Environment Quick News: A Monthly Report From EPRI's Environment Sector October 2007
Environment Quick News: A Monthly Report From EPRI's Environment Sector October 2007
Environment Quick News: A Monthly Report From EPRI's Environment Sector October 2007
Program members can use their EPRI.com ID and password to download Acrobat PDF files of EPRI technical
reports. For assistance, contact the EPRI Customer Assistance Center at (800) 313-3774.
We would like to thank all members who participated in the Cleveland advisory meetings, especially with
regard to our priority setting and planning for the 2009 offering. From our perspective, and hopefully yours, the
input we received was extremely valuable and we appreciate the thought and effort everyone expended in our
first attempt at this process. We are currently sorting through the results and will be providing feedback over
the next several weeks.
In this issue of Quick News, note that there are several articles that pertain to the issue of arsenic. This is an
integrated research effort that involves several programs in Air, Water and Land/Groundwater. As this issue
becomes more front burner, we intend to have our research results available as part of any regulatory process
as well as help those who are dealing with arsenic remediation.
Thank you again for your energy and enthusiasm in Cleveland. We look forward to seeing you all again in
Scottsdale next February or at one of our upcoming technical meetings.
ENVIRONMENT SECTOR
• Physiologically based pharmacokinetic (PBPK) modeling. Researchers are refining comparable models
that predict dose to target tissues based on the kinetics (chemical reaction rates) of inorganic arsenic and its
metabolites in mice and humans.
• Cancer modeling. The research paper presents a hierarchy of cellular responses across a range of doses of
inorganic arsenic. With each dose, a cell enters a different state with new properties that are reflected by the
alteration of genes in key signal pathways. Since—regardless of exposure route—inorganic arsenic acts
through interaction with proteins rather than DNA, a model of inorganic arsenic carcinogenesis must describe
both mutagenic and cell regulatory changes.
• Literature review of genomic responses to arsenic. Published studies have reported a consistent pattern
of gene changes involving seven categories of cellular response to inorganic arsenic. In general, the effects of
inorganic arsenic can be seen as highly specific direct interactions of arsenite (the trivalent form of arsenic)
with critical proteins, superimposed on a background of chemical stress. At low exposure concentrations
(0.01 μM), cells apparently enter an adaptive state. At high concentrations (> 5 μM), they exhibit increased
toxicity shown by cell cycle stasis, apoptosis (programmed cell death), and inhibition of DNA repair. An
observed reversal of gene responses at concentrations below 0.1 μM supports a nonlinear threshold model.
Reported gene changes are consistent with epidemiologic and in vitro effects of arsenic seen under different
exposure conditions.
• Arsenic genomic dose-response studies. The bladder is one of the human target tissues for arsenic
carcinogenesis. The bladder was selected as the initial focus for study for technical reasons. Assuming
adequate resources, the lung as a target tissue will be evaluated next. In work reported here, researchers
found no significant gene expression changes in the bladder tissue of a low-dose (0.05 mg/l) group of mice
exposed to inorganic arsenic in drinking water, compared with controls. Significant changes were seen in the
high-dose group (50 mg/l). A 12-week follow-up study employing multiple concentrations will document
changes in gene expression related to dose over time.
• Application of biologically based dose-response model to arsenic low-dose nonlinear cancer risk
assessment. As an alternative to a linear extrapolation approach, a nonlinear margin-of-exposure approach
that maintains a biological basis will be developed. This margin-of-exposure approach will be built on the
relatively uncomplicated biologically based dose-response (BBDR) model developed by this project. The
BBDR model incorporates multiple gene expressions associated with events that must occur for tumors to
develop as a result of inorganic arsenic exposure.
This research is being performed under a Memorandum of Understand among EPRI, The Hamner Institute
(formerly CIIT Centers for Health Research), and USEPA. For more information, contact Leonard Levin, (650)
855-7929, [email protected].
Panelists will meet periodically with investigators as the project progresses. Drs. Barchowsky, Rossman, and
Styblo served with EPRI consultant Dr. Janice Yager on the USEPA Science Advisory Board Arsenic Review
Panel responsible for the June 2007 report Advisory on EPA’s Assessments of Carcinogenic Effects of Organic
and Inorganic Arsenic: A Report of the US EPA Science Advisory Board. For more information, contact
Leonard Levin, (650) 855-7929, [email protected].
Program 91: Assessment Tools for Ozone, Particulate Matter, and Haze
New EPRI Project Will Link AMSTERDAM and WARMF Models
Air quality models, such as the EPRI-funded AMSTERDAM model, and watershed models, such as EPRI’s
WARMF, are typically applied independently by different groups to simulate atmospheric pollution and
land/water ecosystem impacts, respectively. The end point of the air models is generally the starting point of
the watershed models, but little effort has been made to link them in a rigorous manner. Linkages would allow
researchers to follow the path of pollutants emitted into the air, their transport and transformations in the
atmosphere, their deposition to the earth, and their fate in water bodies/watersheds after interactions with other
anthropogenic and biogenic matter. The linked models could be used to evaluate the water quality
improvement that would result from the reduction of specific air emissions from specific power plants—a
capability that does not currently exist. Therefore, EPRI has undertaken a new Technology Innovation (TI)
project to develop an interface between AMSTERDAM and WARMF. Design of the interface will address the
following issues:
• specification of the spatial domain and time period for testing the interface
• spatial scale and projection conversions
• temporal resolution
• file format specifications
• mapping of chemical species from AMSTERDAM to WARMF
• mapping of meteorological model results used in AMSTERDAM for use in WARMF
• deposition and air concentration results from AMSTERDAM for use in WARMF
The design, coding, testing, and documentation of the interface is expected to be complete by the end of 2008.
For more information, contact Eladio Knipping, (650) 855-2592, [email protected], or Robert Goldstein, (650)
855-2593, [email protected].
EPRI Attends Planning Meeting for California Air Quality/Climate Change Field Study
EPRI researcher Eladio Knipping attended a planning meeting held Sep. 21 in Sacramento, CA, sponsored by
the National Oceanic and Atmospheric Administration, the California Air Resources Board, and the California
Energy Commission. The purpose of this public meeting was to begin the planning process for a possible
combined air quality and climate science field study in California in 2010. EPRI is interested in learning more
about the field study, determining how EPRI might participate or collaborate in the funding or execution of
studies, and exploring which issues to be addressed in California might be of broader interest to EPRI
members throughout the nation. For more information, contact Eladio Knipping, (650) 855-2592,
[email protected].
• P.A. Ruiz, J.E. Lawrence, J.M. Wolfson, S.T. Ferguson, T. Gupta, C.-M. Kang, and P. Koutrakis.
“Development and evaluation of a photochemical chamber to examine the toxicity of coal-fired power plant
emissions.” Inhalation Toxicology 19 (8): 597-606 2007. When investigating the toxicity of individual particle
sources, it is important to consider the contribution of both primary and secondary particles. The authors
present the design of a new photochemical chamber that can be used to form secondary sulfuric acid particles
from diluted coal-fired power plant emissions. The chamber is a relatively small, well-mixed flow reactor that
can fit in a mobile reaction laboratory. It produces high concentrations of hydroxyl radical (OH) from the
photolysis of ozone (O3) in the presence of water vapor. A pilot chamber was tested in the laboratory, using
mixtures of NO and SO2 in air, at concentrations approximately 100 times lower than those in power plant
stack emissions. This chamber was able to oxidize about 20% of the SO2, thereby producing 1350 μg m−3 of
H2SO4 particles. Further tests showed that increasing O3 concentrations and residence time increased the
H2SO4 production. A field chamber was built subsequently and used in a toxicological study. Diluted coal-
fired power plant emissions were introduced in the chamber. Over 19 days of exposure, the chamber, on
average, converted 17% of the supplied SO2 emissions and produced an average of 350 μg m−3 of H2SO4
particles. Particle losses were determined for the pilot chamber, using artificial particles whose size ranged
from 50 to 1000 nm. The determined losses ranged from 21% to 42%, with no trend between the amount of
particle loss and particle size. Losses for the field chamber, estimated using model calculations, were found
to be similar to those of the pilot chamber.
• P.A. Ruiz, T. Gupta, C.-M. Kang, J.E. Lawrence, S.T. Ferguson, J.M. Wolfson, A.C. Rohr, and P. Koutrakis.
“Development of an exposure system for the toxicological evaluation of particles derived from coal-fired power
plants.” Inhalation Toxicology 19 (8): 607-619 2007. To investigate the toxicity of particles originating from
coal-fired power plants, it is necessary to consider the effects of both primary particles and secondary
components formed in the air through atmospheric reactions. The authors describe a new exposure system
that can be used to expose animals to both directly emitted particles and secondary particles. The system
consists of three main components. The first is a sampling system to continuously collect and dilute power
plant stack emissions. The second is a reaction laboratory that contains reaction chambers to simulate
atmospheric reactions. The following atmospheric reactions were simulated: (1) the oxidation of sulfur
dioxide to form sulfuric acid, (2) the neutralization of sulfuric acid by ammonia, and (3) the reaction of α-pinene
with ozone to form secondary organic aerosol. Using these chambers with the diluted emissions, different
typical atmospheric scenarios can be simulated. The final component is a mobile toxicology laboratory where
animals are exposed to the resulting test aerosols. The authors report the characteristics of the test aerosol
exposures obtained at a coal-fired electric power plant. Particle exposures were characterized for
concentrations of mass, elements, elemental carbon, organic species, inorganic ions, strong acidity, particle
number, and size distributions. Mass concentrations ranged from a few micrograms per cubic meter for a
−3
scenario of primary emissions only, to about 250 μg m for the most complex scenario. The authors show
that the different scenarios produced a large variation in the composition of the test aerosol, thus potentially
changing the toxicity of the emissions.
• General analysis results for the WECC region, including an assessment of the impact of climate policy on
power prices, regional emissions, and the cash flows to new and existing fossil and non-emitting generation.
These products will be available to all project funders.
• Utility-level results mapping the regional results to the assets of each participating utility, delivered in a private
presentation to each participating utility’s analysts and management.
The price to participate in this project is $10,000 for entities with capacity of less than 1,500 MW, $25,000 for
entities with 1,501–4,000 MW total capacity, and $45,000 for entities with more than 4,000 MW total capacity.
Companies that fund any Environment program can use Tailored Collaboration (TC) funds for up to half their
price. Six members of WECC have so far expressed strong interest, and others are encouraged to participate
to increase the project’s scope and usefulness. For more information, contact Vic Niemeyer, (650) 855-2744,
[email protected].
Area News
Newest Climate Briefs Discuss Natural Climate Variability, Economic Implications of Carbon Cycle
Uncertainty
The most recent in EPRI’s series of Climate Briefs are as follows:
• Progress in Understanding Natural Climate Variability (1014807). With uncertainty in the degree of natural
climate variability, furthering our understanding and narrowing that uncertainty is critical in discerning the
human influence on the observed global climate change. To help address these uncertainties and to identify
ways to advance the state of knowledge regarding natural climate variation, EPRI, along with both the U.S. and
• Physiologically based pharmacokinetic (PBPK) modeling. Researchers are refining comparable models
that predict dose to target tissues based on the kinetics (chemical reaction rates) of inorganic arsenic and its
metabolites in mice and humans.
• Cancer modeling. The research paper presents a hierarchy of cellular responses across a range of doses of
inorganic arsenic. With each dose, a cell enters a different state with new properties that are reflected by the
alteration of genes in key signal pathways. Since—regardless of exposure route—inorganic arsenic acts
through interaction with proteins rather than DNA, a model of inorganic arsenic carcinogenesis must describe
both mutagenic and cell regulatory changes.
• Literature review of genomic responses to arsenic. Published studies have reported a consistent pattern
of gene changes involving seven categories of cellular response to inorganic arsenic. In general, the effects of
inorganic arsenic can be seen as highly specific direct interactions of arsenite (the trivalent form of arsenic)
with critical proteins, superimposed on a background of chemical stress. At low exposure concentrations
(0.01 μM), cells apparently enter an adaptive state. At high concentrations (> 5 μM), they exhibit increased
toxicity shown by cell cycle stasis, apoptosis (programmed cell death), and inhibition of DNA repair. An
observed reversal of gene responses at concentrations below 0.1 μM supports a nonlinear threshold model.
Reported gene changes are consistent with epidemiologic and in vitro effects of arsenic seen under different
exposure conditions.
• Arsenic genomic dose-response studies. The bladder is one of the human target tissues for arsenic
carcinogenesis. The bladder was selected as the initial focus for study for technical reasons. Assuming
adequate resources, the lung as a target tissue will be evaluated next. In work reported here, researchers
found no significant gene expression changes in the bladder tissue of a low-dose (0.05 mg/l) group of mice
This research is being performed under a Memorandum of Understand among EPRI, The Hamner Institute
(formerly CIIT Centers for Health Research), and USEPA. For more information, contact John W. Goodrich-
Mahoney, (202) 293-7516, [email protected].
• Aaron Barchowsky, PhD, Associate Professor, Department of Environmental and Occupational Health,
Graduate School of Public Health, Pittsburgh, PA
• Barbara Beck, ScD, DABT, Gradient Corporation, Cambridge, MA
• Samuel Cohen, MD, PhD, Professor of Pathology and Microbiology, University of Nebraska Medical Center,
Omaha, NE
• Toby Rossman, PhD, Professor of Environmental Medicine, School of Medicine, New York University, Tuxedo,
NY
• Miroslav Styblo, PhD, Research Associate Professor, Department of Nutrition, University of North Carolina,
Chapel Hill, NC
Panelists will meet periodically with investigators as the project progresses. Drs. Barchowsky, Rossman, and
Styblo served with EPRI consultant Dr. Janice Yager on the USEPA Science Advisory Board Arsenic Review
Panel responsible for the June 2007 report Advisory on EPA’s Assessments of Carcinogenic Effects of Organic
and Inorganic Arsenic: A Report of the US EPA Science Advisory Board. For more information, contact John
W. Goodrich-Mahoney, (202) 293-7516, [email protected].
• The Recent Developments page has been updated with current happenings.
• A new topic on Use of Reclaimed Water for Power Plant Cooling has been added under the Water
Sustainability topic.
• New references on temperature investigations and fish impacts have been added to the Thermal Plume
Assessment – References page.
The eTherm website is available only to members of the Fish Protection Program. For more information,
contact Robert Goldstein, (650) 855-2593, [email protected].
• specification of the spatial domain and time period for testing the interface
• spatial scale and projection conversions
• temporal resolution
• file format specifications
• mapping of chemical species from AMSTERDAM to WARMF
• mapping of meteorological model results used in AMSTERDAM for use in WARMF
• deposition and air concentration results from AMSTERDAM for use in WARMF
The design, coding, testing, and documentation of the interface is expected to be complete by the end of 2008.
For more information, contact Eladio Knipping, (650) 855-2592, [email protected], or Robert Goldstein, (650)
855-2593, [email protected].
• Is the trade effective in reducing the pollutant load (i.e., does the science support it)?
• Does the trade comply with the Clean Water Act (i.e., is it legal)?
• Is the trade attractive (i.e., what are the behavioral and economic outcomes)?
Three existing cross-pollutant trading programs and six proposed programs have been identified, and
researchers are compiling detailed information about each. Researchers have also examined federal and
state programs related to trading, including EPA’s policy—which makes specific provision for cross-pollutant
trades—and policies in Michigan, West Virginia, Colorado, and Pennsylvania. Future work will focus on
methods for cross-pollutant trading; of particular interest are trades related to ammonia involving reductions in
pH and temperature to reduce in-stream un-ionized ammonia concentrations and related toxicity. Modeling
tools that can demonstrate the effectiveness of pollutant load reductions through cross-pollutant trading will
also be considered. For more information, contact Robert Goldstein, (650) 855-2593, [email protected].
Supplemental Project: Assessing Impact of Climate Change on Water Availability for Power
Generation
“Assessing the Impact of Climate Change on Water Availability for Power Generation” (1015484). Climate
change and increasing weather variability can potentially create shortages of high-quality fresh water in many
regions of the United States. The availability of such water is critical for thermoelectric power generation as
well as hydroelectric generation. EPRI has developed an analytical framework that can help individual
companies evaluate the risk that water shortages could affect their power generation capability. EPRI will
customize and apply this framework to evaluate the vulnerability of individual power companies to climate-
induced shortages in water availability. The framework consists of four levels:
• The first level of analysis uses detailed watershed budgets to evaluate water availability. Regional annual and
seasonal inventories of water resources are assembled, and water requirements for electricity generation are
also estimated and compared to other local water demands. Based on this information, conditions under which
significant water availability deficits might occur are identified and analyzed.
• The second level utilizes a mechanistic watershed simulation model and addresses potential impacts of
climate variability (such as droughts and heat waves). Climate scenarios are generated using random or
directed selection of past weather patterns; these scenarios can then be used to identify climate-related risks
to meeting electricity demands. Simulations of strategies (e.g., water sharing agreements and water recycling)
and technologies (e.g., atmospheric cooling and wet cooling tower water vapor capture) to mitigate these risks
can also be evaluated.
• In the third level, outputs from global climate models are used to adjust current weather patterns by applying
predicted average temperature and precipitation changes. The adjusted weather patterns are used to drive the
watershed model and evaluate the ability of different strategies and technologies to mitigate potential risks.
• The fourth level involves adding probabilistic analysis, in which physical watershed modeling is combined with
statistical methods to quantify the risk of climate-induced water shortages. Timescales of up to 80 years can
be evaluated.
EPRI Comments on a Study of Occupational Magnetic Field Exposure and Alzheimer’s Disease
Davanipour Z, et al. “A case-control study of occupational magnetic field exposure and Alzheimer’s disease:
results from the California Alzheimer’s Disease Diagnosis and Treatment Centers.” BMC Neurology, 9 June
2007, 7:13. This case-control study examines the possible association between occupational magnetic field
exposure and Alzheimer’s disease (AD) among patients residing in eight California Alzheimer’s Disease
Diagnosis and Treatment Centers located throughout the state. Cases were 1502 patients, over the age of 65
at first examination, who were diagnosed with probable or definite AD. Controls were 396 similar patients
whose dementia was not related to AD or vascular causes. Subjects were assigned to magnetic field exposure
categories based on their reported primary occupations by one investigator using his expert judgment. Thirty-
two (2.1%) cases and 3 (0.8%) controls had high exposure (average individual exposure > 1 µT or regular
intermittent exposures > 10 µT), while 81 (5.4%) cases and 12 (3.0%) controls had medium exposure (average
individual exposure 0.2–1.0 µT or regular intermittent exposures > 1 µT). High-exposure occupations included
garment workers, pilots, and welders. Logistic regression analyses showed significant risk factors for AD,
including magnetic field exposure, gender, stroke, and age. Combining results from this study with those of
two previous studies by the same authors yielded an odds ratio of 3.7 (p < 0.001) for sewing machine
operators. The authors conclude that, “Elevated occupational MF [magnetic field] exposure was associated
with an increased risk of AD . . . Sewing machine operators . . . are likely to be at increased risk of developing
AD, for some reason. It is important to confirm this statement with targeted studies and, if confirmed, to
determine the etiologically relevant exposure(s).”
EPRI suggests caution in interpreting the authors’ conclusions. Since no information was available on the
length of employment in primary occupations and occupational histories may have been obtained by interviews
with proxies, significant exposure misclassification may have occurred. Increased risk for AD observed among
sewing machine operators could be linked to other exposures in their work environment and not necessarily to
magnetic field exposure. Small numbers of controls in the high- and medium-exposure categories (3 and 12,
respectively) make risk estimates quite unstable. Finally, the authors’ overall conclusion appears to be more in
favor of an association than conclusions reached in recent publications, including an Environmental Health
Criteria monograph on extremely low frequency (ELF) electric and magnetic fields (EMF) by the World Health
Organization (WHO) and a consensus report from an Edinburgh, Scotland workshop on occupational ELF EMF
epidemiology, soon to be a submitted manuscript. According to the workshop report, “although studies of EMF
The BioInitiative Report covers both the extremely low frequency (ELF) EMF associated with electric power
and radio-frequency (RF) EMF from sources such as cellular telephones and RF transmitters. Its main
conclusion is that limits for public EMF exposure set by organizations such as the International Commission on
Non-Ionizing Radiation Protection (ICNIRP) and the Institute of Electrical and Electronics Engineers (IEEE) are
inadequate to protect health and require substantial reduction. For ELF magnetic fields, the report
recommends a more than 800-fold reduction of the ICNIRP limit and a 9,000-fold reduction of the IEEE limit.
For RF fields, a 2,000-fold reduction is recommended.
The current ICNIRP limit for public ELF EMF exposure at power frequency in the United States is 0.83 Gauss
(0.083 millitesla), and the IEEE limit is 9.1 Gauss (0.91 millitesla). The limits for ELF magnetic fields
recommended in the BioInitiative Report “while new ELF limits are being developed and implemented” are 1
milligauss (0.1 microtesla) for “habitable space adjacent to all new or upgraded power lines” and 2 milligauss
(0.2 microtesla) for all other new construction. The report also recommends a 1-milligauss limit for “existing
habitable space for children and/or women who are pregnant.” The recommended limit for outdoor RF
exposure is 0.1 μW/cm2.
EPRI notes that, unlike the expert panels that conducted previous EMF health risk evaluations for IARC, the
U.S. National Institute of Environmental Health Sciences (NIEHS), and the World Health Organization (WHO),
the BioInitiative Working Group was not convened by any governmental bodies or recognized health risk
assessment organizations. Moreover, its conclusions, opinions, and recommendations are not consistent with
those reached by previous panels.
All previous assessments recognize the association found in epidemiologic studies between residential ELF
magnetic fields and childhood leukemia and the unexplained nature of this association. When all EMF health
studies are considered together, the risk of leukemia for children with average residential magnetic fields
above 3 to 4 milligauss (0.3 to 0.4 microtesla) is about 1.7 to 2.0 times the risk of children in residences with
lower fields. For such weak epidemiologic associations, supporting data from laboratory studies are usually
critical for establishing a causal link. Long-term studies of ELF magnetic field exposure in rodents are
consistently negative, and there is no biophysically plausible basis for a causal relationship. Nevertheless, the
BioInitiative Working Group concluded that “up to 80% of childhood leukemia may be caused by exposure to
ELF,” an estimate that is unsubstantiated. Moreover, the 1-milligauss limit the working group calls for would
produce a dose in the body between 200 and 2,000 times less than the lowest dose levels expert panels
believe could cause biological effects. Studies of RF exposure and cancer in rodents have also been negative,
and the BioInitiative Report’s recommended limits for RF exposure would produce a very small deposition of
energy in the body.
The BioInitiative Report will likely be subject to further scrutiny and review by the scientific community. Despite
the report’s position, the scientific community has determined that the ICNIRP and IEEE guidelines are
protective against the known effects of ELF and RF exposures. The ICNIRP and IEEE exposure limits for ELF
magnetic fields are designed to prevent nerve stimulation caused by electric fields induced in the body. As
WHO stated in its June 2007 Environmental Health Criteria document with respect to acute neural stimulation
effects of EMF exposure, “International guidelines exist that have addressed this issue. Compliance with these
guidelines provides adequate protection.” RF exposure limits, such as those published by the Federal
Communications Commission (FCC), ICNIRP, and IEEE are designed to protect against tissue heating from
deposition of thermal energy in the body. WHO plans to review the RF literature in coming years.
EPRI’s EMF Health Assessment and RF Safety program continues to conduct research to investigate the
uncertainties surrounding the association of magnetic fields with childhood leukemia and to address priority
issues related to RF exposure, dosimetry, and safety practices. For more information, contact Rob Kavet, (650)
855-1061, [email protected].
Senate Hearings on Advanced Coal Technologies and CO2 Capture and Storage
On August 1, Jeff Phillips of EPRI testified at a hearing on the, “Status of Advanced Coal Technologies and
RD&D Needs to Enable Readiness for Commercial Application”, before the U.S. Senate Committee on Energy
and Natural Resources. Jeff testified that with the development and deployment of advanced coal
technologies with CO2 capture and storage (CCS) technologies, coal power becomes part of the solution to
satisfying both our energy needs and our global climate change concerns. Jeff referenced EPRI’s “Electricity
Technology in a Carbon-Constrained Future” study (also referred to as the PRISM analysis) that suggests that
it is technically feasible to reduce U.S. electric sector CO2 emissions over the next 25 years while meeting the
increased demand for electricity and that the largest single contributor to emissions reduction would come from
the integration of CCS technologies to advanced coal-based power plants coming on-line after 2020. Economic
analyses of scenarios to achieve the study’s emission reduction goals show that a 2030 U.S. energy mix
including advanced coal technologies with integrated CCS results in electricity at a much lower cost than a
2030 energy mix without advanced coal with CCS. He pointed out that a sustained RD&D program at
heightened levels of investment and resolution of legal and regulatory unknowns for long-term geologic CO2
storage will be required to achieve the promise of clean coal technologies. Jeff’s testimony provided details on
the RD&D program and funding developed under EPRI’s CoalFleet for Tomorrow® program. On August 13,
Program 91: Assessment Tools for Ozone, Particulate Matter, and Haze
• Secondary Particulate Health Effects Research (SPHERES): Exploring the Influence of SO2, NOx and Acids
on the Chemistry and Health Effects of PM-2.5
• Regional PM Characterization to Enable Source-Receptor Modeling: Data Collection
• Theoretical, Experimental, and Ambient Studies of Particulate Oligomers and Organic-Nitrogen Species
(TEASPOONS): A Health Impact Perspective
Program 92: Assessment of Air Quality Impacts on Health and the Environment
• Secondary Particulate Health Effects Research (SPHERES): Exploring the Influence of SO2, NOx and Acids
on the Chemistry and Health Effects of PM-2.5
• Integrated Chamber and Panel Study (ICAPS)
• Tri-City Concentrated Ambient Particle Study (Tri-City CAPS)
• Children’s Air Pollution Asthma Study: Part of the ARIES Program
• Detroit Cardiovascular Health Study
• Health Effects of Air Pollution and Particulate Matter Components: Texas ARIES
• Reanalysis of Key PM-Health Dataset
• Sources and Composition of Particulate Exposures (SCOPE) Study
• Toxicological Evaluation of Realistic Emissions of Source Aerosols (TERESA)
Upcoming Events
Environment Sector
Environment Sector and Area Council Advisory Meetings
Feb. 11–13, 2008, Scottsdale, AZ. Contact: Bryan Hannegan, (650) 855-2858, [email protected].
Air Quality
International Aerosol Modeling Algorithms (IAMA) Conference
Dec. 4–7, Davis, CA. Contact: Eladio Knipping, (650) 855-2592, [email protected].
This conference is sponsored by the UC Davis Air Quality Research Center and the California Air Resources
Board. More information is available at the event website.
MGP 2008
Mar. 4–6, 2008, Dresden, Germany. Contact: Andrew Coleman, (650) 855-2249, [email protected] or
James Lingle, (414) 355-5559, [email protected]. More information is available at the event website.
1016103
Together....Shaping the Future of Electricity®