Acute Bronchopneumonia
Acute Bronchopneumonia
Acute Bronchopneumonia
ACUTE BRONCHOPNEUMONIA
Acute inflammation of the walls of the smaller bronchial tubes, with varying
amounts of pulmonary consolidation due to spread of the inflammation into
peribronchiolar alveoli and the alveolar ducts; may become confluent or may be
hemorrhagic.
Patient’s Profile
Past History
Hypertension
Diabetes Mellitus
Bronchial Asthma
(+ PTB 2009- 6 months)
ANATOMY AND PHYSIOLOGY
The main bronchi branch many times to form the tracheobronchial tree. Each main
bronchus divides into lobar bronchi as they enter their respective lungs. The lobar
(secondary) bronchi, two in he left lung and three in the right lung, conduct air to each
lobe. The lobar bronchi in turn give rise to segmental (tertiary) bronchi, which extend to
the bronchopulmonary segments of the lungs.
The bronchi continue to branch many times,
finally giving rise tobronchio les. The
bronchioles also subdivide numerous times to
give rise to terminal bronchioles, which then
subdivide into respiratory bronchioles. Each
respiratory bronchiole subdivides to form
alveolar ducts, which are like long, branching
hallways with many open doorways. The
doorways open into alveoli, which are small air
sacs. The alveoli become so numerous that the
alveolar duct wall is little more than a succession
of alveoli. The alveolar ducts end as two or three
alveolar sacs, which are chambers connected to
two or more alveoli. There are about three
million alveoli in the lungs.
The bronchioles are very small airways that extend from the bronchi to the
alveoli. The bronchioles are made up of smooth muscle cells and are smaller than 1
millimeter in diameter. The bronchioles do not have glands or cartilage. The epithelial
cells of the bronchioles are cuboidal in shape.