Chronic Visual Loss: UBC Ophthalmology Club 2012
Chronic Visual Loss: UBC Ophthalmology Club 2012
Chronic Visual Loss: UBC Ophthalmology Club 2012
Monthly
injection,
$1600 per
shot
Case 3
68F comes to GP with c/o decreased vision in her L eye
She denies double vision or glares, in fact she said she can
read better with her L eye than her R eye now; she wants
to know if her reading glasses are still necessary
No eye disease or trauma
No family hx of eye diseases
Meds include prednisone 20 mg daily for last 2 months
for RA flare
Case 3
OD 20/30, OS 20/50
Pupils equal reactive no
RAPD
EOM full
Confrontational VF full
Fundus visualized,
unremarkable
Implantable IOL
Case 4
63M with 17 yr hx of Type 2 DM comes to GP to c/o
decrease in vision in both eyes
Denies pain, distortions, double vision
Hb A1c 7.5% despite being on metformin and gliclazide
Also has dyslipidemia, on atorvastatin
No previous eye complaints
Case 4
OD 20/40, OS 20/60
Pupils equal reactive, red
reflex present
EOM full VF intact
AC deep and quiet
Fundoscopy:
(AAO, 2012)
Diagnosis
What is the cause of this pt’s decreased vision?
A. Non-proliferative diabetic retinopathy
B. Age related macular degeneration
C. Proliferative diabetic retinopathy
D. Branch retinal vein occlusion
Diabetic retinopathy
Microvascular complication of DM
Most common cause of vision loss in adults 25-74 yr
In NPDR, vision loss arise from macular edema
In PDR, vision loss can be rapid, secondary to scarring
and vitreous hemorrhage
Ophthalmologist referral when:
Newly diagnosed DM patient
Eye exam every 1-2 years after
Patient who develop rapid vision change
Glycemic control is the cornerstone of systemic
management. DR is managed with laser and anti-VEGF
PDR
Contact:
R Tom Liu
[email protected]