Angina Pektoris Stabil Ing

Download as pptx, pdf, or txt
Download as pptx, pdf, or txt
You are on page 1of 15

STABLE ANGINA PECTORIS

Nur Ramadhani Ulfa


C014191003
Pembimbing
dr. Pendrik Tandean, Sp.PD-KKV
DEFINITION

• Angina pectoris or angina is the symptoms of chest pain or pressure occurs when the
heart does not receive enough sufficient blood and oxygen supply. In general, angina used
to be caused by atheroma plaque which built by piled up of cholesterol lipid or other
structures in the coronary arteries.
CLASSIFICATION

1. Typical Angina
Chest pain fulfill the below 3 characteristics
a) Uneasy feelings in the substernal for certain duration
b) Aggravated by activities and stress
c) Relieved by resting and nitrate
2. Atypical Angina
Has two symptoms of typical angina and same characteristics but do not have trigger factor such as
stress. Pain starts during the resting time which stays up to 15 minutes.
CLASSIFICATION

3. Non Angina Chest Pain


Chest pain which will not relieved by nitrite, that will stay for several hours or even days.
APS DEGREE CLASSIFICATION BASED ON
CANADIAN CARDIOVASCULAR SOCIETY
• Class 1 : Normal activities will not cause angina, such as walking on climbing ladders. Angina will
show up when straining or quick activities and after long time working or sports activities.
• Class 2 : A limited restriction during normal activities. Angina during quick activities or climbing
ladders after eating or cold weather, angina during emotional stress, or a few hours after wake up.
Walking more than two blocks or climbing fastly or in normal condition.
• Class 3 : Distinct restriction on normal physical activities. Angina shows up during walking around
one or two blocks, climbing on floor in normal condition.
• Class 4 : Unable to do physical activities without feeling uncomfortable, angina could show up
when during the resting time.
PATHOPHYSIOLOGY

FACTORS OUTSIDE THE CARDIAC


Hypertension, systemic, tachyarrhythmia, thyrotoxicosis, using drugs to increase demand
on O2 myocardial that will unbalance the demand and supply O2 to myocardial.
Thrombocyte stenosis aggregation in coronary artery will cause turbulence of blood flow
that will cause increasing thrombocyte aggregation which will ended to thrombus formation
and this condition will ease for vasoconstriction of blood vessels.
PATHOGENESIS OF ATHEROSCLEROSIS
RUPTURING PROCESS OF THE ATHEROMA
PLAQUE

1. Can not be modified: age, sex, family history


2. Risk factors that can be modified: smoking, hyperlipidemia, hypertension, obesity,
Diabetes Mellitus
DIAGNOSIS

• ANAMNESIS
• PHYSICAL EXAMINATION
• ELECTROCARDIOGRAPHY (ECG)
• CHEST RADIOGRAPH
• ECHOCARDIOGRAPHY
• MAGNETIC RESONANCE IMAGING (MRI)
• ANAMNESIS
- Which location? In the chest round sternum near epigastrium area
- How is the pain characteristics? Such as chest pressure, tightness, inflame
- Duration over than fifteen minutes
- Aggravating factors and relieving factors. Is it aggravated by activities?
PHYSICAL EXAMINATION
Anemia, hypertension, valvular heart disease, cardiomyopathy, arrhythmia, kidney disease,
Diabetes Mellitus but usually in the physical examination is not specific for stable angina
pectoris.
ELECTROCARDIOGRAPHY
In electrocardiography, stable angina pectoris indicated ST segment depression, sometimes
shows elevation or ST segment normalization, sometimes indicated elevation or ST segment
normalization or T wave usually not specific; can be stated as angina.
RADIOGRAPHY
To evaluate whether there is symptoms of congestive heart failure and other causes such as
pneumothorax, lungs infection, and mass, pulmonal hypertension or whether there is
mediastinal widening.
MRI
For detection whether there is complications of myocardial infarction
FARMACOLOGY

• Aspirin
• Nitrate
• Beta blocker
• Statin
PROGNOSIS

• Symptoms during resting time shows bad prognosis. Besides when the continuous pain
occur or tachycardia, hypotension, and heart failure also the sign of increasing risk.
THANK YOU

You might also like