PEDIA Gastro
PEDIA Gastro
PEDIA Gastro
Disorder
OBJECTIVES
1. Explain important concepts of the
Gastrointestinal system
2. Describe the pathophysiology, clinical
manifestation, collaborative care, and
medical/surgical therapy of GI disorders
3. Plan nursing care for a child with a
gastrointestinal disorder
4. Implement nursing care for a child with a
gastrointestinal disorder
5. Analyze ways that nursing care of a child THE DIGESTIVE SYSTEM
with a gastrointestinal disorder can be more
family-centered Laboratory Studies And Diagnostic Tests
6. Integrate knowledge of gastrointestinal
Stool examination
disorders with nursing process to achieve
Complete blood count
quality child nursing care
Hematocrit – 37% - 47%
Hemoglobin – 12mg% - 16mg%
RBC – 4.0 – 5.5/cu. mm
WBC – 5 – 10 /cu. mm
Differential count
Neutrophils – 50 – 80%
Eosinophils – 0 – 5%
Monocytes – 2 – 10%
Lymphocytes – 25 – 50%
Platelet count – 140 – 400 10^3/cu
Laboratory Studies
Abdominal ultrasound
Computed tomography
Upper GI Imaging
Upper GI Series
Small-bowel series
Barium or Meglumine Diatrizoate
OVERVIEW OF ANATOMY AND (Gastrografin) swallow
PHYSIOLOGY OF THE GI SYSTEM Barium Enema (Lower GI series)
Upper GI endoscopic procedure
(Esophagogastroduodenoscopy)
Fiber-optic Testing
Lower GI endoscopic procedure
(Proctosigmoidoscopy, Colonoscopy)
Gastric Analysis
Gastroesophageal reflux disease (GERD Anticholinergic
[chalasia]) Beta-adrenergic blockers
Calcium channel blockers
Nitrates
Theophylline
Diazepam
Tight, restrictive clothing
Bending, straining
Hiatal Hernia
Is a neuromuscular disturbance
Nissin Fundoplication
Cleft lip
Cleft Palate
Complications
Cause Speech defects
Hereditary Dental and orthodental problems
Environmental Nasal defects
Teratogenic effect Alteration in hearing
Shock, guilt and grief for the parents
Assessment Increased risk of aspiration
abdominal distention URTI
difficulty swallowing Otitis media
involves a notched upper lip border
Diagnostic Findings
Ultrasonography
Medical Management
Cheiloplasty (cleft lip repair surgery)
Breck – feeder
Haberman feeder
Preoperative Nursing Care
1. Assess degree of cleft and ability to
suck
2. Provide postoperative feeding
instructions
3. Encourage parents to verbalize fears,
concerns, negative emotions
4. Facilitate grief responses of shock,
denial, anger, and mourning
5. Encourage touching, holding, cuddling,
and bonding
6. Provide parents with pictures of other
children before and after surgical repair
7. Parents of children with a cleft palate must
be alert to the signs of infection; they need
to report pharyngeal infection
Hirschsprung's disease
(aganglionic megacolon)
Congenital anomaly resulting from an
absence of ganglion cells in colon
Therapeutic Management
1. Placed on an extremely low
phenylalanine formula (Lofenalac)
2. A dietitian may recommend a small Cause
amount of milk in the infant's diet every Familial
day Incidence is higher in children with
Down syndrome and genitourinary
abnormalities
Rectosigmoid region is most commonly
affected
Absence of autonomic parasympathetic
ganglion cells – myenteric and
submucosal plexus are absent
Caused by an abnormal gene on
chromosome 10
Defecation Reflex
a Local reflexes
b Parasympathetic reflexes
Nursing Management
Clinical manifestations in newborns:
Failure to pass meconium stools within 24
hours
Liquid or ribbonlike stools
Reluctant to ingest fluids
Abdominal distention and easily
palpable stool masses Monitor urine specific gravity
Bile-stained emesis or fecal vomiting monitor electrolytes
assess hydration status
Clinical manifestations in infants:
Failure to thrive Postoperative Nursing Care
Constipation Prepare child for surgery and temporary
Abdominal distention placement of colostomy
Vomiting Administer antibiotics as ordered
Episodic diarrhea Monitor VS
measure abdominal girth
Clinical manifestations in toddlers: assess surgical site for redness,
Chronic constipation swelling, drainage after surgery
Foul-smelling stools
Abdominal distention
Visible peristalsis
Palpable fecal mass
Malnourishment
Signs of anemia and hypoproteinemia
Celiac Disease
Overview
• a genetic GI malabsorption condition
• known as gluten-induced enteropathy
• inability to tolerate foods containing
gluten
• inability to fully digest gliadin and
glutenin or protein component
Cause:
• “unknown”
• Genetic predisposition
• possibly influenced by environmental
factors
• an immunologic abnormality
Exposed to gluten
Damage in Intestinal mucosa
Laboratory Studies and Diagnostic Tests • Importance of lifelong compliance with
• Flat mucosal surface, absence or atrophy dietary modifications and follow-up
of villi, and deep crypts visible on biopsy medical care
of small intestine
• Steatorrhea on analysis of 72-hour Intussusception
quantitative fecal fat study Is the invagination of one portion of the
• Presence of serum antigliadin antibody intestine into another
(AGA) and reticulin antibody levels are occurs mostly in the ILEOCECAL
elevated VALVE
Nursing Diagnosis
• Altered nutrition; less than body
requirements, related to malabsorption of
food
Therapeutic Management
Causes:
Meckel’s diverticulum
Polyps
Intussusception
Obstruction
Edema
Venous and
arterial
obstruction
GI bleeding and
fluid loss
Ischemia
Necrosis
Hypertrophy of Peyer’s patches Perforation
Sepsis
Nursing Assessment
Distended and tender abdomen with a
palpable, sausage-shaped abdominal mass
If necrosis has occurred, children
generally have
an elevated temperature,
peritoneal irritation
Bowel tumors Increased WBC,
and often a rapid pulse.
Nursing Assessment
Intermittent attacks (15 min.) of colicky Laboratory Studies and Diagnostic Tests
pain…
History - (duration, intensity, frequency,
Emesis containing bile or fecal material
description, associated manifestations)
After approximately 12 hours, children Sonogram (ULZ)
develop blood in stool, containing mucus
Increased WBC
described as a "currant jelly" appearance.
Abdominal X ray, Ultrasound,
Computerized Tomography
Therapeutic Management
Surgery
Manual Reduction
Resection
Manual Reduction
Nursing care
Pain related to abnormal abdominal
peristalsis
1. Infants need to be held and rocked and
comforted
Clinical Manifestations
Begins at 4-6 weeks of age
Projectile vomiting
Sour but contains no bile
No presenting illnesses
Gastric peristaltic movement passing from
Left to Right across the abdomen
Round and firm olive size lump
Biliary Atresia
Causes:
Prenatal period
Unknown
Viruses
Toxins
Chemicals
Overview:
A progressive inflammatory process that causes
both intrahepatic and extrahepatic bile duct fibrosis.
Nursing Assessment
1. Healthy - appearing infant at birth
2. Jaundice occurs within 2 weeks to 2
months
3. Acholic stools: puttylike, clay-colored
stools
4. Abdominal distention and hepatomegaly
5. Increased bruising of the skin,
prolonged bleeding time
6. Intense itching
7. Tea-colored urine
8. Malnutrition and growth failure
Diagnostic tests
Liver biopsy
Ultrasound
Medical Management
Kasai Procedure Therapeutic Management
1. Weigh daily
Surgery – for temporarily correct
2. Administer TPN with or without
obstruction and supportive care
intralipids as ordered
3. Administer fat-soluble vitamins A, D, E,
and K as ordered
4. Monitor stool pattern
5. Establish an open, caring relationship
with family
6. Refer parents to support groups