Comfort and Pain Management
Comfort and Pain Management
Comfort and Pain Management
• Transmission
• Perception
• Modulation
Transduction
• Begins in periphery
• Pain producing stimuli sends impulse to
nerve fiber
• Pain fiber enter spinal tract
• Pain message is prevented from reaching
brain or enters cerebral cortex
• Once in cerebral cortex pain perception
interpreted causing a response
Transduction
• All cellular damage caused by thermal,
mechanical, or chemical stimuli result in the
release of pain producing substances
• Bradykinin, Histamine, Substance P
• These pain producing substances surround
the pain fibers in the extracellular fluid,
spreading the pain message and causing the
inflammatory response
Transduction
• Nerve impulses resulting from the
painful stimulus travel along peripheral
nerve fibers
• Two types of peripheral nerve fibers
conduct pain
1. Fast, myelinated A-delta
2. Slow, unmyelinated C
Transmission
• Neuroregulators affect the transmission
of nerve stimuli
• Substances are found at the site of a
nociceptor at nerve terminals within the
dorsal horn of the spinal tract and at
receptor sites within the spinothalmic
tract
Transmission
• Neurotransmitters
1. Substance P
2. Serotonin
3. Prostaglandins
• Neuromodulators
1. Endorphins
2. Bradykinin
Gate Control Theory of
Pain
• Pain impulses can be regulated or even
blocked by gating mechanism along CNS
• Theory suggests that pain impulses pass
when gate is open and blocked when gate is
closed
• Closing the gate is basis for pain relief
interventions
Gate Control Theory of
Pain
• Involves the addition of mechanoreceptors
(A-beta neurons), which releases inhibiting
neurotransmitter (Serotonin)
• If dominant input is from A-beta fibers, gating
mechanism will close, pain reduced, due to
release of Serotonin (Back rub)
• If dominant input from A-delta fiber, gate will
be open and pain perceived
• Release of endorphins also close gate
Perception
• Point at which person is aware of pain
• Pain stimuli are transmitted up spinal cord to
thalamus and midbrain
• From thalamus, fibers transmit pain message
to cortex, frontal lobe, & limbic system
• Somatosensory cortex-identifies location &
intensity of pain
• Association cortex- how we feel pain
Perception
• Limbic system-controls emotion,
anxiety, & emotional reaction to pain
• Responses to pain can be physiological
and behavioral
Physiological Response
to Pain
• ANS stimulated as pain impulses ascend the
spinal cord
• Pain of low to moderate intensity and
superficial pain elicit the “fight or flight”
reaction
• Sympathetic stimulation results in physiologic
responses (Increased heart rate, peripheral
vasoconstriction, dilatation of bronchial tubes,
increased blood sugar)
Physiological Response
to Pain
• Continuous pain or severe, deep pain
(visceral) involving organs puts the
parasympathetic system into effect
• Parasympathetic stimulation results in
pallor, muscle tension, decreased heart
rate and BP, N/V, weakness,
exhaustion
Behavioral Responses to
Pain
• Pain threatens physical & psychological well-
being
• Some people choose not to express pain
(belief, value, cultural influences)
• Typical body movements that indicate pain:
clenching teeth, grimace, holding area, bent
posture
Modulation
• Process of inhibiting or changing pain
impulse
• Final process in nociception
• Involves release of serotonin and endorphins
• Work to inhibit pain or provide an analgesic
effect
• Release of endorphins can raise an
individuals pain threshold
Acute Pain
• Follows acute injury, disease, surgical
intervention
• Rapid onset
• Varies in intensity (mild-severe)
• Lasts a brief period of time (less than 6
months)
Chronic Pain
• Prolonged
• Varies in intensity
• Lasts longer than 6 months
• Also known as chronic non-malignant
pain
• Arthritis, headache, myofascial pain,
low back pain
Cancer Pain
• Pain that is due to tumor progression
• Related to pathology, invasive procedures,
infection, toxicities of Rx
• Can be acute or chronic, nociceptive or
neuropathic
• At the actual site or distant to the site
(Referred pain)
Factors Influencing Pain
• Age
• Gender
• Culture
• Meaning of pain
• Attention
• Anxiety
• Fatigue
• Previous Experience
• Coping Style
• Family & Social Support
Nursing Process
Assessment
• AHCPR guidelines for assessing pain
• Clients expression of pain
• Characteristics of pain
• Onset & duration
• Location
• Intensity (Pain scales-numerical,
FACES)
Assessment
• Quality
• Pain pattern
• Concomitant Symptoms
• Effect of pain on client (physical,
behavioral, effect on ADL)
Nursing Process
Nursing Diagnosis
• Anxiety
• Alteration in Comfort
• Self-care Deficit
• Sleep Pattern Dysfunction
• Sexual Dysfunction
Nursing Process
Implementation
• Non-Pharmacological and
pharmacological Methods
• Non-pharmacologic methods-lessen
pain, can be used at home or in hospital
• Utilize cognitive-behavioral & physical
approaches
• Allow patients some control
Non-pharmacological
Methods
Acupuncture
• Relaxation
• Guided Imagery
• Distraction
• Music
• Biofeedback
• Self-Hypnosis
• Reducing Pain Perception
• Cutaneous Stimulation (Heat or Cold
application, massage, TENS unit)
Pharmacologic Methods
• Require a physicians order
• Guidelines set by regulatory agencies
• Analgesics most common method
• Tendency to under treat with pain meds
Analgesics
• Non-opioid or non-narcotic agents &
non-steroidal anti-inflammatory agents
(NSAIDS)
• Narcotics, Opioids
• Adjuvants, Co-analgesics
NSAIDS
• Relief of mild to moderate pain
• Believed to inhibit prostaglandins & inhibits
cellular response during inflammation
• Acts on peripheral nerve receptors to reduce
the transmission & reception of pain
• Does not cause sedation or respiratory
depression or interfere with bowel/bladder
function
• Avoid prolonged or overuse in elderly
NSAIDS
• Used in arthritic pain, minor surgical,
dental procedures, low back pain,
should be initially used in mild-moderate
post-op pain
• Motrin, Naprosyn, Indocin, Toradol
Opioids
• Moderate to severe pain
• Act on CNS, act on higher brain centers
& spinal cord binding with opiate
receptors to modify perception of or
reaction to pain
• Risk for depression of vital nervous
system functions
Opioids
• If pain is anticipated for longer than 12-24
hours, ATC timing should be used instead of
PRN timing
• Opioids can be used effectively with elderly,
START LOW & GO SLOW
• Morphine, Demerol, Codeine, Percocet,
Fentanyl, Hydromorphone
• Opioid antagonist- NARCAN-reverses effect
Adjuvant Therapy
• Sedatives, anti-anxiety, & muscle
relaxants
• Enhance pain control or relieve
symptoms associated with pain
• Vistaril, Elavil, Thorazine, Valium,
Ativan, Xanax
Patient-Controlled
Analgesia PCA
• Drug delivery system
• Patients have control over pain therapy
• Safe method for post-op, traumatic, or
cancer pain
• Self-administration without risk of
overdose
• IV administration
PCA Prescription
• Loading Dose
• Basal (Continuous rate)
• On demand dose
• Hourly maximum amounts can be
prescribed
Local & Regional
Anesthetics
• Wound suturing
• Delivery of baby
• Performing simple surgery
• Epidural Analgesia for post-op pain
management, L&D pain, chronic cancer
pain
Epidural Pain
Management
• Short or long term
• Administered into spinal epidural space
• Catheter is left in place, secured with
tape and dressing
• Can be continuous infusion or daily
injection
Epidural Pain
Management
• Monitor hourly for:
1. Catheter Displacement
2. Catheter Function
3. Respiratory Depression
4. Side effects: N/V, itching, urinary
retention, constipation
5. Pain effect
Cancer Pain
Management
• Long acting preparations, sustained
release
• Drug dependence low in cancer related
pain
• Can develop tolerance, requiring higher
doses
• Goal is to minimize pain, rather than
cure it
Clicker Question
43 - 73
Clicker Question
43 - 74
Clicker Question
43 - 75