Traumatic Brain Injury

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TRAUMATIC BRAIN

INJURY

By
M.SATWIK
Head trauma is the leading cause
of death in patients with trauma.
Traumatic brain injury (TBI)
is a non-degenerative, non-
congenital insult to the brain
from an external mechanical
force, possibly leading to
permanent or temporary
impairment of cognitive,
physical, and psychosocial
functions, with an associated
diminished or altered state of
consciousness.
A TBI can be focal or diffuse, meaning damage may
be isolated to one specific area of the brain in focal
injuries or wide spread in case of diffuse injuries.

Types:
CLOSED BRAIN INJURY
Without the skull being broken or penetrated and
the brain has not been exposed.
Eg: Acceleration-Deceleration movement
OPEN BRAIN INJURY
Open or penetrating head injury.
TRAUMATIC BRAIN INJURIES
COUP AND CONTRECOUP INJURY
COUP INJURY: sudden hyperextension of the
head and neck forces the brain forward against the
skull.

CONTRECOUP INJURY: hyperflexion of


the head and neck forces the brain backward
against the skull.
WHIPLASH INJURIES
Consequences of whiplash
injuries:
Aetilogy:
Falls
RTAs
Violence
Gunshot injuries
Child abuse
Shaken baby syndrome
Sport injuries
Scoccer
Boxing
Pathophysiolog
y
TBI may be divided into
Primary injury — induced by mechanical
forces and occurs at the moment of injury.
Secondary injury — not mechanically
induced.
Systemic insults due to TBI are ⬆️
intracranial pressure & ⬇️cerebral
perfusion pressure.
Clinical features:
loss of consciousness (few minutes - seconds)
Memory or concentration problems
Headache
Dizziness
Loss of balance
Nausea
Vomiting
Difficultly Sleeping
loss of consciousness (few minutes - hours)
Slurred speech
Loss of coordination
Weakness
Numbness
Brain injury: LUCID INTERVAL:
The period of time between regaining
Can result in hypoxia or hypertension consciousness after a short period of
unconsciousness, resulting from a head injury
Grading is done by GCS and deteriorating after onset of neurological
Minor ——15/15 without loss of consciousness signs and symptoms caused by that injury.
Mild ——14/15 with loss of consciousness
Moderate—9-13
Severe ——<8
GLASGOW COMA SCALE:
Eye opening
Verbal response
Motor response
Brain injury is of two types:
1. Primary brain injury
2. Secondary brain injury
PRIMARY BRAIN INJURY:
The 2 main mechanisms are contact and acceleration-deceleration.
Contact injuries result in skull fractures and surface contusions.
Acceleration-deceleration injuries results in shear, tensile and compressive strains.
1. Concussion
Mild
Colorado classification
Type 1: confusion
Types 2: amnesia
Types 3: loss of consciousness
Chronic traumatic encephalopathy aka Post Concussion Syndrome
1. Diffuse Axonal Injury
Most severe
Shearing force between grey and white matter
Investigation of choice - MRI ( punctuate hemorrhages at
grey and white mater junction Corpus callosum.
Worst prognosis.
1. Intracranial hemorrhages
Most common cause of death
4 types
Epidural hemorrhage
Subdural hemorrhage
Subarachnoid hemorrhage
Intraparenchymal hemorrhage
1. Extra cranial hemorrhages
Scalp lacerations
Nasal injuries
Facial and neck injuries
EPIDURAL HEMORRHAGE:
High velocity impact
Usually caused by temporal bone fracture and rupture of middle meningeal
artery
Investigation of choice: NCCT
BICONVEX LENS SHAPED BLEED (bleeds between skull and dura)
Management:
Craniotomy
>30cc clot size
> 5mm midline shift
> 1.5cm thickness
Burr hole
Sx done at site of bleed
If we can’t localise site of bleed do Sx on side of pupillary dilatation
False localising sign: (KERNOHAN NOTCH PHENOMENON)
Pupillary dilatation
6th CN palsy
Hemiparesis( ipsilateral to lesion)
B/L plantar extension
SUBDURAL HEMORRHAGE:
Trauma is most common cause
Rupture of bridging veins in sub dural space
Investigation of choice: NCCT
CONCAVO-CONVEX BLEED (crescentric)
Bleeds between dura and arachnoid
Management:
Sx—craniotomy and burr hole
>1cm thickness
>5mm midline shift
Fall in GCS >2 points
Fixed/delated pupil
ICP > 20 mmhg
Extent of brain damage more in SDH>EDH
SUBARACHNOID HEMORRHAGE:
Trauma is most common cause
Rupture of berry aneurysm
Conservative management
INTRA PARENCHYMAL
HEMORRHAGE:
Most common traumatic type
Temporal > frontal region
SECONDARY BRAIN INJURY:
occurs due to ⬆️ICT which is due to edema.
Compounded by hypoxia, hypercapnia, hypotension.
Monro kellie doctrine -states that brain has tremendous capacity to
compensate, but once the point of decompensation is met, there
can be sudden decompression and hermination of brain.
Cerebral perfusion pressure
Complications:
Altered consciousness
Coma
Vegetative state
Minimally conscious state
Locked in syndrome
Investigations:
Imaging tests done are
Infections
CT, MRI Blood vessel damage leads
to stroke, blood clots
Nerve damage
Management: Mx of mild head injury
Adequate Oxygenation Evaluate
Adequate perfusion by maintaining SBP> 100 Do CT If indicated
mmhg Discharge if GCS 15/15
Avoid hyperglycaemia Normal CT
IV mannitol Not under influence of alcohol/drugs
Hyperventilation Accompanied by responsible adult
Barbiturate coma Mx of moderate head injury
Hypothermia Admit
Steroids CT
Prophylactic anti epileptics
monitor and do serial CECT
Diuretics
Mx of severe head injury GCS<8
Goals:
Intubation
ICP: 20-25mmhg
NCCT
CPP: >60mmhg
PaO2: >100 mmhg Brain death
PbtO2(brain tissue oxygen tension)>15mmhg GCS=3
Serum Na+ : 135-145 Non reactive pupils
Glucose: 80-180mg/dl Absent brain stem reflex
Physiotherapy No spontaneous ventilatory effort
Thank you

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