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    Hypermagnesemia

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    Nader Smadi
    The document discusses hypermagnesemia, or high magnesium levels, which can occur in patients receiving magnesium sulfate. It notes that toxicity may begin around a magnesium level of 4 mEq/L, while most patients receiving magnesium sulfate for preeclampsia prevention are maintained between 4-6 mEq/L. Symptoms of hypermagnesemia include decreased patellar reflexes, respiratory depression above 8-10 mEq/L, and potential cardiotoxicity at any level. Management involves monitoring magnesium levels, administering calcium gluconate to counteract magnesium's effects, and maintaining adequate urine output.

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    Hypermagnesemia

    Uploaded by

    Nader Smadi
    776 views5 pages
    The document discusses hypermagnesemia, or high magnesium levels, which can occur in patients receiving magnesium sulfate. It notes that toxicity may begin around a magnesium level of 4 mEq/L, while most patients receiving magnesium sulfate for preeclampsia prevention are maintained between 4-6 mEq/L. Symptoms of hypermagnesemia include decreased patellar reflexes, respiratory depression above 8-10 mEq/L, and potential cardiotoxicity at any level. Management involves monitoring magnesium levels, administering calcium gluconate to counteract magnesium's effects, and maintaining adequate urine output.

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    The document discusses hypermagnesemia, or high magnesium levels, which can occur in patients receiving magnesium sulfate. It notes that toxicity may begin around a magnesium level of 4 mEq/L, while most patients receiving magnesium sulfate for preeclampsia prevention are maintained between 4-6 mEq/L. Symptoms of hypermagnesemia include decreased patellar reflexes, respiratory depression above 8-10 mEq/L, and potential cardiotoxicity at any level. Management involves monitoring magnesium levels, administering calcium gluconate to counteract magnesium's effects, and maintaining adequate urine output.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT or read online from Scribd
    Download as ppt
    776 views5 pages

    Hypermagnesemia

    Uploaded by

    Nader Smadi
    The document discusses hypermagnesemia, or high magnesium levels, which can occur in patients receiving magnesium sulfate. It notes that toxicity may begin around a magnesium level of 4 mEq/L, while most patients receiving magnesium sulfate for preeclampsia prevention are maintained between 4-6 mEq/L. Symptoms of hypermagnesemia include decreased patellar reflexes, respiratory depression above 8-10 mEq/L, and potential cardiotoxicity at any level. Management involves monitoring magnesium levels, administering calcium gluconate to counteract magnesium's effects, and maintaining adequate urine output.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as PPT or read online from Scribd
    Download as ppt
    of 5

    Hyperm agnesemia

    Immediate Questions
    A. What dose of magnesium sulfate was the
    patient receiving? Most patients receive 1-2
    MgSO4 IV every hour. This dose is regulated
    by following serum levels and changes in
    physical examination such as decreased
    patellar reflexes, which are suggestive of
    high serum magnesium levels. Magnesium
    sulfate used in patients with premature
    labor may require doses of up to 3g/h.
    Immediate Questions
    B. What was her last serum magnesium
    level? Clinically significant
    hypermagnesemia can begin to be seen at
    levels as low as 4 mEq/L. Most patients who
    are being maintained on magnesium sulfate
    for the prevention of eclampsia will be held
    at 4-6 mEq/L.
    Management
    1. Magnesium. Toxicity may be seen at
    levels as low as 4 meq/l. Most patients
    are maintained at 4-6 meq/l to prevent
    eclampsia. Respiratory depression can
    appear at magnesium levels of 8-10
    meq/l, and cardiotoxicity, while not
    usually seen until levels exceed 10
    meq/l, can occur at any serum
    magnesium level.

    2. Electrocardiogram. Obtain a baseline


    study. Hypermagnesemia may manifest
    itself as a shortened Q-T interval up
    Management
    3. Calcium gluconate. Administer 1 g IV
    over approximately one minute, while
    waiting for serum magnesium and
    electrolyte values. If calcium
    administration improves the patient's
    status, then magnesium toxicity is most
    likely the correct diagnosis: magnesium
    is a calcium antagonist, and calcium
    should reverse its toxicity.
    4. Urine output. Magnesium is secreted
    by the kidneys. Urine output should be
    maintained at a minimum of 30 mL/h.

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