Pages that link to "Q37281615"

The following pages link to Loss-of-function analysis suggests that Omi/HtrA2 is not an essential component of the PINK1/PARKIN pathway in vivo (Q37281615):

Displaying 47 items.

• PINK1 is selectively stabilized on impaired mitochondria to activate Parkin (Q21145802) (← links)
• MUL1 acts in parallel to the PINK1/parkin pathway in regulating mitofusin and compensates for loss of PINK1/parkin (Q24298771) (← links)
• BAG5 protects against mitochondrial oxidative damage through regulating PINK1 degradation (Q24324175) (← links)
• Oxidative stress-induced signaling pathways implicated in the pathogenesis of Parkinson's disease (Q24337079) (← links)
• Mitochondrial dynamics and mitophagy in Parkinson's disease: disordered cellular power plant becomes a big deal in a major movement disorder (Q27001551) (← links)
• Drosophila HtrA2 is dispensable for apoptosis but acts downstream of PINK1 independently from Parkin (Q28752238) (← links)
• HTRA2-related serine protease Dmel_CG8464 (Q29819537) (← links)
• Silencing of PINK1 expression affects mitochondrial DNA and oxidative phosphorylation in dopaminergic cells (Q33415997) (← links)
• Novel mitochondrial substrates of omi indicate a new regulatory role in neurodegenerative disorders (Q33504585) (← links)
• Mitochondrial quality control: insights on how Parkinson's disease related genes PINK1, parkin, and Omi/HtrA2 interact to maintain mitochondrial homeostasis (Q33603972) (← links)
• Drosophila Models of Parkinson's Disease (Q33817688) (← links)
• A pivotal role for PINK1 and autophagy in mitochondrial quality control: implications for Parkinson disease (Q33871331) (← links)
• Drosophila melanogaster in the study of human neurodegeneration (Q34022667) (← links)
• Two rare human mitofusin 2 mutations alter mitochondrial dynamics and induce retinal and cardiac pathology in Drosophila (Q34408084) (← links)
• Mitochondrial morphogenesis, distribution, and Parkinson disease: insights from PINK1 (Q34610341) (← links)
• Modulation of mitochondrial function and morphology by interaction of Omi/HtrA2 with the mitochondrial fusion factor OPA1. (Q34713631) (← links)
• Drosophila models of Parkinson's disease: discovering relevant pathways and novel therapeutic strategies (Q34787767) (← links)
• A Link Between Impaired Purine Nucleotide Synthesis and Apoptosis in Drosophila melanogaster (Q35065493) (← links)
• Phosphorylation of HtrA2 by cyclin-dependent kinase-5 is important for mitochondrial function. (Q35315489) (← links)
• Mitochondrial dynamics and Parkinson's disease: focus on parkin (Q35799161) (← links)
• Drosophila as a model to study mitochondrial dysfunction in Parkinson's disease (Q36526622) (← links)
• The roles of PINK1, parkin, and mitochondrial fidelity in Parkinson's disease (Q36611426) (← links)
• Protein degradation in Parkinson disease revisited: it's complex (Q37111325) (← links)
• Mitochondrial dynamics in Parkinson's disease (Q37364337) (← links)
• X11/Mint genes control polarized localization of axonal membrane proteins in vivo (Q37397941) (← links)
• PINK1 as a molecular checkpoint in the maintenance of mitochondrial function and integrity (Q37461824) (← links)
• Inside an enigma: do mitochondria contribute to cell death in Drosophila? (Q37500083) (← links)
• Modelling Parkinson's disease in Drosophila (Q37621281) (← links)
• The PARL family of mitochondrial rhomboid proteases (Q37666154) (← links)
• What have we learned from Drosophila models of Parkinson’s disease? (Q37796006) (← links)
• Mitophagy: the latest problem for Parkinson's disease (Q37819135) (← links)
• New animal models of Parkinson's disease (Q37951958) (← links)
• Parkin, PINK1 and mitochondrial integrity: emerging concepts of mitochondrial dysfunction in Parkinson's disease (Q37953288) (← links)
• Mitochondrial quality control: a matter of life and death for neurons (Q37987023) (← links)
• Function and characteristics of PINK1 in mitochondria (Q38093581) (← links)
• Mitochondrial Dysfunction Precedes Other Sub-Cellular Abnormalities in an In Vitro Model Linked with Cell Death in Parkinson’s Disease (Q39503922) (← links)
• Mitochondrial defects and neurodegeneration in mice overexpressing wild-type or G399S mutant HtrA2. (Q40278299) (← links)
• Bar-coding neurodegeneration: identifying subcellular effects of human neurodegenerative disease proteins using Drosophila leg neurons. (Q41440551) (← links)
• Toxin-Induced and Genetic Animal Models of Parkinson's Disease (Q41605202) (← links)
• In a flurry of PINK, mitochondrial bioenergetics takes a leading role in Parkinson's disease. (Q42206178) (← links)
• Control of mitochondrial integrity in Parkinson's disease (Q48095146) (← links)
• The HtrA2 Drosophila model of Parkinson's disease is suppressed by the pro-survival Bcl-2 Buffy (Q51325327) (← links)
• PINK1: one protein, multiple neuroprotective functions (Q57243906) (← links)
• Atg1 mediated autophagy suppresses tissue degeneration in pink1/parkin mutants by promoting mitochondrial fission in Drosophila (Q57793295) (← links)
• Mitochondrial Regulation by PINK1-Parkin Signaling (Q58905388) (← links)
• Mitochondrial HTRA2 Plays a Positive, Protective Role in Dictyostelium discoideum but Is Cytotoxic When Overexpressed. (Q64932780) (← links)
• Enhancing glycolysis attenuates Parkinson's disease progression in models and clinical databases (Q90121106) (← links)