ECG Study Guide
ECG Study Guide
ECG Study Guide
- Electrode placement
PQRST Significance
Axis determination
- QRS is most important to determine
- Normal: -30 to +90, downward and to the left
- Left axis deviation: -30 to -90
o QRS is + in Lead I and – in lead aVF
o Causes: inferior wall MI, LV hypertrophy, LBBB, ventricular ectopy
- Right axis deviation: +90 to +180
o QRS is – in Lead I and + in aVF
o Causes: lateral wall MI, RBBB, right ventricular hypertrophy, dextrocardia, pulmomary
embolism
o
o Causes: MI, CAD, aortic stenosis, cardiomyopathies, myocarditis, hyperkalemia, dig toxicity
- RBBB
o Large R wave in V1, V2, slurred S wave in V1, V5, V6
o
o Causes: 2/2 HTN, MI, myocarditis, COPD, cor pulmonae, RHF, pulmonary embolism
Rhythm Patterns
- NSR: 60-100
o P before QRS followed by T wave
- Sinus brady: <60
o SSS, BB, hypothermia, hypoglycemia, hypothyroid, CAD
- Sinus tachy : >100
o Fever, anxiety, hyperthyroid, albuterol, dobutamine, anemia, pain
Atrial Rhythms
- Premature atrial contraction (PAC)
o Ectopic focus within atria conduction to ventricles
o Frequent= palpiatations
o
- Atrial Fibrillation
o Irregularly irregular
o Rapid firing within atria
o No P waves, no PR interval
o s/s= SOB, dizy, fatigue, chest pain
o
- Atrial Flutter
o More organized
o “sawtooth”
o
- Supraentricular tachycardia
o Originates in AV node
o Narrow complex tachycardia appearing rapid and regular
Ventricular rhythms
- Premature ventricular contraction (PVC)
o Early impulse originating from ventricle
o P wave is absent, QRS is wide and bizarre
o MI, cardiomyopathy, electrolytes (hypokalemia), hypoxia
o
- Ventricular tachycardia
o Abnormal electrical signals from ventricle
o Loss of pulse and consciousness
o ACLS
o
- Ventricular fibrillation
o High frequency disorganization excitation of myocardium
o No P wave or QRS, instead fibrillatory waves
o Sudden cardiac death defib
AV blocks
- 1st degree AV block
o rhythm with prolonged PR interval >.2
o Well trained atheletes, inferior MI, hypokalemia
o s/s: dizzy, dyspnea, fatigue
o
- 2nd AV block (Mobitz I or wenchebach)
o Gradual increase in PR interval until blociked impulse and subsequent PR is shorter
o
nd
- 2 degree AV block II (Mobitz II)
o Constant PR interval leading to dropped QRS
o Dixzzy, syncome, hypotension or brady
o ? pacemaker
o
rd
- 3 degree AV block (complete heart block)
o No conduction through AV leading to AV dissociation
o Regular P-P interval, regular R-R, failure of P and QRS
o AMS, SOB, CP, syncope
o Pacemaker
Pacing
- Oversensins: device does not initiate when it should
- Undersensisng: fails to turn off
- Capture: sufficient impulse
- Atrial pacing: pacing prior to P wave
o
- Ventricular pacing: pacing prior to QRS
o
- AV pacing: pacing prior to P and prior to QRS
o
Digitalis toxicity
- Parasoxysmal atrial tachycardia
- Accelerated junctional rhythm
- Bidirectional ventricular tachycardia
- AV blocks
Hypothermia
- Bradyarrythmias
- Prolonged PR, QRS, QT
- Osborn and J waves
-
Pericarditis
- Stage I: Diffuse, concave upward ST segment elevation in most leads and subtle or overt PR depression
- Stage II: ST segment elevation and PR depression return to baseline; may have normal or flattened T
waves
- Stage III: T waves are inverted; the ECG is otherwise normal
- Stage IV: ECG returns back to normal
Pulmonary Embolism
- Tachycardia
- S1Q3T3 pattern
- Classic findings of right heart strain
o Tall, peaked p waves in Lead II
o Right axis deviation
o Right bundle branch block