(Q47881793)

English

Analysis of partner of inscuteable, a novel player of Drosophila asymmetric divisions, reveals two distinct steps in inscuteable apical localization

scientific article published in February 2000

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Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

Analysis of partner of inscuteable, a novel player of Drosophila asymmetric divisions, reveals two distinct steps in inscuteable apical localization (English)

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

1

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

author name string

Morin X

2

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

Cai Y

3

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

Yang X

4

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

Chia W

5

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

language of work or name

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publication date

1 February 2000

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

100

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

4

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

399-409

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

The embryonic central nervous system lineages of Drosophila melanogaster. I. Neuroblast lineages derived from the ventral half of the neuroectoderm.

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Staufen-dependent localization of prospero mRNA contributes to neuroblast daughter-cell fate.

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Functional cDNA libraries from Drosophila embryos

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Binary sibling neuronal cell fate decisions in the Drosophila embryonic central nervous system are nonstochastic and require inscuteable-mediated asymmetry of ganglion mother cells

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Numb diverts notch pathway off the tramtrack

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inscuteable and numb mediate asymmetric muscle progenitor cell divisions during Drosophila myogenesis

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Asymmetric cell division and neurogenesis

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The prospero gene specifies cell fates in the drosophila central nervous system

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Asymmetrically distributed PAR-3 protein contributes to cell polarity and spindle alignment in early C. elegans embryos

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The Drosophila Numb protein inhibits signaling of the Notch receptor during cell-cell interaction in sensory organ lineage

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The TPR snap helix: a novel protein repeat motif from mitosis to transcription

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Molecular genetics of asymmetric cleavage in the early Caenorhabditis elegans embryo

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Control of daughter cell fates during asymmetric division: interaction of Numb and Notch

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Asymmetric segregation of the homeodomain protein Prospero during Drosophila development.

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Mechanisms of asymmetric cell division: Two Bs or not two Bs, that is the question

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Miranda directs Prospero to a daughter cell during Drosophila asymmetric divisions

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An atypical PKC directly associates and colocalizes at the epithelial tight junction with ASIP, a mammalian homologue of Caenorhabditis elegans polarity protein PAR-3

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Asymmetric cell division

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Asymmetric segregation of Numb and Prospero during cell division

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Deletion analysis of the Drosophila Inscuteable protein reveals domains for cortical localization and asymmetric localization

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7 January 2021

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inscuteable, a neural precursor gene of Drosophila, encodes a candidate for a cytoskeleton adaptor protein

1 reference

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7 January 2021

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Role of inscuteable in orienting asymmetric cell divisions in Drosophila

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Control of spindle orientation in Drosophila by the Par-3-related PDZ-domain protein Bazooka

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7 January 2021

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Inscuteable and Staufen mediate asymmetric localization and segregation of prospero RNA during Drosophila neuroblast cell divisions.

1 reference

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7 January 2021

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Partner of Numb colocalizes with Numb during mitosis and directs Numb asymmetric localization in Drosophila neural and muscle progenitors

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Asymmetric cell division: lessons from flies and worms

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Cloning of the Drosophila prospero gene and its expression in ganglion mother cells

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Identification and cDNA cloning of a novel human mosaic protein, LGN, based on interaction with G alpha i2

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armadillo, bazooka, and stardust are critical for early stages in formation of the zonula adherens and maintenance of the polarized blastoderm epithelium in Drosophila.

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Asymmetric distribution of numb protein during division of the sensory organ precursor cell confers distinct fates to daughter cells

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A modular misexpression screen in Drosophila detecting tissue-specific phenotypes

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The embryonic central nervous system lineages of Drosophila melanogaster. II. Neuroblast lineages derived from the dorsal part of the neuroectoderm.

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Bazooka recruits Inscuteable to orient asymmetric cell divisions in Drosophila neuroblasts.

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Miranda mediates asymmetric protein and RNA localization in the developing nervous system

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Protein localization and cell fate in bacteria

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Miranda Is Required for the Asymmetric Localization of Prospero during Mitosis in Drosophila

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Miranda as a multidomain adapter linking apically localized Inscuteable and basally localized Staufen and Prospero during asymmetric cell division in Drosophila

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Numb antagonizes Notch signaling to specify sibling neuron cell fates

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Identifiers

10.1016/S0092-8674(00)80676-5

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 February 2018

http://europepmc.org/abstract/MED/10693757

ResearchGate publication ID

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