(Q73766127)

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Osteoprotegerin blocks bone cancer-induced skeletal destruction, skeletal pain and pain-related neurochemical reorganization of the spinal cord

scientific article published on 01 May 2000

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10802707%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

Osteoprotegerin blocks bone cancer-induced skeletal destruction, skeletal pain and pain-related neurochemical reorganization of the spinal cord (English)

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6 November 2019

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Honore P

1

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6 November 2019

Luger NM

2

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6 November 2019

Sabino MA

3

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6 November 2019

Schwei MJ

4

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6 November 2019

Rogers SD

5

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6 November 2019

Mach DB

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6 November 2019

O'keefe PF

7

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6 November 2019

Ramnaraine ML

8

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6 November 2019

Clohisy DR

9

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6 November 2019

Mantyh PW

10

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6 November 2019

publication date

1 May 2000

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6 November 2019

Nature Medicine

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6 November 2019

6

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6 November 2019

5

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6 November 2019

521-528

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6 November 2019

Pain causes in 200 patients referred to a multidisciplinary cancer pain clinic

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A randomised phase II study of oral pamidronate for the treatment of bone metastases from breast cancer

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The clinical course of bone metastases from breast cancer

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The role of bisphosphonates in the treatment of painful metastatic bone disease: a review of phase III trials

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Breakthrough pain in cancer patients: pathophysiology and treatment

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Malignant bone pain: pathophysiology and treatment

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Breakthrough pain: characteristics and impact in patients with cancer pain

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Mechanisms and management of bone pain

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Osteoclasts are required for bone tumors to grow and destroy bone

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OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

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Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

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Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

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Neurochemical and cellular reorganization of the spinal cord in a murine model of bone cancer pain

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7 January 2021

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Induction of c-fos-like protein in spinal cord neurons following sensory stimulation

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Substance P receptor (neurokinin-1)-expressing neurons in lamina I of the spinal cord encode for the intensity of noxious stimulation: a c-Fos study in rat

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Towards a mechanism-based classification of pain?

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Skeletal unloading causes resistance of osteoprogenitor cells to parathyroid hormone and to insulin-like growth factor-I.

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An acid sensing ion channel (ASIC) localizes to small primary afferent neurons in rats

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Tumour necrosis factor-α induces ectopic activity in nociceptive primary afferent fibres

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Tumour necrosis factor-alpha mediates carrageenin-induced knee-joint incapacitation and also triggers overt nociception in previously inflamed rat knee-joints

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Substance P- and CGRP-immunoreactive nerves in bone

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Distribution of CGRP-, VIP-, D beta H-, SP-, and NPY-immunoreactive nerves in the periosteum of the rat

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Noradrenergic and peptidergic innervation of the mouse femur bone marrow

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Ascites sarcoma 180, a tumor associated with hypercalcemia, secretes potent bone-resorbing factors including transforming growth factor α, interleukin-1α and interleukin-6

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Characterization of cytokine-induced hyperalgesia

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Contribution of interleukin‐1β to the inflammation‐induced increase in nerve growth factor levels and inflammatory hyperalgesia

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7 January 2021

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Cytokines, nerve growth factor and inflammatory hyperalgesia: the contribution of tumour necrosis factor alpha

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Up-regulation of opioid gene expression in spinal cord evoked by experimental nerve injuries and inflammation

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In situ hybridization histochemistry and immunocytochemistry reveal an increase in spinal dynorphin biosynthesis in a rat model of peripheral inflammation and hyperalgesia

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Dissociation of microglial activation and neuropathic pain behaviors following peripheral nerve injury in the rat

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The effect of site and type of nerve injury on spinal glial activation and neuropathic pain behavior

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Spinal substance P receptor expression and internalization in acute, short-term, and long-term inflammatory pain states

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7 January 2021

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Are there long-term changes in the basal or evoked Fos expression in the dorsal horn of the spinal cord of the mononeuropathic rat?

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Inflammation Increases the Distribution of Dorsal Horn Neurons That Internalize the Neurokinin-1 Receptor in Response to Noxious and Non-Noxious Stimulation

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C-fos expression in rat lumbar spinal cord following peripheral stimulation in adjuvant-induced arthritic and normal rats

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Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

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Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulation

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Rapid endocytosis of a G protein-coupled receptor: substance P evoked internalization of its receptor in the rat striatum in vivo

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Inhibition of Hyperalgesia by Ablation of Lamina I Spinal Neurons Expressing the Substance P Receptor

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The cytoarchitectonic organization of the spinal cord in the rat. I. The lower thoracic and lumbosacral cord

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Identifiers

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10802707%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10802707%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

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