears when you tell her you do not have enough documentation of illness to warrant T & A. I had to go on welfare because Ive missed so much work from her being out sick. You hesitate. She adds, Her grades have dropped from all As to all Fs. If she misses any more school, shell be held back.
Case study
You confirm with her pediatrician that she has had 4 episodes of tonsillitis this year and agree to T & A. Because of her history of epistaxis and menorrhagia, you order a PT, PTT, CBC, BT. She has a mild microcytic anemia and prolonged bleeding time. You order vWF activity level and consult hematology
Case study
She has a subnormal level of vWF, which responds to a DDAVP challenge (rise in vWF and Factor VII greater than 100%). You advise her to stop taking motrin. Before surgery, she receives desmopressin 0.3 microg/kg IV over 30 min and amicar 200mg/kg.
Case study
She receives the same dose of DDVAP 12 hours postoperatively and every morning. Amicar is given 100mg/kg PO q 6 hr. Before each dose of DDAVP, serum sodium is drawn. Sodium levels drop to 130. Desmopressin is discontinued and substituted with cryoprecipitate.
Case study
Patient presents to the ER on POD # 7 complaining of intermittent bleeding from her mouth. You order cryoprecipitate, draw a Factor VII level and CBC, and call her hematologist. Hemoglobin has dropped from 11.9 to 9.6.
Case study
PE reveals no active bleeding; an old clot is present You establish IV access, admit the patient for overnight observation, have her gargle with ice water, and administer crypoprecipitate No further bleeding occurs, patient is discharged the next day