(Q48846848)

16 February 2018

title

Intraneuronal Abeta accumulation precedes plaque formation in beta-amyloid precursor protein and presenilin-1 double-transgenic mice (English)

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16 February 2018

author name string

Wirths O

1

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16 February 2018

Multhaup G

2

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16 February 2018

Czech C

3

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16 February 2018

Blanchard V

4

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16 February 2018

Moussaoui S

5

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16 February 2018

Tremp G

6

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16 February 2018

Pradier L

7

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16 February 2018

Beyreuther K

8

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16 February 2018

Bayer TA

9

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16 February 2018

language of work or name

English

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publication date

1 June 2001

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16 February 2018

published in

Neuroscience Letters

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16 February 2018

volume

306

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16 February 2018

issue

1-2

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16 February 2018

page(s)

116-120

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16 February 2018

Plaque formation in brain transplants exposed to human beta-amyloid precursor protein 695.

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Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation.

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Alzheimer's Aβ(1–42) is generated in the endoplasmic reticulum/intermediate compartment of NT2N cells

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Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein

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Intraneuronal Abeta42 accumulation in human brain.

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Amyloid beta-peptide is produced by cultured cells during normal metabolism

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Distinct sites of intracellular production for Alzheimer's disease A beta40/42 amyloid peptides.

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Rapid cellular uptake of Alzheimer amyloid betaA4 peptide by cultured human neuroblastoma cells

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Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43).

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The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor

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Sequence of deposition of heterogeneous amyloid beta-peptides and APO E in Down syndrome: implications for initial events in amyloid plaque formation.

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Reduced antioxidant enzyme activity in brains of mice transgenic for human presenilin-1 with single or multiple mutations.

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Mutagenesis identifies new signals for beta-amyloid precursor protein endocytosis, turnover, and the generation of secreted fragments, including Abeta42

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7 January 2021

Human neurons derived from a teratocarcinoma cell line express solely the 695-amino acid amyloid precursor protein and produce intracellular beta-amyloid or A4 peptides

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7 January 2021

Generation of Alzheimer beta-amyloid protein in the trans-Golgi network in the apparent absence of vesicle formation

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Identifiers

DOI

1 reference

16 February 2018

1 reference

16 February 2018