Pages that link to "Q49090173"
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The following pages link to Immunoglobulins and complement factors in senile plaques. An immunoperoxidase study (Q49090173):
Displaying 50 items.
- Variant of TREM2 Associated with the Risk of Alzheimer's Disease (Q22250874) (← links)
- Using animal models to determine the significance of complement activation in Alzheimer's disease (Q24796236) (← links)
- What can imaging tell us about cognitive impairment and dementia? (Q26749292) (← links)
- Genetics ignite focus on microglial inflammation in Alzheimer's disease (Q26781865) (← links)
- From development to dysfunction: microglia and the complement cascade in CNS homeostasis (Q26865892) (← links)
- Inflammation and Alzheimer's disease (Q28138617) (← links)
- No effect of one-year treatment with indomethacin on Alzheimer's disease progression: a randomized controlled trial (Q28471970) (← links)
- Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides (Q30665192) (← links)
- Complement component C3 and complement receptor type 3 contribute to the phagocytosis and clearance of fibrillar Aβ by microglia (Q30665205) (← links)
- The amyloid cascade-inflammatory hypothesis of Alzheimer disease: implications for therapy (Q30668465) (← links)
- Can oral infection be a risk factor for Alzheimer's disease? (Q33361553) (← links)
- Alzheimer's disease and oxidative stress: implications for novel therapeutic approaches (Q33546151) (← links)
- The importance of inflammatory mechanisms for the development of Alzheimer's disease (Q33703419) (← links)
- Proteomics analysis of methylglyoxal-induced neurotoxic effects in SH-SY5Y cells (Q33803021) (← links)
- Tracking neuroinflammation in Alzheimer's disease: the role of positron emission tomography imaging (Q33904501) (← links)
- Amyloid beta peptide: a century of discoveries. (Q33937049) (← links)
- Focal inflammation in the brain: role in Alzheimer's disease (Q33942016) (← links)
- The role of complement in Alzheimer's disease pathology (Q33971345) (← links)
- Complement components of the innate immune system in health and disease in the CNS. (Q33974402) (← links)
- Aggregation of vascular risk factors and risk of incident Alzheimer disease (Q33989502) (← links)
- Treatment of Alzheimer's disease: rationale and strategies (Q34079586) (← links)
- Increased plasma TACE activity in subjects with mild cognitive impairment and patients with Alzheimer's disease (Q34129473) (← links)
- The Alzheimer's A beta -peptide is deposited at sites of complement activation in pathologic deposits associated with aging and age-related macular degeneration (Q34154466) (← links)
- The complement system in central nervous system diseases (Q34325902) (← links)
- Alzheimer's disease risk genes and mechanisms of disease pathogenesis (Q34425904) (← links)
- Roles of the complement system in human neurodegenerative disorders: pro-inflammatory and tissue remodeling activities. (Q34560179) (← links)
- The significance of neuroinflammation in understanding Alzheimer's disease. (Q34573524) (← links)
- Evaluation of selective COX-2 inhibitors for the treatment of Alzheimer's disease (Q34626110) (← links)
- BioAge: toward a multi-determined, mechanistic account of cognitive aging (Q34644175) (← links)
- Serine protease HtrA1 accumulates in corneal transforming growth factor beta induced protein (TGFBIp) amyloid deposits (Q34671631) (← links)
- Demonstration of microglial cells in and around senile (neuritic) plaques in the Alzheimer brain. An immunohistochemical study using a novel monoclonal antibody (Q34678250) (← links)
- Apolipoprotein E isoform-dependent microglia migration (Q35003241) (← links)
- Risk factors for Alzheimer's disease: role of multiple antioxidants, non-steroidal anti-inflammatory and cholinergic agents alone or in combination in prevention and treatment (Q35025023) (← links)
- Sulfated glycoprotein 2: new relationships of this multifunctional protein to neurodegeneration (Q35150586) (← links)
- Yin and Yang: complement activation and regulation in Alzheimer’s disease (Q35563953) (← links)
- Isoprenoids and related pharmacological interventions: potential application in Alzheimer's disease (Q35634151) (← links)
- Humoral and cellular immunologic repertoire in Alzheimer's disease (Q35656627) (← links)
- Alzheimer's disease neuropathology. Current status of interpretation of lesion development (Q35659487) (← links)
- Temporal accrual of complement proteins in amyloid plaques in Down's syndrome with Alzheimer's disease (Q35745403) (← links)
- Presenilin-1 is associated with Alzheimer's disease amyloid (Q35763893) (← links)
- Up-regulated production and activation of the complement system in Alzheimer's disease brain (Q35786312) (← links)
- Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysis (Q35829491) (← links)
- Homology of the amyloid beta protein precursor in monkey and human supports a primate model for beta amyloidosis in Alzheimer's disease. (Q35831302) (← links)
- Accumulation of intercellular adhesion molecule-1 in senile plaques in brain tissue of patients with Alzheimer's disease. (Q35833354) (← links)
- Extensive innate immune gene activation accompanies brain aging, increasing vulnerability to cognitive decline and neurodegeneration: a microarray study (Q36164195) (← links)
- Epistasis analysis links immune cascades and cerebral amyloidosis (Q36337137) (← links)
- C1q ablation exacerbates amyloid deposition: A study in a transgenic mouse model of ATTRV30M amyloid neuropathy. (Q36346078) (← links)
- Complement-dependent proinflammatory properties of the Alzheimer's disease beta-peptide (Q36401494) (← links)
- Ageing and neuronal vulnerability (Q36428039) (← links)
- Amyloid Precursor Protein (APP) and ΒZA4 Amyloid in the Etiology of Alzheimer's Disease: Precursor-Product Relationships in the Derangement of Neuronal Function (Q36476991) (← links)